12 results on '"Bussolino F"'
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2. Erythrocyte stages of Plasmodium falciparum exhibit a high nitric oxide synthase (NOS) activity and release an NOS-inducing soluble factor.
3. Middle T antigen-transformed endothelial cells exhibit an increased activity of nitric oxide synthase.
4. Tumor necrosis factor alpha-induced angiogenesis depends on in situ platelet-activating factor biosynthesis.
5. Antiinflammatory peptides (antiflammins) inhibit synthesis of platelet-activating factor, neutrophil aggregation and chemotaxis, and intradermal inflammatory reactions.
6. Synthesis and release of platelet-activating factor is inhibited by plasma alpha 1-proteinase inhibitor or alpha 1-antichymotrypsin and is stimulated by proteinases.
7. Tumor necrosis factor/cachectin stimulates peritoneal macrophages, polymorphonuclear neutrophils, and vascular endothelial cells to synthesize and release platelet-activating factor.
8. MRCKα is activated by caspase cleavage to assemble an apical actin ring for epithelial cell extrusion.
9. PDK1-mediated activation of MRCKα regulates directional cell migration and lamellipodia retraction.
10. Essential role of PDK1 in regulating endothelial cell migration.
11. Stable interaction between alpha5beta1 integrin and Tie2 tyrosine kinase receptor regulates endothelial cell response to Ang-1.
12. Ghrelin and des-acyl ghrelin inhibit cell death in cardiomyocytes and endothelial cells through ERK1/2 and PI 3-kinase/AKT.
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