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Static and stationary regular black holes are examined under a minimal set of requirements consisting of (i) the existence of a well defined event horizon and (ii) the weak energy condition for matter sourcing the geometry. We perform our analysis by means of the gravitational decoupling approach and find hairy solutions free of curvature singularities. We identify the matter source producing a deformation of the Minkowski vacuum such that the maximum deformation is the Schwarzschild solution for the static case, and the Kerr metric for the stationary case.

We consider the quantum description of a toy model universe in which the Schwarzschild-de Sitter geometry emerges from the coherent state of a massless scalar field. Although highly idealised, this simple model allows us to find clear hints supporting the conclusion that the reaction of the de Sitter background to the presence of matter sources induces i) a modified Newtonian dynamics at galactic scales and ii) different values measured for the present Hubble parameter. Both effects stem from the conditions required to have a normalisable quantum state.

We try to shed some light on the role of matter in the final stages of black hole evaporation from the fundamental frameworks of classicalization and the black-to-white hole bouncing scenario. Despite being based on very different grounds, these two approaches attempt at going beyond the background field method and treat black holes as fully quantum systems rather than considering quantum field theory on the corresponding classical manifolds. They also lead to the common prediction that the semiclassical description of black hole evaporation should break down and the system be disrupted by internal quantum pressure, but they both arrive at this conclusion neglecting the matter that formed the black hole. We instead estimate this pressure from the bootstrapped description of black holes, which allows us to express the total Arnowitt–Deser–Misner mass in terms of the baryonic mass still present inside the black hole. We conclude that, although these two scenarios provide qualitatively similar predictions for the final stages, the corpuscular model does not seem to suggest any sizeable deviation from the semiclassical time scale at which the disruption should occur, unlike the black-to-white hole bouncing scenario. This, in turn, makes the phenomenology of corpuscular black holes more subtle from an astrophysical perspective.

We consider the effects of gravitons in the collapse of baryonic matter that forms a black hole. We first note that the effective number of (soft off-shell) gravitons that account for the (negative) Newtonian potential energy generated by the baryons is conserved and always in agreement with Bekenstein's area law of black holes. Moreover, their (positive) interaction energy reproduces the expected post-Newtonian correction and becomes of the order of the total ADM mass of the system when the size of the collapsing object approaches its gravitational radius. This result supports a scenario in which the gravitational collapse of regular baryonic matter produces a corpuscular black hole without central singularity, in which both gravitons and baryons are marginally bound and form a Bose–Einstein condensate at the critical point. The Hawking emission of baryons and gravitons is then described by the quantum depletion of the condensate and we show the two energy fluxes are comparable, albeit negligibly small on astrophysical scales. Keywords: Gravitational collapse, Black holes, No singularity

Summary: Obesity is characterized by the accumulation of T cells in insulin-sensitive tissues, including the visceral adipose tissue (VAT), that can interfere with the insulin signaling pathway eventually leading to insulin resistance (IR) and type 2 diabetes. Here, we found that PD-1+CD4 conventional T (Tconv) cells, endowed with a transcriptomic and functional profile of partially dysfunctional cells, are diminished in VAT of obese patients with dysglycemia (OB-Dys), without a concomitant increase in apoptosis. These cells showed enhanced capacity to recirculate into the bloodstream and had a non-restricted TCRβ repertoire divergent from that of normoglycemic obese and lean individuals. PD-1+CD4 Tconv were reduced in the circulation of OB-Dys, exhibited an altered migration potential, and were detected in the liver of patients with non-alcoholic steatohepatitis. The findings suggest a potential role for partially dysfunctional PD-1+CD4 Tconv cells as inter-organ mediators of IR in obese patients with dysglycemic.