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2. The affordability of lecanemab, an amyloid-targeting therapy for Alzheimer's disease: an EADC-EC viewpoint

3. BPSD Patterns in Patients With Severe Neuropsychiatric Disturbances: Insight From the RECAGE Study

5. Association of a CAMK2A genetic variant with logical memory performance and hippocampal volume in the elderly

6. Quantitative Interaction Proteomics of Neurodegenerative Disease Proteins

7. PLCG2 Protective Variant p.P522R Modulates Tau Pathology and Disease Progression in Patients with Mild Cognitive Impairment

8. 24-month intervention with a specific multinutrient in people with prodromal Alzheimer's disease (LipiDiDiet): a randomised, double-blind, controlled trial

9. The frequency and influence of dementia risk factors in prodromal Alzheimer's disease

10. The European DTI Study on Dementia — A multicenter DTI and MRI study on Alzheimer's disease and Mild Cognitive Impairment

11. Electroconvulsive therapy selectively enhances amyloid β 1–42 in the cerebrospinal fluid of patients with major depression: A prospective pilot study

12. Cerebrospinal fluid cortisol and clinical disease progression in MCI and dementia of Alzheimer's type

13. Investigation of the role of rare TREM2 variants in frontotemporal dementia subtypes

15. Measurements of medial temporal lobe atrophy for prediction of Alzheimer's disease in subjects with mild cognitive impairment

18. The association between white matter hyperintensities and executive decline in mild cognitive impairment is network dependent

19. The future of Alzheimer's disease: The next 10 years

21. Multicentre variability of MRI-based medial temporal lobe volumetry in Alzheimer's disease

23. Association of SORL1 gene variants with Alzheimer's disease

24. Safety and efficacy of galantamine (Reminyl) in severe Alzheimer's disease (the SERAD study): a randomised, placebo-controlled, double-blind trial

25. Influence of SORL1 gene variants: Association with CSF amyloid-β products in probable Alzheimer's disease

31. Multimodal imaging of residual function and compensatory resource allocation in cortical atrophy: a case study of parietal lobe function in a patient with Huntington's disease

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