26 results on '"Tang,Chengyuan"'
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2. Phosphorylation by GSK-3β increases the stability of SIRT6 to alleviate TGF-β-induced fibrotic response in renal tubular cells
3. Phosphorylation by GSK-3β Increases the Stability of SIRT6 to Alleviate TGF-β-Induced Fibrotic Response in Renal Tubular Cells
4. Long-Term Glucocorticoid Exposure Undermines CD4+ T-Cell Immunity Against Infection by Epigenetically Interfering with the Dynamic Activity of mTORC1 Pathway
5. Post-treatment of CsPbI3 nanocrystals by p-iodo-D-Phenylalanine for efficient perovskite LEDs
6. NAM protects against cisplatin-induced acute kidney injury by suppressing the PARP1/p53 pathway
7. Corrigendum to “Endoplasmic reticulum stress is activated in post-ischemic kidneys to promote chronic kidney disease” [EBioMedicine 37 (2018) 269–280]
8. The PINK1/PARK2/optineurin pathway of mitophagy is activated for protection in septic acute kidney injury
9. Norcantharidin protects against renal interstitial fibrosis by suppressing TWEAK-mediated Smad3 phosphorylation
10. Irisin is induced in renal ischemia-reperfusion to protect against tubular cell injury via suppressing p53
11. The deacetylase sirtuin 6 protects against kidney fibrosis by epigenetically blocking β-catenin target gene expression
12. Corrigendum to ‘Reactive oxygen species promote tubular injury in diabetic nephropathy: The role of the mitochondrial ros-txnip-nlrp3 biological axis’ [Redox Biology 16 (2018) 32–46]
13. Disulfide-bond A oxidoreductase-like protein protects against ectopic fat deposition and lipid-related kidney damage in diabetic nephropathy
14. Epigenetic Regulation in Kidney Toxicity: Insights From Cisplatin Nephrotoxicity
15. P53 in kidney injury and repair: Mechanism and therapeutic potentials
16. Endoplasmic reticulum stress is activated in post-ischemic kidneys to promote chronic kidney disease
17. Reactive oxygen species promote tubular injury in diabetic nephropathy: The role of the mitochondrial ros-txnip-nlrp3 biological axis
18. The mitochondria-targeted antioxidant MitoQ ameliorated tubular injury mediated by mitophagy in diabetic kidney disease via Nrf2/PINK1
19. DNA damage response in nephrotoxic and ischemic kidney injury
20. Mitochondrial dysfunction in obesity-related kidney disease: a novel therapeutic target
21. BNIP3 Protein Suppresses PINK1 Kinase Proteolytic Cleavage to Promote Mitophagy
22. Therapeutic potential of FGF21 in cardiorenal syndrome
23. Validation of the interstitial fibrosis and tubular atrophy on the new pathological classification in patients with diabetic nephropathy: A single-center study in China
24. Clinical and Pathological Predictors of Tubulointerstitial Injury in New Pathological Classification of Diabetic Nephropathy
25. Epigenetic regulation in acute kidney injury: new light in a dark area
26. Mitochondrial Function and Disturbances in the Septic Kidney
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