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1. The inflammasome adaptor protein ASC promotes amyloid deposition in cryopyrin-associated periodic syndromes.

2. Pathogenic NLRP3 mutants form constitutively active inflammasomes resulting in immune-metabolic limitation of IL-1β production.

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3. The NLRP3 inflammasome is released as a particulate danger signal that amplifies the inflammatory response.

4. LTBP2 and CYP1B1 mutations and associated ocular phenotypes in the Roma/Gypsy founder population.

5. Biallelic loss-of-function LACC1/FAMIN Mutations Presenting as Rheumatoid Factor-Negative Polyarticular Juvenile Idiopathic Arthritis.