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9. The Collected Works of Jeremy Bentham: The Correspondence of Jeremy Bentham, Vol. 12: July 1824 to June 1828

Author

Canning, George, primary, Smith, Sydney, additional, Bowring, Sir John, additional, Brailsford, Henry Noel, additional, Fonblanque, Albany William, additional, Blaquiere, Edward, additional, Fletcher, Elizabeth, additional, Bentham, Jeremy, additional, Mill, John Stuart, additional, baronet, Robert Peel, third, additional, Dumont, Étienne, additional, Koe, John Herbert, additional, Place, Francis, additional, Say, Jean-Baptise, additional, baronet, Sir Francis Burdett, fifth, additional, Louriottos, Andreas, additional, Hill, Matthew Davenport, additional, Bickersteth, Henry, additional, Young, James O., additional, Austin, Sarah, additional, Herrera, Prospero de, additional, Humphreys, James, additional, Brougham, Henry, additional, baronet, Sir Robert Peel, second, additional, Adam, William, additional, Butler, Charles, additional, Vaux, Henry Peter Brougham, first Baron B, additional, Mill, James, additional, Say, Jean-Baptiste, additional, Cartwright, John, additional, Allen, William, additional, MacCulloch, John, additional, Plumer, William, additional, Bonfield, William, additional, Rebecque, Benjamin Constant de, additional, [D'Arusmont], Frances Wright, additional, Rey, Joseph-Philippe-Étienne, additional, [Taylor], Sarah Austin, additional, Grote, George, additional, Blanco, Simón José Antonio de la Santísima, additional, Orlandos, Ioannis, additional, Louriottis, Andreas, additional, Bodin, Félix, additional, Harrington, Leicester Fitzgerald Charles Stanho, additional, Hill, Sir Rowland, additional, Hill, Thomas Wright, additional, Parkes, Joseph, additional, Langdale, Henry Bickersteth, first Baron, additional, Roebuck, John Arthur, additional, Fry, A., additional, Zaïmis, Ioannis, additional, Lévy, Armand, additional, Carlanstown, George Nugent Grenville, second Bar, additional, Cooper, Charles Purton, additional, Barker, Edmund Henry, additional, Antoniadis, Emmanuel, additional, Rallis, Eustratios, additional, Young, Lt.-Col. James, additional, [Lewin], Harriet Grote, additional, Moyes, James, additional, Armstrong, John, additional, Crawfurd, John, additional, Colls, John Flowerdew, additional, Marshall, John, additional, Neal, John, additional, Valle, José Cecilio Díaz del, additional, Hume, Joseph, additional, Barrot, Camille Hyacinthe Odilon, additional, Herrera, Próspero de, additional, Martin, Richard, additional, Lewin, Samuel Hawtayne, additional, Sharpe, Sutton, additional, Calder, Taplin, additional, Lansdowne, Henry Petty-Fitzmaurice, third marq, additional, Hunter, W. G. P., additional, and Devon, William Courtenay, tenth Earl of, additional

Published
2006
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11. The Collected Works of Jeremy Bentham: The Correspondence of Jeremy Bentham, Vol. 8: January 1809 to December 1816

