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Your search keyword '"Chang, Ting-Yu"' showing total 17 results
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17 results on '"Chang, Ting-Yu"'

3. Comprehensive Investigation of Constant Voltage Stress Time-Dependent Breakdown and Cycle-to-Breakdown Reliability in Y-Doped and Si-Doped HfO2 Metal-Ferroelectric-Metal Memory

Author

Chang, Ting-Yu, primary, Wang, Kuan-Chi, additional, Liu, Hsien-Yang, additional, Hseun, Jing-Hua, additional, Peng, Wei-Cheng, additional, Ronchi, Nicolò, additional, Celano, Umberto, additional, Banerjee, Kaustuv, additional, Van Houdt, Jan, additional, and Wu, Tian-Li, additional

Published
2023
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6. Head and Neck Cancer Types and Risks of Cervical–Cranial Vascular Complications within 5 Years after Radiation Therapy

Author

Liu, Chi-Hung, primary, Huang, Bing-Shen, additional, Lin, Chien-Yu, additional, Yeh, Chih-Hua, additional, Lee, Tsong-Hai, additional, Wu, Hsiu-Chuan, additional, Chang, Chien-Hung, additional, Chang, Ting-Yu, additional, Huang, Kuo-Lun, additional, Jiang, Jian-Lin, additional, Chang, Joseph Tung-Chieh, additional, and Chang, Yeu-Jhy, additional

Published
2022
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13. Whole Exome Sequencing with Comprehensive Gene Set Analysis Identified a Biparental-Origin Homozygous c.509G>A Mutation in PPIB Gene Clustered in Two Taiwanese Families Exhibiting Fetal Skeletal Dysplasia during Prenatal Ultrasound

Author

Chang, Ting-Yu, primary, Chung, I-Fang, additional, Wu, Wan-Ju, additional, Chang, Shun-Ping, additional, Lin, Wen-Hsiang, additional, Ginsberg, Norman A., additional, Ma, Gwo-Chin, additional, and Chen, Ming, additional

Published
2020
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14. A Silicon-based Coral-like Nanostructured Microfluidics to Isolate Rare Cells in Human Circulation: Validation by SK-BR-3 Cancer Cell Line and Its Utility in Circulating Fetal Nucleated Red Blood Cells

Author

Ma, Gwo-Chin, primary, Lin, Wen-Hsiang, additional, Huang, Chung-Er, additional, Chang, Ting-Yu, additional, Liu, Jia-Yun, additional, Yang, Ya-Jun, additional, Lee, Mei-Hui, additional, Wu, Wan-Ju, additional, Chang, Yun-Shiang, additional, and Chen, Ming, additional

Published
2019
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16. Comprehensive Investigation of Constant Voltage Stress Time-Dependent Breakdown and Cycle-to-Breakdown Reliability in Y-Doped and Si-Doped HfO 2 Metal-Ferroelectric-Metal Memory.

Author

Chang TY, Wang KC, Liu HY, Hseun JH, Peng WC, Ronchi N, Celano U, Banerjee K, Van Houdt J, and Wu TL

Abstract

In this study, we comprehensively investigate the constant voltage stress (CVS) time-dependent breakdown and cycle-to-breakdown while considering metal-ferroelectric-metal (MFM) memory, which has distinct domain sizes induced by different doping species, i.e., Yttrium (Y) (Sample A) and Silicon (Si) (Sample B). Firstly, Y-doped and Si-doped HfO 2 MFM devices exhibit domain sizes of 5.64 nm and 12.47 nm, respectively. Secondly, Si-doped HfO 2 MFM devices (Sample B) have better CVS time-dependent breakdown and cycle-to-breakdown stability than Y-doped HfO 2 MFM devices (Sample A). Therefore, a larger domain size showing higher extrapolated voltage under CVS time-dependent breakdown and cycle-to-breakdown evaluations was observed, indicating that the domain size crucially impacts the stability of MFM memory.

Published
2023
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17. Prenatal Diagnosis of Autosomal Recessive Renal Tubular Dysgenesis with Anhydramnios Caused by a Mutation in the AGT Gene.

Author

Ma GC, Chen YC, Wu WJ, Chang SP, Chang TY, Lin WH, and Chen M

Abstract

Autosomal recessive renal tubular dysgenesis (ARRTD) is a rare and lethal disorder that causes stillbirth or early neonatal death. Most of the reported cases are diagnosed postnatally by a histopathological hallmark of the absence or paucity of differentiated proximal tubules in kidneys. Prenatal diagnosis of ARRTD is challenging because only a few fetal features (e.g., oligohydramnios/anhydramnios, anuria) are associated with this condition. In this study, we report a fetus with ARRTD, which showed anhydramnios and invisible urinary bladder since the second trimester, followed by growth restriction and reversed end diastolic flow in the middle cerebral artery (MCA-REDF). No morphological anomaly was detected on the fetal kidneys during an ultrasound scan. The baby died of refractory hypotension the day after their birth. Genetic analysis of genes that are involved in the renin-angiotensin-aldosterone system (RAAS), which are the known genetic causes of ARRTD, identified a novel, biparental-origin homozygous c.857-619_1269+243delinsTTGCCTTGC mutation in the AGT gene. The mutation is considered as pathogenic because it is cosegregated with ARRTD and detected in other unrelated ARRTD families. Our findings link the fetal ultrasound manifestations to the ARRTD, highlighting clues that are useful for prenatal diagnosis, which warrants confirmatory genotyping of the RAAS genes including oligohydramnios/anhydramnios, anuria (absent filling of a fetal urinary bladder), MCA-REDF, and a morphologically normal kidney., Competing Interests: The authors declare no conflict of interest. The funders had no role in the design of the study; in the collection, analyses, or interpretation of the data; in the writing of the manuscript; or in the decision to publish the results.

Published
2019
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