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1. DNA polymerase θ protects leukemia cells from metabolically induced DNA damage.

2. Tyrosine kinase inhibitor-induced defects in DNA repair sensitize FLT3(ITD)-positive leukemia cells to PARP1 inhibitors.

3. Ruxolitinib-induced defects in DNA repair cause sensitivity to PARP inhibitors in myeloproliferative neoplasms.

4. Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases.

5. Small-Molecule Disruption of RAD52 Rings as a Mechanism for Precision Medicine in BRCA-Deficient Cancers.

6. Personalized synthetic lethality induced by targeting RAD52 in leukemias identified by gene mutation and expression profile.

7. Genomic instability may originate from imatinib-refractory chronic myeloid leukemia stem cells.

8. Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors.

9. BCR-ABL1 kinase: hunting an elusive target with new weapons.

10. Targeting RAD51 phosphotyrosine-315 to prevent unfaithful recombination repair in BCR-ABL1 leukemia.

11. STI571 reduces NER activity in BCR/ABL-expressing cells.

12. Enhanced phosphorylation of Nbs1, a member of DNA repair/checkpoint complex Mre11-RAD50-Nbs1, can be targeted to increase the efficacy of imatinib mesylate against BCR/ABL-positive leukemia cells.

13. BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistance.

14. BCR/ABL modifies the kinetics and fidelity of DNA double-strand breaks repair in hematopoietic cells.

15. Imatinib mesylate (STI571) abrogates the resistance to doxorubicin in human K562 chronic myeloid leukemia cells by inhibition of BCR/ABL kinase-mediated DNA repair.

16. BCR/ABL oncogenic kinase promotes unfaithful repair of the reactive oxygen species-dependent DNA double-strand breaks.

17. p210 BCR/ABL kinase regulates nucleotide excision repair (NER) and resistance to UV radiation.

18. Complementary functions of the antiapoptotic protein A1 and serine/threonine kinase pim-1 in the BCR/ABL-mediated leukemogenesis.

19. Role of p53 in hematopoietic recovery after cytotoxic treatment.

20. The SH3 domain contributes to BCR/ABL-dependent leukemogenesis in vivo: role in adhesion, invasion, and homing.

21. Antisense oligodeoxynucleotide combination therapy of primary chronic myelogenous leukemia blast crisis in SCID mice.

22. Phosphatidylinositol-3 kinase activity is regulated by BCR/ABL and is required for the growth of Philadelphia chromosome-positive cells.

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