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The article focuses on a medical report which discusses hypothalamic involvement in nonparaneoplastic, nonherpetic limbic encephalitis. Hypothermia is a life-threatening state that can be caused by several different mechanisms, such as extended exposure to a cold environment, drug overdose, sepsis, and endocrine disturbance. Here, the article report a nonherpetic nonparaneoplastic limbic encephalitis patient with a severe episodic hypothermia attack. Episodic hypothermia in this patient was thought to be due to the direct association of the central thermoregulatory center by encephalitis.

Thalamic stimulation alleviated coarse action tremor in a patient with spinocerebellar degeneration (SCD) and led to substantial improvement in daily functioning, though disability from ataxia remained. Intraoperative microrecording revealed a significant correlation between neuronal activity of the ventral intermediate nucleus (Vim) and tremor electromyograms. The tremor-correlated activities of Vim neurons and abolition of the tremor by Vim stimulation suggest that Vim may be related to the generation of action tremor in SCD. Thalamic stimulation can be a useful treatment option for SCD patients with disabling tremor. Copyright © 2005 S. Karger AG, Basel [ABSTRACT FROM AUTHOR]

The article presents a case report of a 79 year old man who complained of progressive weakness of extremities and was admitted to hospital. Neurological and electromyographical examinations revealed that he was suffering from sensorimotor polyneuropathy. His cerebrospinal fluid contained 2 cells/mm2 with a slightly elevated protein level. On admission, he was drowsy but easily irritated, could not maintain concentration. An examination of his cerebrospinal fluid showed no malignant cells and normal cell count with normal concentrations of protein and glucose. Magnetic resonance imaging showed moderate atrophy of bilateral temporal lobes. Electroencephalography was abnormal with diffuse appearance of slow irregular waves.