22 results on '"Gorin, Yves"'
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2. Hydrogen sulfide inhibits high glucose-induced NADPH oxidase 4 expression and matrix increase by recruiting inducible nitric oxide synthase in kidney proximal tubular epithelial cells
3. Novel role of dual oxidase 2 as a mediator of podocyte injury in the diabetic environment
4. Activation of AMP-Activated Protein Kinase Prevents TGF-β1–Induced Epithelial-Mesenchymal Transition and Myofibroblast Activation
5. Tadalafil Integrates Nitric Oxide-Hydrogen Sulfide Signaling to Inhibit High Glucose-induced Matrix Protein Synthesis in Podocytes
6. Symmetric dimethylarginine alters endothelial nitric oxide activity in glomerular endothelial cells
7. Nox4 NADPH Oxidase Mediates Peroxynitrite-dependent Uncoupling of Endothelial Nitric-oxide Synthase and Fibronectin Expression in Response to Angiotensin II
8. Nox4 and diabetic nephropathy: With a friend like this, who needs enemies?
9. Nox4 as a potential therapeutic target for treatment of uremic toxicity associated to chronic kidney disease
10. Nephrogenic Systemic Fibrosis
11. Myofibroblast differentiation during fibrosis: role of NAD(P)H oxidases
12. AMP-activated Protein Kinase (AMPK) Negatively Regulates Nox4-dependent Activation of p53 and Epithelial Cell Apoptosis in Diabetes
13. The NADPH Oxidase Subunit p22 Inhibits the Function of the Tumor Suppressor Protein Tuberin
14. 84: Mechanisms of Epithelial Cells Injury in Diabetes: Role of Signaling Pathways Mediated By Nadph Oxidases
15. Nox4 NAD(P)H Oxidase Mediates Src-dependent Tyrosine Phosphorylation of PDK-1 in Response to Angiotensin II
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17. NAD(P)H Oxidases Regulate HIF-2α Protein Expression
18. PI 3 kinase-dependent Akt kinase and PKCε independently regulate interferon-γ-induced STAT1α serine phosphorylation to induce monocyte chemotactic protein-1 expression
19. Nox4 NAD(P)H Oxidase Mediates Hypertrophy and Fibronectin Expression in the Diabetic Kidney
20. Involvement of Calcineurin in Transforming Growth Factor-β-mediated Regulation of Extracellular Matrix Accumulation
21. Angiotensin II and growth factors in the pathogenesis of diabetic nephropathy
22. Inhibition of thyroid NADPH-oxidase by 2-iodohexadecanal in a cell-free system
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