16 results on '"BITO, Virginie"'
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2. Abstract 10507: Pyridoxamine Protects Against Cardiotoxicity After Doxorubicin Chemotherapy
3. Selective Modulation of Coupled Ryanodine Receptors During Microdomain Activation of Calcium/Calmodulin-Dependent Kinase II in the Dyadic Cleft
4. Combined Na + /Ca 2+ Exchanger and L-Type Calcium Channel Block as a Potential Strategy to Suppress Arrhythmias and Maintain Ventricular Function
5. Abstract P089: Estradiol Impairs Cardiac Contractile Function in Male Rodents
6. Ultrastructural and Functional Remodeling of the Coupling Between Ca 2+ Influx and Sarcoplasmic Reticulum Ca 2+ Release in Right Atrial Myocytes From Experimental Persistent Atrial Fibrillation
7. Response to Letter Regarding Article, “Ventricular Phosphodiesterase-5 Expression Is Increased in Patients With Advanced Heart Failure and Contributes to Adverse Ventricular Remodeling After Myocardial Infarction in Mice”
8. Ventricular Phosphodiesterase-5 Expression Is Increased in Patients With Advanced Heart Failure and Contributes to Adverse Ventricular Remodeling After Myocardial Infarction in Mice
9. Abstract 1534: Structural and Functional Remodeling of the Coupling between Sarcolemmal Ca 2+ Channels and the Ryanodine Receptor in Atrial Fibrillation
10. Pharmacological Inhibition of Na/Ca Exchange Results in Increased Cellular Ca 2+ Load Attributable to the Predominance of Forward Mode Block
11. Remodeling of T-Tubules and Reduced Synchrony of Ca 2+ Release in Myocytes From Chronically Ischemic Myocardium
12. Abstract 319: Cardiomyocyte-specific Overexpression of Type 5 Phosphodiesterase Impairs Postinfarction Myocardial Function and Left Ventricular Remodeling.
13. Early Exercise Training Normalizes Myofilament Function and Attenuates Left Ventricular Pump Dysfunction in Mice With a Large Myocardial Infarction
14. Reduced Force Generating Capacity in Myocytes From Chronically Ischemic, Hibernating Myocardium
15. Cellular Mechanisms of Contractile Dysfunction in Hibernating Myocardium
16. Spatial and Temporal Inhomogeneities During Ca 2+ Release From the Sarcoplasmic Reticulum in Pig Ventricular Myocytes
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