41 results on '"Chignard M"'
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2. WS10.4 Transcriptomic analysis of normal and cystic fibrosis human bronchial epithelial cells infected with Pseudomonas aeruginosa reveals distinct gene activation
3. LasB elastase, a T2SS Pseudomonas aeruginosa virulence factor kills mice in part by subverting host alveolar macrophage activity, through IL-1β down-regulation
4. Étude comparative du transcriptome de cellules épithéliales bronchiques, issues de sujets sains ou mucoviscidosiques, infectées par Pseudomonas aeruginosa
5. LasB elastase, a T2SS Pseudomonas aeruginosa virulence factor subverts host alveolar macrophage activity through IL-1β down-regulation ex vivo and in vivo
6. Étude des interactions entre cellules bronchiques et Aspergillus fumigatus
7. WS5.2 Toll-like receptor 9 deficiency protects mice against Pseudomonas aeruginosa pulmonary infection
8. WS11.3 Role of pulmonary cytosolic phospholipase A2 in mouse mortality by Pseudomonas aeruginosa infection
9. WS9.6 A role for TLR9 in Pseudomonas aeruginosa-induced lung inflammation
10. 176 A role of host cytosolic phospholipase A2 in acute lung infection by Pseudomonas aeruginosa
11. 166* Role of Pseudomonas aeruginosa flagellin in airway mucin production: relevance for cystic fibrosis disease
12. Bactericidal effect of sPLA2-IIA on antibiotics multi-resistant Pseudomonas aeruginosa
13. Trypsin and neutrophil elastase regulate CFTR expression and function in cystic fibrosis
14. Both type II (T2SS) and type III (T3SS) secretion systems of Pseudomonas aeruginosa play roles in death due to lung disease
15. Cytosolic phospholipase A2α mediates Pseudomonas aeruginosa LPS-induced airway constriction of CFTR −/− mice
16. Flagellin/TLR5-dependent modulation of alveolar macrophage and epithelial cell activity by the antimicrobial molecule trappin-2
17. Lung defense against Pseudomonas aeruginosa: the recognition by host cells of either LPS or flagellin is necessary and sufficient
18. Role of cytosolic phospholipase A2 in Pseudomonas aeruginosa-induced inflammation
19. Lack of MyD88 protects the immunodeficient host against fatal lung inflammation triggered by the opportunistic bacteria Burkholderia cenocepacia
20. Modulation of the pro-inflammatory phenotype of CFTR mutated epithelial cells
21. Lack of control of Pseudomonas aeruginosa pulmonary infection in the absence of recognition of both lipopolysaccharide and flagellin
22. Role of cytosolic phospholipase A2 in Pseudomonas aeruginosa-induced inflammation
23. Lack of control of Pseudomonas aeruginosa pulmonary infection in the absence of recognition of both lipopolysaccharide and flagellin
24. Aspergillus fumigatus-induced IL-8 synthesis by bronchial epithelial cells: lack of involvement of the TLR-MyD88 pathway
25. 097 Innate immune response to the respiratory bacteria Burkholderia cenocepacia: role of the Toll-like receptors
26. 098 The double-stranded RNA receptors TLR3 and RIG-I differentially regulate inflammatory and antiviral responses in human lung epithelial cells infected by influenza A virus
27. 084 Experimental acute lung disease due to Pseudomonas aeruginosa: analysis of the role of flagellin
28. 018 Régulation protéolytique du récepteur de l’urokinase (uPAR) dans un contexte d’inflammation pulmonaire
29. 019 Rôle des Toll-like receptors (TLR) 2 et 4 au cours d’une infection pulmonaire aiguë à Pseudomonas aeruginosa
30. 032 The Toll-like receptor (TLR)-3 médiates influenza A virus-induced acute pneumonia
31. Biosynthesis of platelet-activating factor (PAF-acether) II. Involvement of phospholipase A2 in the formation of PAF-acether and lyso-PAF-acether from rabbit platelets
32. Present concepts on the mechanisms of platelet aggregation
33. Leukocytic functions in burn-injured patients
34. Inhibition by sulfhydryl agents of arachidonic acid-induced platelet aggregation and release of potential inflammatory substances
35. Synthesis of thromboxane B2 in incubates of dog platelet-rich plasma with arachidonic acid and its inhibition by different drugs
36. Interference of transmethylation inhibitors with thromboxane synthesis in rat platelets
37. Possible participation of adenosine 5′-diphosphate, arachidonic acid and paf-acether in the platelet pro-aggregating effect of uncontrolled insulin-dependent diabetic erythrocytes in vitro
38. Leukocytic function in burn-injured patients
39. 3 deazaadenosine and L-homocysteine inhibit platelet aggregation induced by collagen and convulxin through a phospholipase A2 independent mechanism
40. Inhibitory effects of three new synthetic compounds on human platelet aggregation
41. Inhibition by ticlopidine of Paf-acether-induced in vitro aggregation of rabbit and human platelets
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