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39 results on '"Hoorn, Ewout J'

1. Dietary sodium restriction prevents vascular endothelial growth factor inhibitor-induced hypertension

Author

van Doorn, Leni, primary, Visser, Wesley J., additional, van Dorst, Daan C. H., additional, Mirabito Colafella, Katrina M., additional, Koolen, Stijn L. W., additional, de Mik, Anneke van Egmond-, additional, Garrelds, Ingrid M., additional, Bovée, Dominique M., additional, de Hoop, Esther Oomen-, additional, Bins, Sander, additional, Eskens, Ferry A. L. M., additional, Hoorn, Ewout J., additional, Jan Danser, A. H., additional, Mathijssen, Ron H. J., additional, and Versmissen, Jorie, additional

Published
2022
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2. Adult human kidney organoids originate from CD24+ cells and represent an advanced model for adult polycystic kidney disease

Author

Xu, Yaoxian, primary, Kuppe, Christoph, additional, Perales-Patón, Javier, additional, Hayat, Sikander, additional, Kranz, Jennifer, additional, Abdallah, Ali T., additional, Nagai, James, additional, Li, Zhijian, additional, Peisker, Fabian, additional, Saritas, Turgay, additional, Halder, Maurice, additional, Menzel, Sylvia, additional, Hoeft, Konrad, additional, Kenter, Annegien, additional, Kim, Hyojin, additional, van Roeyen, Claudia R. C., additional, Lehrke, Michael, additional, Moellmann, Julia, additional, Speer, Thimoteus, additional, Buhl, Eva M., additional, Hoogenboezem, Remco, additional, Boor, Peter, additional, Jansen, Jitske, additional, Knopp, Cordula, additional, Kurth, Ingo, additional, Smeets, Bart, additional, Bindels, Eric, additional, Reinders, Marlies E. J., additional, Baan, Carla, additional, Gribnau, Joost, additional, Hoorn, Ewout J., additional, Steffens, Joachim, additional, Huber, Tobias B., additional, Costa, Ivan, additional, Floege, Jürgen, additional, Schneider, Rebekka K., additional, Saez-Rodriguez, Julio, additional, Freedman, Benjamin S., additional, and Kramann, Rafael, additional

Published
2022
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3. Comment to “Recommendation on an updated standardization of serum magnesium reference ranges”

Author

de Baaij, Jeroen H. F., primary, Bockenhauer, Detlef, additional, Claverie-Martin, Felix, additional, Hoenderop, Joost G. J., additional, Hoorn, Ewout J., additional, Houillier, Pascal, additional, Knoers, Nine V. A. M., additional, Konrad, Martin, additional, Müller, Dominik, additional, Nijenhuis, Tom, additional, Schlingmann, Karl Peter, additional, and Vargas Poussou, Rosa, additional

Published
2022
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6. Personalized screening intervals for kidney function in patients with chronic heart failure: a modeling study

Author

Schuurman, Anne-Sophie, primary, Tomer, Anirudh, additional, Akkerhuis, K. Martijn, additional, Hoorn, Ewout J., additional, Brugts, Jasper J., additional, Manintveld, Olivier C., additional, van Ramshorst, Jan, additional, Umans, Victor A., additional, Boersma, Eric, additional, Rizopoulos, Dimitris, additional, and Kardys, Isabella, additional

Published
2021
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8. Long-term prognosis after kidney donation: a propensity score matched comparison of living donors and non-donors from two population cohorts

Author

Janki, Shiromani, primary, Dehghan, Abbas, additional, van de Wetering, Jacqueline, additional, Steyerberg, Ewout W., additional, Klop, Karel W. J., additional, Kimenai, Hendrikus J. A. N., additional, Rizopoulos, Dimitris, additional, Hoorn, Ewout J., additional, Stracke, Sylvia, additional, Weimar, Willem, additional, Völzke, Henry, additional, Hofman, Albert, additional, and Ijzermans, Jan N. M., additional

Published
2020
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10. Correction to: Outcomes of parathyroidectomy versus calcimimetics for secondary hyperparathyroidism and kidney transplantation: a propensity-matched analysis

Author

Schelto Kruijff, Abbey Schepers, Ewout J. Hoorn, Willemijn Y. van der Plas, Tessa M van Ginhoven, Anton F. Engelsman, Ezra Y Koh, Robert A. Pol, Joris I. Rotmans, Els J. M. Nieveen van Dijkum, Natasha M. Appelman-Dijkstra, Liffert Vogt, Martin H. de Borst, Roderick R. Dulfer, Surgery, Intensive Care Medicine, AII - Infectious diseases, AII - Inflammatory diseases, Amsterdam Gastroenterology Endocrinology Metabolism, Nephrology, APH - Health Behaviors & Chronic Diseases, and ACS - Microcirculation

Subjects
Parathyroidectomy, medicine.medical_specialty, business.industry, medicine.medical_treatment, Mistake, medicine.disease, Eleventh, Surgery, Propensity score matching, Medicine, Secondary hyperparathyroidism, business, Kidney transplantation
Abstract

The original version of this article unfortunately contained a mistake on the fifth and eleventh author names, from Schelto Kruijf to Schelto Kruijff and from Tessa van Ginhoven to Tessa M. van Ginhoven. The corrected author names are shown below. Schelto Kruijff and Tessa M. van Ginhoven.

