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2. [SRSF3: from mRNA decay to mRNA nuclear export. Epstein-Barr virus helps to make a choice].

Author

Gruffat H, Mure F, and Manet E

Subjects
Active Transport, Cell Nucleus genetics, Cell Nucleus genetics, Cell Nucleus virology, Epstein-Barr Virus Infections genetics, Epstein-Barr Virus Infections metabolism, Humans, RNA Splicing physiology, Cell Nucleus metabolism, Herpesvirus 4, Human physiology, RNA Stability genetics, RNA, Messenger metabolism, Serine-Arginine Splicing Factors physiology
Published
2019
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3. [EBV/KSHV co-infection: an effective partnership].

Author

Gruffat H and Manet E

Subjects
Animals, Epstein-Barr Virus Infections complications, Herpesviridae Infections complications, Herpesviridae Infections virology, Herpesvirus 4, Human pathogenicity, Herpesvirus 8, Human pathogenicity, Humans, Mice, Sarcoma, Kaposi complications, Coinfection virology, Epstein-Barr Virus Infections virology, Herpesvirus 4, Human physiology, Herpesvirus 8, Human physiology, Sarcoma, Kaposi virology
Abstract

Kaposi's sarcoma human herpesvirus (KSHV) is the etiologic agent of primary effusion lymphoma (PEL) in which Epstein-Barr virus (EBV) is also very often present. By using a humanized mouse model, Pr. Münz's team has been able to demonstrate that EBV/KSHV co-infection increases KSHV persistence and cell transformation through the stimulation of EBV replication. This is the first model of PEL in small animals, opening up exciting prospects for future studies on this unique lymphoma., (© 2018 médecine/sciences – Inserm.)

Published
2018
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