16 results on '"Wang, Xiaoquan"'
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2. Influenza virus infection activates TAK1 to suppress RIPK3-independent apoptosis and RIPK1-dependent necroptosis
3. Amino acid mutations PB1-V719M and PA-N444D combined with PB2-627K contribute to the pathogenicity of H7N9 in mice
4. The haemagglutinin–neuraminidase protein of velogenic Newcastle disease virus enhances viral infection through NF-κB-mediated programmed cell death
5. G1-like M and PB2 genes are preferentially incorporated into H7N9 progeny virions during genetic reassortment
6. N-linked glycosylation at site 158 of the HA protein of H5N6 highly pathogenic avian influenza virus is important for viral biological properties and host immune responses
7. Molecular identification and characterization of Anaplasma capra and Anaplasma platys-like in Rhipicephalus microplus in Ankang, Northwest China
8. Current situation of H9N2 subtype avian influenza in China.
9. The T160A hemagglutinin substitution affects not only receptor binding property but also transmissibility of H5N1 clade 2.3.4 avian influenza virus in guinea pigs.
10. Characterization of virulent Newcastle disease viruses from vaccinated chicken flocks in Eastern China.
11. The M, F and HN genes of genotype VIId Newcastle disease virus are associated with the severe pathological changes in the spleen of chickens.
12. Novel H5 clade 2.3.4.6 viruses with both α-2,3 and α-2,6 receptor binding properties may pose a pandemic threat.
13. Surveillance of avirulent Newcastle disease viruses at live bird markets in Eastern China during 2008-2012 reveals a new sub-genotype of class I virus.
14. Strong innate immune response and cell death in chicken splenocytes infected with genotype VIId Newcastle disease virus.
15. Lack of detection of host associated differences in Newcastle disease viruses of genotype VIId isolated from chickens and geese.
16. Molecular evolution of the H6 subtype influenza A viruses from poultry in eastern China from 2002 to 2010.
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