Author

Hunt, (James Henry) Leigh, primary, White, Joseph Blanco, additional, Baillie, Joanna, additional, Wilberforce, Henry William, additional, Blaquiere, Edward, additional, Bentham, Jeremy, additional, Mill, John Stuart, additional, Dumont, Étienne, additional, Romilly, Sir Samuel, additional, Bentham, Sir Samuel, additional, Burr, Aaron, additional, Mill, James, additional, Koe, John Herbert, additional, Beckett, John, additional, Warton, Richard, additional, Vizard, William, additional, Arbuthnot, Charles, additional, Pickton, John, additional, Bailie, Joanna, additional, Evans, William David, additional, Holford, George, additional, Woodforde, Charles, additional, Ricardo, David, additional, baronet, Sir John Sinclair, first, additional, Foxley, Henry Richard Fox [Vassall], third, additional, baronet, Sir Thomas Charles Bunbury, sixth, additional, Sidmouth, Henry Addington, first Viscount, additional, Huskisson, William, additional, Wilberforce, William, additional, Roget, Peter Mark, additional, Helens, Alleyne Fitzherbert, Baron St, additional, Horner, Francis, additional, Bentham, George, additional, Jekyll, Joseph, additional, [Fordyce], Mary [Maria Sophia] Bentham, additional, Mackintosh, Sir James, additional, Evans, Sir William David, additional, Russia, Alexander I of, additional, Taylor, Arthur, additional, Slaughton, Edward Burtenshaw Sugden, Baron St, additional, Miranda, Francisco de, additional, Jovellanos, Ramirez-Gaspar Melchor de, additional, Harrison, Sir George, additional, Vaux, Henry Peter Brougham, first Baron B, additional, Goulburn, Henry, additional, Rhodes, Henry, additional, Coulson, Henry Tonkin, additional, Dunfermline, James Abercromby, first Baron, additional, Madison, James, additional, Say, Jean-Baptiste, additional, Bragge, John, additional, Cartwright, John, additional, Hullock, Sir John, additional, James, John, additional, [McCreery], John M'Creery, additional, Lowe, Joseph, additional, Vernon, Joseph, additional, [Garbett], Lady Anne Romilly, additional, [Delessert], Madeleine Gautier, additional, Chichagov, Pavel Vasil'evich, additional, Payne, Thomas, additional, Foss, Henry, additional, Czartoryski, Prince Adam Jerzy, additional, Wharton, Richard, additional, Nickolls, Rev. Robert Boucher, additional, Bentham, Samuel, additional, Lasteyrie, Charles Philibert de Lasteyrie du S, additional, Beeby, Thomas, additional, Northmore, Thomas, additional, Allen, William, additional, Collins, William, additional, Hill, William, additional, and Salisbury, William, additional

Published
1988
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13. The Collected Works of Jeremy Bentham: The Correspondence of Jeremy Bentham, Vol. 12: July 1824 to June 1828

Author

George Canning, Sydney Smith, Sir John Bowring, Henry Noel Brailsford, Albany William Fonblanque, Edward Blaquiere, Elizabeth Fletcher, Jeremy Bentham, John Stuart Mill, Robert Peel, third baronet, Étienne Dumont, John Herbert Koe, Francis Place, Jean-Baptise Say, Sir Francis Burdett, fifth baronet, Andreas Louriottos, Matthew Davenport Hill, Henry Bickersteth, James O. Young, Sarah Austin, Prospero de Herrera, James Humphreys, Henry Brougham, Sir Robert Peel, second baronet, William Adam, Charles Butler, Henry Peter Brougham, first Baron B Vaux, James Mill, Jean-Baptiste Say, John Cartwright, William Allen, John MacCulloch, William Plumer, William Bonfield, Benjamin Constant de Rebecque, Frances Wright [D'Arusmont], Joseph-Philippe-Étienne Rey, Sarah Austin [Taylor], George Grote, Simón José Antonio de la Santísima Blanco, Ioannis Orlandos, Andreas Louriottis, Félix Bodin, Leicester Fitzgerald Charles Stanho Harrington, Sir Rowland Hill, Thomas Wright Hill, Joseph Parkes, Henry Bickersteth, first Baron Langdale, John Arthur Roebuck, A. Fry, Ioannis Zaïmis, Armand Lévy, George Nugent Grenville, second Bar Carlanstown, Charles Purton Cooper, Edmund Henry Barker, Emmanuel Antoniadis, Eustratios Rallis, Lt.-Col. James Young, Harriet Grote [Lewin], James Moyes, John Armstrong, John Crawfurd, John Flowerdew Colls, John Marshall, John Neal, José Cecilio Díaz del Valle, Joseph Hume, Camille Hyacinthe Odilon Barrot, Próspero de Herrera, Richard Martin, Samuel Hawtayne Lewin, Sutton Sharpe, Taplin Calder, Henry Petty-Fitzmaurice, third marq Lansdowne, W. G. P. Hunter, and William Courtenay, tenth Earl of Devon

Published
2006
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25. Transgressive segregation, hopeful monsters, and phenotypic selection drove rapid genetic gains and breakthroughs in predictive breeding for quantitative resistance to Macrophomina in strawberry.