Published
2021

11. Yield of diagnostic tests in unexplained renal hypophosphatemia: a case series

Author

Ewout J. Hoorn, Robert Zietse, Anneke P. Bech, Jack F.M. Wetzels, Tom Nijenhuis, and Internal Medicine

Subjects
Adult, Male, 0301 basic medicine, Nephrology, medicine.medical_specialty, Hypophosphatemia, 030209 endocrinology & metabolism, lcsh:RC870-923, urologic and male genital diseases, Contraceptives, Oral, Hormonal, Phosphates, 03 medical and health sciences, All institutes and research themes of the Radboud University Medical Center, 0302 clinical medicine, Tubulopathy, FGF23, Internal medicine, medicine, Humans, Referral and Consultation, Netherlands, Retrospective Studies, Genetic testing, Osteomalacia, medicine.diagnostic_test, Renal hypophosphatemia, Diagnostic Tests, Routine, business.industry, Middle Aged, lcsh:Diseases of the genitourinary system. Urology, medicine.disease, Fibroblast Growth Factors, Fibroblast Growth Factor-23, stomatognathic diseases, Renal disorders Radboud Institute for Molecular Life Sciences [Radboudumc 11], 030104 developmental biology, Somatostatin, Female, Renal disorders Radboud Institute for Health Sciences [Radboudumc 11], business, Tumor-induced osteomalacia, Progressive disease, Research Article
Abstract

Background Isolated renal hypophosphatemia may be inherited or acquired. An increasing number of patients with unexplained renal hypophosphatemia is being referred to our clinics, but the optimal diagnostic work-up is not known. Therefore, the aim of this study was to assess the diagnostic yield in these patients. Methods We retrospectively evaluated all patients who were referred because of unexplained isolated renal hypophosphatemia to two academic tertiary referral centers in The Netherlands in the period of 2013–2017. Results We evaluated 17 patients. In five female patients renal hypophosphatemia could be attributed to the use of oral contraceptives. The other 12 patients had a median age of 48 years (10 males). There were no other signs of tubulopathy and none of the patients used drugs known to be associated with hypophosphatemia. FGF23 levels were above normal (> 125 RU/ml) in 2/12 patients. Genetic testing, performed in all patients, did not identify a mutation in genes known to be associated with renal phosphate wasting. A scan with a radiolabeled somatostatin analogue was performed in 8 patients. In one patient, with an FGF23 level of 110 RU/ml, an increased uptake of the somatostatin analog was observed due to tumor induced osteomalacia (TIO). Conclusions Oral contraceptive use is an important but under-recognized cause of renal hypophosphatemia. The cause of isolated renal hypophosphatemia remained unexplained in the majority of other patients despite extensive and expensive additional investigations. The pre-test probability for tumor-induced osteomalacia or inherited renal hypophosphatemia in a patient with aspecific complaints and a normal FGF23 level is low. Further research is needed to investigate which patients should be screened for TIO. At present we suggest to perform somatostatin scans only in patients with severe complaints, elevated FGF23 levels, or progressive disease.

Published
2018

12. A preliminary survey of practice patterns across several European kidney stone centers and a call for action in developing shared practice

Author

Ferraro, Pietro Manuel, primary, Arrabal-Polo, Miguel Ángel, additional, Capasso, Giovambattista, additional, Croppi, Emanuele, additional, Cupisti, Adamasco, additional, Ernandez, Thomas, additional, Fuster, Daniel G., additional, Galan, Juan Antonio, additional, Grases, Felix, additional, Hoorn, Ewout J., additional, Knauf, Felix, additional, Letavernier, Emmanuel, additional, Mohebbi, Nilufar, additional, Moochhala, Shabbir, additional, Petkova, Kremena, additional, Pozdzik, Agnieszka, additional, Sayer, John, additional, Seitz, Christian, additional, Strazzullo, Pasquale, additional, Trinchieri, Alberto, additional, Vezzoli, Giuseppe, additional, Vitale, Corrado, additional, Vogt, Liffert, additional, Unwin, Robert J., additional, Bonny, Olivier, additional, and Gambaro, Giovanni, additional

Published
2019
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13. Everything you need to know about distal renal tubular acidosis in autoimmune disease

Author

Jan A. M. van Laar, Virgil A. S. H. Dalm, Paul L A van Daele, Tim Both, P. Martin van Hagen, Ewout J. Hoorn, Robert Zietse, Internal Medicine, and Immunology

