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Your search keyword '"Yang, Yingying"' showing total 17 results
Start Over Author "Yang, Yingying" Publisher hindawi limited
17 results on '"Yang, Yingying"'

1. The Efficacy and Safety of Hyperthermic Intravesical Chemotherapy Compared with Other Instillation Methods in Treating Intermediate- and High-Risk Non-Muscle Invasive Bladder Cancer: A Systematic Review and Meta-Analysis

Author

Yang, Yingying, primary, Liu, Hongquan, additional, Chu, Yongli, additional, Wang, Jipeng, additional, Ma, Jian, additional, Ding, Guixin, additional, Bao, Xingjun, additional, Cui, Yuanshan, additional, and Wu, Jitao, additional

Published
2024
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5. Protective Effects of the Wenfei Buqi Tongluo Formula on the Inflammation in Idiopathic Pulmonary Fibrosis through Inhibiting the TLR4/MyD88/NF-κB Pathway

Author

Song, Siyu, primary, Wang, Jing, additional, Liu, Guanwen, additional, Ding, Lu, additional, Li, Yaxin, additional, Qi, Hongyu, additional, Wei, Lai, additional, Zhao, Jiachao, additional, Chen, Tian, additional, Zhao, Meiru, additional, Wang, Ziyuan, additional, Yang, Yingying, additional, Zhao, Daqing, additional, Li, Xiangyan, additional, and Wang, Zeyu, additional

Published
2022
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16. Effect of TLR4/MyD88 Signaling Pathway on Expression of IL-1β and TNF-α in Synovial Fibroblasts from Temporomandibular Joint Exposed to Lipopolysaccharide.

Author

Lin, Xuefen, Kong, Jingjing, Wu, Qingting, Yang, Yingying, and Ji, Ping

Subjects
TOLL-like receptors, CELLULAR signal transduction, GENE expression, INTERLEUKIN-1, TUMOR necrosis factors, FIBROBLASTS, TEMPOROMANDIBULAR joint
Abstract

Accumulating evidence from previous studies suggested that interleukin-1 (IL-1β) and tumor necrosis factor-α (TNF-α) play an important role in pathogenesis of temporomandibular disorders (TMD). However, the cell surface receptors and the intracellular signal pathways leading to these cytokines expression are not fully understood. In the current study, we investigated the roles of Toll-like receptor 4 (TLR4) and its adaptor myeloid differentiation factor 88 (MyD88) in the expression of IL-1β and TNF-α in synovial fibroblasts (SFs) separated from rat temporomandibular joint (TMJ) with lipopolysaccharide (LPS) stimulation. The results showed that treatment with LPS could increase TLR4, MyD88, IL-1β, and TNF-α expression at both mRNA and protein levels. In addition, increased expression of IL-1β and TNF-α could be blocked by treatment with TAK-242, a blocker of TLR4 signaling, and also by MyD88 inhibitory peptide (MIP). These findings suggested that maybe TLR4/MyD88 signal transduction pathway participates in enhanced expression of IL-1 and TNF-α in patients with TMD. The activation of TLR4/MyD88 signal transduction pathway which results in production of proinflammatory factors may play a role in the pathogenesis of TMD. [ABSTRACT FROM AUTHOR]

Published
2015
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