63 results on '"Brown, Joan Heller"'
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2. RhoA regulates Drp1 mediated mitochondrial fission through ROCK to protect cardiomyocytes
3. CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α
4. Selective coupling of the S1P 3 receptor subtype to S1P-mediated RhoA activation and cardioprotection
5. Sphingosine 1-phosphate elicits RhoA-dependent proliferation and MRTF-A mediated gene induction in CPCs
6. Reductions in the Cardiac Transient Outward K+ Current Ito Caused by Chronic β-Adrenergic Receptor Stimulation Are Partly Rescued by Inhibition of Nuclear Factor κB
7. Thrombin Promotes Sustained Signaling and Inflammatory Gene Expression through the CDC25 and Ras-associating Domains of Phospholipase Cϵ
8. CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca2+ leak and the pathophysiological response to chronic β-adrenergic stimulation
9. PLCε mediated sustained signaling pathways
10. Induction of the matricellular protein CCN1 through RhoA and MRTF-A contributes to ischemic cardioprotection
11. CaMKII-dependent phosphorylation of cardiac ryanodine receptors regulates cell death in cardiac ischemia/reperfusion injury
12. The Ras-related Protein, Rap1A, Mediates Thrombin-stimulated, Integrin-dependent Glioblastoma Cell Proliferation and Tumor Growth
13. Functional Modulation of Cardiac ATP-Sensitive Potassium Channels by Nitric Oxide via Intracellular Signaling
14. Lysophospholipid receptor activation of RhoA and lipid signaling pathways
15. Role of phospholipase Cε in physiological phosphoinositide signaling networks
16. CaMKIIδC Slows [Ca]i Decline in Cardiac Myocytes by Promoting Ca Sparks
17. CaMKII in myocardial hypertrophy and heart failure
18. Crossing signals: relationships between β-adrenergic stimulation and CaMKII activation
19. Overexpression of CaMKIIδc in RyR2R4496C+/− Knock-In Mice Leads to Altered Intracellular Ca2+ Handling and Increased Mortality
20. CaM Kinase IIδ Is Required for Catecholamine-Induced Cardiac Fibrosis but Not Hypertrophy
21. β-Adrenergic receptor signaling in the heart: Role of CaMKII
22. β-Adrenergic receptor stimulated Ncx1 upregulation is mediated via a CaMKII/AP-1 signaling pathway in adult cardiomyocytes
23. Akt Increases Sarcoplasmic Reticulum Ca2+ Cycling by Direct Phosphorylation of Phospholamban at Thr17
24. Cardioprotective stimuli mediate phosphoinositide 3-kinase and phosphoinositide dependent kinase 1 nuclear accumulation in cardiomyocytes
25. Focal Adhesion Kinase as a RhoA-activable Signaling Scaffold Mediating Akt Activation and Cardiomyocyte Protection
26. Pulsatile equibiaxial stretch inhibits thrombin-induced RhoA and NF-κB activation
27. S1P1 Receptor Localization Confers Selectivity for Gi-mediated cAMP and Contractile Responses
28. CaMKIIδ Isoforms Differentially Affect Calcium Handling but Similarly Regulate HDAC/MEF2 Transcriptional Responses
29. Constitutive Activation of CaMKII Reduces Cardiac Function and Viability in Feline Ventricular Myocytes
30. Reduced contractile response to α1-adrenergic stimulation in atria from mice with chronic cardiac CaMKII inhibition
31. RhoA signaling in cardiomyocyte survival and apoptosis
32. Akt mediates cardioprotection through phosphorylation of mitochondrial hexokinase-II
33. RhoA/Rho Kinase Up-regulate Bax to Activate a Mitochondrial Death Pathway and Induce Cardiomyocyte Apoptosis
34. Tumor Necrosis Factor-α-stimulated Cell Proliferation Is Mediated through Sphingosine Kinase-dependent Akt Activation and Cyclin D Expression
35. Gαq expression activates EGFR and induces Akt mediated cardiomyocyte survival: dissociation from Gαq mediated hypertrophy
36. Ca2+ Dysregulation Induces Mitochondrial Depolarization and Apoptosis
37. Akt-mediated Cardiomyocyte Survival Pathways Are Compromised by Gαq-induced Phosphoinositide 4,5-Bisphosphate Depletion
38. Initiation and Transduction of Stretch-induced RhoA and Rac1 Activation through Caveolae
39. Marked Perinatal Lethality and Cellular Signaling Deficits in Mice Null for the Two Sphingosine 1-Phosphate (S1P) Receptors, S1P2/LPB2/EDG-5 and S1P3/LPB3/EDG-3
40. Inositol Polyphosphate 1-Phosphatase Is a Novel Antihypertrophic Factor
41. The Cardiac-specific Nuclear δB Isoform of Ca2+/Calmodulin-dependent Protein Kinase II Induces Hypertrophy and Dilated Cardiomyopathy Associated with Increased Protein Phosphatase 2A Activity
42. Selective Loss of Sphingosine 1-Phosphate Signaling with No Obvious Phenotypic Abnormality in Mice Lacking Its G Protein-coupled Receptor, LPB3/EDG-3
43. Agonist-induced GLUT1 expression and glucose uptake in neonatal rat cardiomyocytes: Dissociation from hypertrophy
44. Physical and Functional Interactions of Gαq with Rho and Its Exchange Factors
45. A Rho Exchange Factor Mediates Thrombin and Gα12-induced Cytoskeletal Responses
46. Cardiac hypertrophy induced by mitogen-activated protein kinase kinase 7, a specific activator for c-Jun NH2-terminal kinase in ventricular muscle cells.
47. Requirement for Rho-mediated Myosin Light Chain Phosphorylation in Thrombin-stimulated Cell Rounding and Its Dissociation from Mitogenesis
48. Cardiac Hypertrophy Induced by Mitogen-activated Protein Kinase Kinase 7, a Specific Activator for c-Jun NH2-terminal Kinase in Ventricular Muscle Cells
49. The Low Molecular Weight GTPase Rho Regulates Myofibril Formation and Organization in Neonatal Rat Ventricular Myocytes
50. Cardiac Muscle Cell Hypertrophy and Apoptosis Induced by Distinct Members of the p38 Mitogen-activated Protein Kinase Family
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