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1. Club Cell Protein, CC10, Attenuates Acute Respiratory Distress Syndrome Induced by Smoke Inhalation

4. The Selective Vasopressin Type 1a Receptor Agonist Selepressin (FE 202158) Blocks Vascular Leak in Ovine Severe Sepsis*

5. Placental Findings in Singleton Stillbirths

6. γ-Tocopherol Nebulization Decreases Oxidative Stress, Arginase Activity, and Collagen Deposition After Burn and Smoke Inhalation in the Ovine Model

7. Nebulization With γ-Tocopherol Ameliorates Acute Lung Injury After Burn and Smoke Inhalation in the Ovine Model

12. The Angiotensin-Converting Enzyme Inhibitor Captopril Inhibits Poly(Adp-Ribose) Polymerase Activation and Exerts Beneficial Effects in an Ovine Model of Burn and Smoke Injury

13. Acute Lung Injury-Induced Collagen Deposition is Associated with Elevated Asymmetric Dimethylarginine and Arginase Activity

14. The Peroxynitrite Catalyst WW-85 Improves Pulmonary Function in Ovine Septic Shock

15. Muscarinic receptor antagonist therapy improves acute pulmonary dysfunction after smoke inhalation injury in sheep

16. Combined neuronal and inducible nitric oxide synthase inhibition in ovine acute lung injury*

17. Neuronal nitric oxide synthase inhibition attenuates cardiopulmonary dysfunctions after combined burn and smoke inhalation injury in sheep

18. UPPER AIRWAY MUCUS DEPOSITION IN LUNG TISSUE OF BURN TRAUMA VICTIMS

22. Arteriovenous CO2 Removal Improves Survival Compared to High Frequency Percussive and Low Tidal Volume Ventilation in a Smoke/Burn Sheep Acute Respiratory Distress Syndrome Model

23. INHIBITION OF NEURONAL NITRIC OXIDE SYNTHASE ATTENUATES PULMONARY TRANSVASCULAR FLUID FLUX AFTER BURN AND SMOKE INHALATION IN AN OVINE MODEL.

25. REGULATORY ROLE OF NEURONAL NITRIC OXIDE SYNTHASE INHIBITION IN OVINE ACUTE RESPIRATORY DISTRESS SYNDROME.

27. GAMMA-TOCOPHEROL NEBULIZATION ATTENUATES PULMONARY FAILURE IN SHEEP EXPOSED TO BURN AND SMOKE INHALATION INJURIES.

28. Recombinant human activated protein C improves pulmonary function in ovine acute lung injury resulting from smoke inhalation and sepsis

30. Combined burn and smoke inhalation injury impairs ovine hypoxic pulmonary vasoconstriction*

31. THE INHIBITION OF INDUCIBLE NITRIC OXIDE SYNTHASE IN OVINE SEPSIS MODEL

34. AEROSOLIZED ALPHA-TOCOPHEROL AMELIORATES ACUTE LUNG INJURY FOLLOWING COMBINED BURN AND SMOKE INHALATION INJURY IN SHEEP

35. SPECIFIC INOS INHIBITION BY 1400W IMPROVES PULMONARY FUNCTION FOLLOWING COMBINED BURN AND SMOKE INHALATION INJURY IN SHEEP.

37. LOW-DOSE VASOPRESSIN ATTENUATES CARDIOPULMONARY DYSFUNCTION IN SHEEP WITH COMBINED BURN AND SMOKE INHALATION INJURY: POTENTIAL ROLE OF 3-NITROTYROSINE.

39. NEURONAL NITRIC OXIDE SYNTHASE INHIBITION PREVENTS 3-NITROTYROSINE FORMATION AND ATTENUATES PULMONARY DYSFUNCTION IN OVINE LUNG INJURY

41. Inhibition of Poly (ADP-ribose) Polymerase Attenuates Acute Lung Injury in an Ovine Model of Sepsis

49. Effects of Insulin on Wound Healing

50. Complement C5a, TGF-β1, and MCP-1, in Sequence, Induce Migration of Monocytes Into Ischemic Canine Myocardium Within the First One to Five Hours After Reperfusion

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