Subjects
medicine.medical_specialty, Acid–base balance, Urinary system, Immunology, Autoimmunity, Review Article, Acid–base homeostasis, Urinalysis, Nephrolithiasis, Gastroenterology, Autoimmune Diseases, Renal tubular acidosis, Rheumatology, Distal renal tubular acidosis, Predictive Value of Tests, Risk Factors, Potassium Citrate, Internal medicine, medicine, Animals, Humans, Immunology and Allergy, Kidney Tubules, Distal, Acidosis, Acid-Base Equilibrium, Autoimmune disease, business.industry, Acidosis, Renal Tubular, Hydrogen-Ion Concentration, medicine.disease, Renal physiology, Treatment Outcome, Endocrinology, medicine.symptom, business, Homeostasis
Abstract

Renal acid-base homeostasis is a complex process, effectuated by bicarbonate reabsorption and acid secretion. Impairment of urinary acidification is called renal tubular acidosis (RTA). Distal renal tubular acidosis (dRTA) is the most common form of the RTA syndromes. Multiple pathophysiologic mechanisms, each associated with various etiologies, can lead to dRTA. The most important consequence of dRTA is (recurrent) nephrolithiasis. The diagnosis is based on a urinary acidification test. Potassium citrate is the treatment of choice.

Published
2014

21. Bone Mineral Density in Sjögren Syndrome Patients with and Without Distal Renal Tubular Acidosis

Author

Both, Tim, primary, Zillikens, M. Carola, additional, Hoorn, Ewout J., additional, Zietse, Robert, additional, van Laar, Jan A. M., additional, Dalm, Virgil A. S. H., additional, van Duijn, Cornelia M., additional, Versnel, Marjan A., additional, Maria, Naomi I., additional, van Hagen, P. Martin, additional, and van Daele, Paul L. A., additional

Published
2016
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23. Distal renal tubular acidosis in primary Sjögren syndrome

Author

Paul L A van Daele, Tim Both, Robert Zietse, Zana Brkic, Jan Am van Laar, Ewout J. Hoorn, Marjan A. Versnel, and P. Martin van Hagen

Subjects
musculoskeletal diseases, Pathology, medicine.medical_specialty, Exocrine gland, lcsh:Medicine, Urine, General Biochemistry, Genetics and Molecular Biology, stomatognathic system, Distal renal tubular acidosis, medicine, Secretion, Primary Sjögren Syndrome, Medicine(all), Lymphocytic infiltration, Biochemistry, Genetics and Molecular Biology(all), business.industry, lcsh:R, General Medicine, medicine.disease, Chronic inflammatory disorder, eye diseases, stomatognathic diseases, medicine.anatomical_structure, Poster Presentation, business, Duct (anatomy)
Abstract

Primary Sjogren syndrome (pSS) is a chronic inflammatory disorder characterized by lymphocytic infiltration of exocrine glands. pSS can also cause distal renal tubular acidosis (dRTA). dRTA is a disorder in which patients are unable to acidify their urine because of impaired hydrogen ion secretion in the collecting duct.

Published
2012

25. Erratum to: Clinical practice guideline on diagnosis and treatment of hyponatraemia

Author

Spasovski, Goce, primary, Vanholder, Raymond, additional, Allolio, Bruno, additional, Annane, Djillali, additional, Ball, Steve, additional, Bichet, Daniel, additional, Decaux, Guy, additional, Fenske, Wiebke, additional, Hoorn, Ewout J., additional, Ichai, Carole, additional, Joannidis, Michael, additional, Soupart, Alain, additional, Zietse, Robert, additional, Haller, Maria, additional, van der Veer, Sabine, additional, Van Biesen, Wim, additional, and Nagler, Evi, additional

Published
2014
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35. The calcineurin inhibitor tacrolimus activates the renal sodium chloride cotransporter to cause hypertension

Author

Hoorn, Ewout J, primary, Walsh, Stephen B, additional, McCormick, James A, additional, Fürstenberg, Antje, additional, Yang, Chao-Ling, additional, Roeschel, Tom, additional, Paliege, Alexander, additional, Howie, Alexander J, additional, Conley, James, additional, Bachmann, Sebastian, additional, Unwin, Robert J, additional, and Ellison, David H, additional

Published
2011
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39. [Untitled]

Author

Denis F. Geary, Ewout J. Hoorn, E. van der Voort, M Robb, Desmond Bohn, and Mitchell L. Halperin

Subjects
Pediatrics, medicine.medical_specialty, Plasma sodium, business.industry, musculoskeletal, neural, and ocular physiology, Emergency department, Critical Care and Intensive Care Medicine, medicine.disease, Gastroenterology, Excretion, Internal medicine, biological sciences, cardiovascular system, Medicine, Observational study, business, Hyponatremia, tissues, Antidiuretic, Electrolyte Disorder, Hormone
Abstract

To develop hyponatremia (plasma sodium concentration (PNa) < 136 mM), there must be a source of electrolyte free water (EFW) and actions of antidiuretic hormone (ADH) to prevent its excretion. A low PNa is the most common electrolyte disorder in hospitalized children and it makes them more prone to neurological damage.

Published
2002

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