32 results on '"Costa, Robert H."'
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2. FoxM1 Regulates Transcription of JNK1 to Promote the G1/S Transition and Tumor Cell Invasiveness
3. FoxM1 Regulates Growth Factor-induced Expression of Kinase-interacting Stathmin (KIS) to Promote Cell Cycle Progression
4. Transplanted Hepatocytes Over-expressing FoxM1B Efficiently Repopulate Chronically Injured Mouse Liver Independent of Donor Age
5. The Forkhead Box M1 Transcription Factor Contributes to the Development and Growth of Mouse Colorectal Cancer
6. Increased Expression of Hepatocyte Nuclear Factor 6 Stimulates Hepatocyte Proliferation During Mouse Liver Regeneration
7. Functional Characterization of Evolutionarily Conserved DNA Regions in Forkhead Box F1 Gene Locus
8. The Forkhead Box M1 Transcription Factor Is Essential for Embryonic Development of Pulmonary Vasculature
9. Stability of the Hepatocyte Nuclear Factor 6 Transcription Factor Requires Acetylation by the CREB-binding Protein Coactivator
10. Structure of the Hepatocyte Nuclear Factor 6α and Its Interaction with DNA
11. The mouse Forkhead Box m1 transcription factor is essential for hepatoblast mitosis and development of intrahepatic bile ducts and vessels during liver morphogenesis
12. Elevated hepatocyte levels of the Forkhead box A2 (HNF-3β) transcription factor cause postnatal steatosis and mitochondrial damage
13. Foxf1 +/− mice exhibit defective stellate cell activation and abnormal liver regeneration following CCl 4 injury
14. Increased Hepatic Forkhead Box M1B (FoxM1B) Levels in Old-aged Mice Stimulated Liver Regeneration through Diminished p27Kip1 Protein Levels and Increased Cdc25B Expression
15. Haploinsufficiency of the Mouse Forkhead Box f1 Gene Causes Defects in Gall Bladder Development
16. Maintaining HNF6 expression prevents AdHNF3β-mediated decrease in hepatic levels of Glut-2 and glycogen
17. Diminished hepatic expression of the HNF-6 transcription factor during bile duct obstruction
18. Adenovirus-mediated increase in HNF-3β or HNF-3α shows differences in levels of liver glycogen and gene expression
19. Human Peroxisome Proliferator-activated Receptor α (PPARα) Supports the Induction of Peroxisome Proliferation in PPARα-deficient Mouse Liver
20. Defects in Pulmonary Vasculature and Perinatal Lung Hemorrhage in Mice Heterozygous Null for the Forkhead Box f1 Transcription Factor
21. Earlier expression of the transcription factor HFH-11B diminishes induction of p21CIP1/WAF1 levels and accelerates mouse hepatocyte entry into S-phase following carbon tetrachloride liver injury
22. HNF-3/Forkhead Homologue-4 Influences Lung Morphogenesis and Respiratory Epithelial Cell Differentiation in Vivo
23. 49 The mouse forkhead box M1B transcription factor is required for development of hepatocellular carcinomas
24. 1282 FOXF1 haploinsufficiency in mice accelerates biliary infarction and hepatic apoptosis following biliary obstruction
25. Ubiquitous expression of the forkhead box M1B transgene accelerates proliferation of distinct pulmonary cell types following lung injury. Vol. 278 (2003) 37888–37894
26. In SituHybridization with33P-Labeled RNA Probes for Determination of Cellular Expression Patterns of Liver Transcription Factors in Mouse Embryos
27. Murine chromosomal location of eight members of the hepatocyte nuclear factor 3/fork head winged helix family of transcription factors
28. The Winged Helix Transcriptional Activator HFH-3 Is Expressed in the Distal Tubules of Embryonic and Adult Mouse Kidney
29. Differential Regulation of Interleukin-8 and Intercellular Adhesion Molecule-1 by H2O2 and Tumor Necrosis Factor-α in Endothelial and Epithelial Cells
30. Members of the HNF-3/forkhead Family of Transcription Factors Exhibit Distinct Cellular Expression Patterns in Lung and Regulate the Surfactant Protein B Promoter
31. The Cut-Homeodomain Transcriptional Activator HNF-6 Is Coexpressed with Its Target Gene HNF-3β in the Developing Murine Liver and Pancreas
32. In SituHybridization with33P-Labeled RNA Probes for Determination of Cellular Expression Patterns of Liver Transcription Factors in Mouse Embryos: Volume 16, Number 1 (1998), pages 29–41
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