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Line segment matching is important in applications that require recovering the 3D structure of objects (e.g., manmade objects in street-level scenarios). However, differentiating between true and false line matches is generally difficult without strong geometric constraints for line segments. Hence, additional constraints are forced to be used, sacrificing many true line matches. This study proposes a robust line segment matching method based on global projective transformation modeling. We develop a non-parametric motion regression formulation with a specially designed direct linear transformation-based cost function that reformulates the piecewise smoothly varying projective transformations as a global continuous model from highly noisy point matches. The resultant model can effectively approximate the real underlying image transformation and derive high-quality point matches. We apply the computed model and high-quality point matches to a point-correspondence-based line matching pipeline, which provides sufficient strict geometric constraints for first generating the pair-to-pair matches and then distilling the line-to-line matches. Extensive experiments conducted on two challenging line matching datasets show that the proposed method can obtain considerable correct line segment matches, outperforming the comparison methods in mean F-score by at least 15.5% on the benchmark dataset and 16.9% on the local dataset. Code is available at https://github.com/geovsion/SLEM .

Acute lung injury (ALI), as a life-threatening syndrome, is mainly characterized with diffuse alveolar injury, excessive pulmonary inflammation, edema and apoptosis of lung epithelial cells. This study investigated the effects of LncRNA Hsp4 (Hsp4, ENSMUST00000175718) on lipopolysaccharide (LPS)-induced apoptosis of MLE-12 cells. In our research, we found that LPS treatment remarkably induced apoptosis of MLE-12 cells and decreased the expression of Hsp4. Overexpression of Hsp4 significantly reversed LPS-induced cell apoptosis through inhibiting mTOR signaling, while suppression of Hsp4 presented opposite effects. Further results showed that Hsp4 positively regulated the expression of miR-466m-3p. Knockdown of miR-466m-3p reversed LPS-induced cell apoptosis via increasing the levels of DNAjb6 which was confirmed to be the target gene of miR-466m-3p. This finding will be helpful for further understanding the critical roles of Hsp4 in ALI and may provide potential targets for ALI diagnosis and treatment.