20 results on '"Wilton, A. D."'
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2. Induction of cryptic pre-mRNA splice-switching by antisense oligonucleotides
3. NEAT1 polyA-modulating antisense oligonucleotides reveal opposing functions for both long non-coding RNA isoforms in neuroblastoma
4. Splice modulating antisense oligonucleotides restore some acid-alpha-glucosidase activity in cells derived from patients with late-onset Pompe disease
5. Reduction of integrin alpha 4 activity through splice modulating antisense oligonucleotides
6. Breakpoint junction features of seven DMD deletion mutations
7. The potential of antisense oligonucleotide therapies for inherited childhood lung diseases
8. A novel BRD4-NUT fusion in an undifferentiated sinonasal tumor highlights alternative splicing as a contributing oncogenic factor in NUT midline carcinoma
9. Erratum: Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice
10. Erratum: Corrigendum: Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice
11. Translation from a DMD exon 5 IRES results in a functional dystrophin isoform that attenuates dystrophinopathy in humans and mice
12. Regulation of eukaryotic gene expression by the untranslated gene regions and other non-coding elements
13. Mismatched single stranded antisense oligonucleotides can induce efficient dystrophin splice switching
14. Hybridisation within Brassica and allied genera: evaluation of potential for transgene escape
15. Antisense oligonucleotide induced exon skipping and the dystrophin gene transcript: cocktails and chemistries
16. Antisense oligonucleotide-induced exon skipping restores dystrophin expression in vitro in a canine model of DMD
17. Induction of revertant fibres in the mdx mouse using antisense oligonucleotides
18. Systemic delivery of morpholino oligonucleotide restores dystrophin expression bodywide and improves dystrophic pathology
19. Functional amounts of dystrophin produced by skipping the mutated exon in the mdx dystrophic mouse
20. A mutation in the α tropomyosin gene TPM3 associated with autosomal dominant nemaline myopathy
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