41 results on '"Beyreuther, Konrad"'
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2. Independent Inhibition of Alzheimer Disease β- and γ-Secretase Cleavage by Lowered Cholesterol Levels
3. Locomotor activity and evoked dopamine release are reduced in mice overexpressing A30P-mutated human α-synuclein
4. APP intracellular domain is increased and soluble Aβ is reduced with diet-induced hypercholesterolemia in a transgenic mouse model of Alzheimer disease
5. The Transmembrane Domain of the Amyloid Precursor Protein in Microsomal Membranes Is on Both Sides Shorter than Predicted
6. Detrimental effects of arachidonic acid and its metabolites in cellular and mouse models of Alzheimer's disease: structural insight
7. No alterations of hippocampal neuronal number and synaptic bouton number in a transgenic mouse model expressing the β-cleaved C-terminal APP fragment
8. Sublethal Concentrations of Prion Peptide PrP106–126 or the Amyloid Beta Peptide of Alzheimer's Disease Activates Expression of Proapoptotic Markers in Primary Cortical Neurons
9. Accumulation of Insoluble α-Synuclein in Dementia with Lewy Bodies
10. Correct heteroduplex formation for mutation detection analysis
11. Prion Protein-Deficient Neurons Reveal Lower Glutathione Reductase Activity and Increased Susceptibility to Hydrogen Peroxide Toxicity
12. Corrigendum to “Detrimental effects of arachidonic acid and its metabolites in cellular and mouse models of Alzheimer's disease: structural insight” [Neurobiol. Aging 33 (2012) 831.e21–831.e31]
13. Assembly of APP anterograde transport vesicles
14. Crystal structure of the amyloid precursor protein intracellular domain in complex with Fe65-PTB2
15. Crystal structure of the crossroad in APP transport and signaling: The Fe65-PTB1 domain
16. Alzheimer’s disease:: An inflammatory disease?
17. P3-075: Implementing Aβ ratios into the neurochemical diagnosis of early Alzheimer's disease
18. P2-031: Analysis of novel PAT1a interaction partners and their relevance for APP processing
19. P1-389: The subcellular sorting of APP in non- neuronal and neuronal cells underlies different molecular mechanisms
20. O4-02-01: Altered expression of APP gene family members in Drosophila cause defects in axonal transport and affect synaptic plasticity
21. P2-114 Cell-type specific processing of the APP protein family in drosophila
22. O4-03-07 The APP family functions in anterograde transport
23. O3-04-06 Structural and functional analysis of the Alzheimer's disease amyloid precursor protein copper binding domain
24. O2-02-03 Change in plasma amyloid concentration is associated with memory decline in MCI
25. Relationships between ganglioside GM1 and the transcription of APP gene and the metabolism of APP
26. Cerebrospinal fluid protein tau levels in the differential diagnosis of Alzheimer's disease
27. Pathogenic mechanisms underlying biogenesis of a β from amyloid precursor protein
28. Physiological function of APP processing
29. Reduced cerebrospinal fluid estradiol levels are associated with early onset and increased β-amyloid levels in Alzheimers disease
30. APP gene family: unique age-associated changes in splicing of Alzheimer's βA4-amyloid protein precursor
31. Nomenclature of amyloid A4 proteins and their precursors in Alzheimer's disease and down's syndrome
32. Determination of the N-Terminal Sequence of Human Red Cell Rh(D) Polypeptide and Demonstration That the Rh(D), (c), and (E) Antigens Are Carried by Distinct Polypeptide Chains
33. 789 Exon 15 of APP regulates βA4 production and polarized secretion of APP
34. 329 Multiple amyloidogenic pathways — where is the therapeutic target?
35. The blood-brain barrier in Alzheimer's disease and normal aging
36. The βA4 amyloid protein and its precursors as biological markers for Alzheimer's disease
37. Processing of precursors of amyloid A4 protein in Alzheimer's disease
38. Aggregation and amyloidogenicity of fragments of the amyloid A4 protein precursor of Alzheimer's disease
39. Induced neuronal expression and alternative splicing pattern of the amyloid gene of Alzheimer's disease
40. Alzheimer protein precursors: Tyrosine phosphorylation of the cytoplasmic domain
41. The abnormality of plasma amyloid protein precursor in Alzheimer's disease
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