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18. Persistence of Inflammatory Phenotype in Residual Psoriatic Plaques in Patients on Effective Biologic Therapy

23. Cutaneous p38 mitogen-activated protein kinase activation triggers psoriatic dermatitis

24. IL-17A inhibition by secukinumab induces early clinical, histopathologic, and molecular resolution of psoriasis

31. Dupilumab progressively improves systemic and cutaneous abnormalities in patients with atopic dermatitis

32. Baseline IL-22 expression in patients with atopic dermatitis stratifies tissue responses to fezakinumab

40. Early-onset pediatric atopic dermatitis is TH2 but also TH17 polarized in skin

42. A mild topical steroid leads to progressive anti-inflammatory effects in the skin of patients with moderate-to-severe atopic dermatitis

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