Author

Knapp SJ, Cole GS, Pincot DDA, Dilla-Ermita CJ, Bjornson M, Famula RA, Gordon TR, Harshman JM, Henry PM, and Feldmann MJ

Abstract

Two decades have passed since the strawberry ( Fragaria x ananassa ) disease caused by Macrophomina phaseolina , a necrotrophic soilborne fungal pathogen, began surfacing in California, Florida, and elsewhere. This disease has since become one of the most common causes of plant death and yield losses in strawberry. The Macrophomina problem emerged and expanded in the wake of the global phase-out of soil fumigation with methyl bromide and appears to have been aggravated by an increase in climate change-associated abiotic stresses. Here we show that sources of resistance to this pathogen are rare in gene banks and that the favorable alleles they carry are phenotypically unobvious. The latter were exposed by transgressive segregation and selection in populations phenotyped for resistance to Macrophomina under heat and drought stress. The genetic gains were immediate and dramatic. The frequency of highly resistant individuals increased from 1% in selection cycle 0 to 74% in selection cycle 2. Using GWAS and survival analysis, we found that phenotypic selection had increased the frequencies of favorable alleles among 10 loci associated with resistance and that favorable alleles had to be accumulated among four or more of these loci for an individual to acquire resistance. An unexpectedly straightforward solution to the Macrophomina disease resistance breeding problem emerged from our studies, which showed that highly resistant cultivars can be developed by genomic selection per se or marker-assisted stacking of favorable alleles among a comparatively small number of large-effect loci., (© The Author(s) 2024. Published by Oxford University Press on behalf of Nanjing Agricultural University.)

Published
2024
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26. Gene Expression Networks in the Murine Pulmonary Myocardium Provide Insight into the Pathobiology of Atrial Fibrillation.

Author

Boutilier JK, Taylor RL, Mann T, McNamara E, Hoffman GJ, Kenny J, Dilley RJ, Henry P, Morahan G, Laing NG, and Nowak KJ

Subjects
Animals, Atrial Fibrillation metabolism, Atrial Fibrillation physiopathology, Biomarkers, Calcium metabolism, Chromosome Mapping, Computational Biology methods, Databases, Genetic, Gene Expression Profiling, Genetic Association Studies, MAP Kinase Kinase Kinases genetics, Mice, Molecular Sequence Annotation, Phenotype, Protein Serine-Threonine Kinases, Quantitative Trait Loci, Transcriptome, Atrial Fibrillation genetics, Gene Regulatory Networks, Genetic Predisposition to Disease
Abstract

The pulmonary myocardium is a muscular coat surrounding the pulmonary and caval veins. Although its definitive physiological function is unknown, it may have a pathological role as the source of ectopic beats initiating atrial fibrillation. How the pulmonary myocardium gains pacemaker function is not clearly defined, although recent evidence indicates that changed transcriptional gene expression networks are at fault. The gene expression profile of this distinct cell type in situ was examined to investigate underlying molecular events that might contribute to atrial fibrillation. Via systems genetics, a whole-lung transcriptome data set from the BXD recombinant inbred mouse resource was analyzed, uncovering a pulmonary cardiomyocyte gene network of 24 transcripts, coordinately regulated by chromosome 1 and 2 loci. Promoter enrichment analysis and interrogation of publicly available ChIP-seq data suggested that transcription of this gene network may be regulated by the concerted activity of NKX2-5, serum response factor, myocyte enhancer factor 2, and also, at a post-transcriptional level, by RNA binding protein motif 20. Gene ontology terms indicate that this gene network overlaps with molecular markers of the stressed heart. Therefore, we propose that perturbed regulation of this gene network might lead to altered calcium handling, myocyte growth, and contractile force contributing to the aberrant electrophysiological properties observed in atrial fibrillation. We reveal novel molecular interactions and pathways representing possible therapeutic targets for atrial fibrillation. In addition, we highlight the utility of recombinant inbred mouse resources in detecting and characterizing gene expression networks of relatively small populations of cells that have a pathological significance., (Copyright © 2017 Boutilier et al.)

Published
2017
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