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Your search keyword '"Pochet, Jean-Michel"' showing total 4 results
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4 results on '"Pochet, Jean-Michel"'

1. The patient with metabolic alkalosis

Author

UCL - SSS/IREC/NEFR - Pôle de Néphrologie, UCL - (SLuc) Service de néphrologie, Gillion, Valentine, Jadoul, Michel, Devuyst, Olivier, Pochet, Jean-Michel, UCL - SSS/IREC/NEFR - Pôle de Néphrologie, UCL - (SLuc) Service de néphrologie, Gillion, Valentine, Jadoul, Michel, Devuyst, Olivier, and Pochet, Jean-Michel

Abstract

Metabolic alkalosis defined by the increase of both plasma HCO3- level (>26 mmol/L) and blood arterial pH (>7.43) is quite frequent and usually accompanied by hypokalemia. Its pathogenesis requires both the generation of alkalosis and its maintenance. Generation may be due to excessive hydrogen ion loss by the gastrointestinal tract (e.g. vomiting) or by the kidney (e.g. use of loop diuretics) or may be due to exogenous base gain. Maintenance reflects the inability of the kidney to excrete the excess of bicarbonate because of hypovolemia, chloride depletion, hypokalemia, hyperaldosteronism, renal failure or a combination of these factors. The evaluation of volemic status and measurement of urinary Cl- and plasma levels of renin and aldosterone are crucial to identify the cause(s) of metabolic alkalosis. The cornerstone of treatment is the correction of existing depletions and the prevention of further losses. In vomiting-induced chloride depletion alkalosis, infusion of potassium chloride restores the excretion of bicarbonate by the kidney.

Published
2019

2. Belgian consensus statement on the diagnosis and management of patients with atypical hemolytic uremic syndrome

Author

UCL - SSS/IREC/NEFR - Pôle de Néphrologie, UCL - SSS/IREC/SLUC - Pôle St.-Luc, UCL - SSS/IREC/PEDI - Pôle de Pédiatrie, UCL - (SLuc) Service de néphrologie, UCL - (SLuc) Service de pédiatrie générale, Claes, Kathleen, Massart, Annick, Collard, Laure, Weekers, Laurent, Goffin, Eric, Pochet, Jean-Michel, Dahan, Karin, Morelle, Johann, Adams, Brigitte, Broeders, Nilufer, Stordeur, Patrick, Abramowicz, Daniel, Bosmans, Jean-Louis, Van Hoeck, Koen, Janssens, Peter, Pipeleers, Lissa, Peeters, Patrick, Van Laecke, Steven, Levtchenko, Elena, Sprangers, Ben, van den Heuvel, Lambertus, Godefroid, Nathalie, Van de Walle, Johan, UCL - SSS/IREC/NEFR - Pôle de Néphrologie, UCL - SSS/IREC/SLUC - Pôle St.-Luc, UCL - SSS/IREC/PEDI - Pôle de Pédiatrie, UCL - (SLuc) Service de néphrologie, UCL - (SLuc) Service de pédiatrie générale, Claes, Kathleen, Massart, Annick, Collard, Laure, Weekers, Laurent, Goffin, Eric, Pochet, Jean-Michel, Dahan, Karin, Morelle, Johann, Adams, Brigitte, Broeders, Nilufer, Stordeur, Patrick, Abramowicz, Daniel, Bosmans, Jean-Louis, Van Hoeck, Koen, Janssens, Peter, Pipeleers, Lissa, Peeters, Patrick, Van Laecke, Steven, Levtchenko, Elena, Sprangers, Ben, van den Heuvel, Lambertus, Godefroid, Nathalie, and Van de Walle, Johan

Published
2018

3. The patient with metabolic alkalosis.

Author

Gillion V, Jadoul M, Devuyst O, and Pochet JM

Subjects
Aged, 80 and over, Alkalosis diagnosis, Alkalosis therapy, Diagnosis, Differential, Female, Humans, Alkalosis etiology
Abstract

Metabolic alkalosis defined by the increase of both plasma HCO3- level (>26 mmol/L) and blood arterial pH (>7.43) is quite frequent and usually accompanied by hypokalemia. Its pathogenesis requires both the generation of alkalosis and its maintenance. Generation may be due to excessive hydrogen ion loss by the gastrointestinal tract (e.g. vomiting) or by the kidney (e.g. use of loop diuretics) or may be due to exogenous base gain. Maintenance reflects the inability of the kidney to excrete the excess of bicarbonate because of hypovolemia, chloride depletion, hypokalemia, hyperaldosteronism, renal failure or a combination of these factors. The evaluation of volemic status and measurement of urinary Cl- and plasma levels of renin and aldosterone are crucial to identify the cause(s) of metabolic alkalosis. The cornerstone of treatment is the correction of existing depletions and the prevention of further losses. In vomiting-induced chloride depletion alkalosis, infusion of potassium chloride restores the excretion of bicarbonate by the kidney.

Published
2019
Full Text
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4. Belgian consensus statement on the diagnosis and management of patients with atypical hemolytic uremic syndrome.

Author

Claes KJ, Massart A, Collard L, Weekers L, Goffin E, Pochet JM, Dahan K, Morelle J, Adams B, Broeders N, Stordeur P, Abramowicz D, Bosmans JL, Van Hoeck K, Janssens P, Pipeleers L, Peeters P, Van Laecke S, Levtchenko E, Sprangers B, van den Heuvel L, Godefroid N, and Van de Walle J

Subjects
Adult, Antibodies, Monoclonal, Humanized therapeutic use, Belgium, Child, Consensus, Humans, Kidney Transplantation, Nephrology organization & administration, Practice Guidelines as Topic, Atypical Hemolytic Uremic Syndrome diagnosis, Atypical Hemolytic Uremic Syndrome therapy
Published
2018
Full Text
View/download PDF

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