22 results on '"Cuezva, José M."'
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2. Metformin overcomes metabolic reprogramming-induced resistance of skin squamous cell carcinoma to photodynamic therapy
3. Metabolic reprogramming and disease progression in cancer patients
4. MYC Induces a Hybrid Energetics Program Early in Cell Reprogramming
5. Different mitochondrial genetic defects exhibit the same protein signature of metabolism in skeletal muscle of PEO and MELAS patients: A role for oxidative stress
6. Dysfunction of the immune system in conditional IF1 (Atp5if1) knockout mice in colon
7. Mitochondrial ROS Production Protects the Intestine from Inflammation through Functional M2 Macrophage Polarization
8. ATPase Inhibitory Factor 1 (IF1): A main driver of metabolic reprogramming and nuclear signaling in cancer
9. The ATPase Inhibitory Factor 1 (IF1): A master regulator of energy metabolism and of cell survival
10. PKA Phosphorylates the ATPase Inhibitory Factor 1 and Inactivates Its Capacity to Bind and Inhibit the Mitochondrial H+-ATP Synthase
11. Sensitivity to anti-Fas is independent of increased cathepsin D activity and adrenodoxin reductase expression occurring in NOS-3 overexpressing HepG2 cells
12. The H+-ATP synthase: A gate to ROS-mediated cell death or cell survival
13. miR-127-5p targets the 3′UTR of human β-F1-ATPase mRNA and inhibits its translation
14. The Mitochondrial ATPase Inhibitory Factor 1 Triggers a ROS-Mediated Retrograde Prosurvival and Proliferative Response
15. Post-transcriptional regulation of the mitochondrial H+-ATP synthase: A key regulator of the metabolic phenotype in cancer
16. Up-regulation of the ATPase Inhibitory Factor 1 (IF1) of the Mitochondrial H+-ATP Synthase in Human Tumors Mediates the Metabolic Shift of Cancer Cells to a Warburg Phenotype
17. The tumor suppressor function of mitochondria: Translation into the clinics
18. Cancer Abolishes the Tissue Type-Specific Differences in the Phenotype of Energetic Metabolism
19. Glucose avidity of carcinomas
20. Epigenetic regulation of the binding activity of translation inhibitory proteins that bind the 3′ untranslated region of β-F1-ATPase mRNA by adenine nucleotides and the redox state
21. A Conserved Mechanism for Controlling the Translation of β-F1-ATPase mRNA between the Fetal Liver and Cancer Cells
22. Changing Patterns of Transcriptional and Post-transcriptional Control of β-F1-ATPase Gene Expression during Mitochondrial Biogenesis in Liver
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