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'Phantom vibration syndrome,' or perceived vibrations from a device that is not really vibrating, is a recent psychological phenomenon that has attracted the attention of the media and medical community. Most (89%) of the 290 undergraduates in our sample had experienced phantom vibrations, and they experienced them about once every two weeks, on average. However, few found them bothersome. Those higher in conscientiousness experienced phantom vibrations less frequently, and those who had strong reactions to text messages (higher in the emotional reaction subscale of text message dependence) were more bothered by phantom vibrations. These findings suggest that targeting individuals' emotional reactions to text messages might be helpful in combating the negative consequences of both text message dependency and phantom vibrations. However, because few young adults were bothered by these phantom vibrations or made attempts to stop them, interventions aimed at this population may be unnecessary.

A common procedure for studying the ability of animals to time is the peak procedure. With the peak procedure animals are first trained on a fixed interval schedule (i.e., 30 seconds). After the animals have been well trained on the fixed interval schedule, probe trials are introduced. On probe trials the stimulus is presented longer (i.e., 90 seconds) and the animal does not receive reinforcement for responding. When animals are first presented with probe trials responding remains flat following the point that reinforcement normally occurs on fixed interval trials. The descending slope that eventually emerges is acquired with experience with probe trials. The present experiments manipulated the percentage of probe trials compared to FI trials across groups of rats. It was hypothesized that the descending limb of peak responding would be acquired more quickly when there were many probe trials per session as this might facilitate extinction of responding beyond the interval that reinforcement normally occurs. It was found, however, that acquisition of peak responding occurred best when there were few probe trials per session.

In earlier research using constant-delay matching with pigeons, there is evidence that delay of reinforcement of sample-orienting behavior may contribute to the decline in matching accuracy with increasing delay between sample and comparison stimuli. In the present research using this procedure, we found that a significant decline in matching accuracy between the first and second session can occur when delays are relatively long. This effect cannot be accounted for in terms of either additional memory loss or surprise (generalization decrement) associated with the increase in delay. Furthermore, the decline in matching accuracy occurred regardless of whether the delay was inserted between samples and comparisons (where it would be expected to affect the use of sample memory in making the comparison choice response) or between comparisons and reinforcement (where it would not be expected to affect the use of sample memory in making the comparison choice response). Thus, the decrease in matching accuracy between Session 1 and 2 following an increase in delay appears to be unrelated to sample memory at the time of choice. Instead, the results suggest that delay of reinforcement of sample-orienting behavior may play an important role in the negative slope of the retention functions obtained when constant- or mixed-delay matching procedures are used to assess animal memory.

This study examined the effects of neonatal drug exposure on performance in a digging maze. Subjects were Sprague-Dawley rats, artificially reared (AR) and fed through a gastrostomy tube from postnatal days (PND) 4-10. The AR groups included a cocaine group (20 mg/kg/day cocaine hydrochloride), an ethanol group (4 g/kg/day ethanol), a cocaine/ethanol group (20 mg/kg/day cocaine and 4 g/kg/day ethanol), and an AR control group. A suckled control raised by its dam was also included. At approximately PND 55, subjects were tested in a digging maze paradigm. The digging maze required subjects to use a species typical behavior (digging) to solve a novel problem (gaining access to water). While neonatal treatment had no effect on acquisition of a simple runway task for water reward, neonatal exposure to cocaine and ethanol in combination resulted in impaired performance on the digging maze task. None of the other neonatal treatment groups showed impairments on this task. These findings suggest that exposure to these doses of cocaine and ethanol during neonatal development may have more serious effects on problem solving tasks in rats than exposure to either drug alone.

This study examined the effects of neonatal cocaine exposure on running wheel activity and subsequent responsivity to cocaine using a rodent model. Subjects were artificially reared from postnatal (PND) days 4-10 via an intragastric cannula. The four treatment groups included two cocaine doses (20 mg/kg/day and 40 mg/kg/day), an artificially reared control and a normally reared suckled control. Subjects were tested at either PND 21 through PND 24 (Experiment 1) or PND 60 through PND 70 (Experiment 2) for 2 consecutive days. Testing consisted of a 30-min habituation period followed by injection of either saline (Day 1) or cocaine (Day 2) and an additional 60-min test session. Neonatal treatment had little effect on baseline activity or activity following saline injection at either age. All subjects showed an activation with cocaine injections, however, the activation was more pronounced in juveniles. Again, neonatal treatment did not interact with response to cocaine. These findings suggest that neonatal cocaine exposure does not alter activity or long-term responsivity to 20 mg/kg cocaine as measured in the running wheel apparatus.