Search

We study the electric field around a continuous one-dimensional loop of static charge, under the assumption that the charge is distributed uniformly along the loop. For rectangular or stadium-shaped loops in the plane, we find that the electric field can undergo a symmetry-breaking pitchfork bifurcation as the loop is elongated; the field can have either one or three zeros, depending on the loop's aspect ratio. For knotted charge distributions in three-dimensional space, we compute the electric field numerically and compare our results to previously published theoretical bounds on the number of equilibrium points around charged knots. Our computations reveal that the previous bounds are far from sharp. The numerics also suggest conjectures for the actual minimum number of equilibrium points for all charged knots with five or fewer crossings. In addition, we provide the first images of the equipotential surfaces around charged knots and visualize their topological transitions as the level of the potential is varied.

Contagion, broadly construed, refers to anything that can spread infectiously from peer to peer. Examples include communicable diseases, rumors, misinformation, ideas, innovations, bank failures, and electrical blackouts. Sometimes, as in the 1918 Spanish flu epidemic, a contagion mutates at some point as it spreads through a network. Here, using a simple susceptible-infected model of contagion, we explore the downstream impact of a single mutation event. Assuming that this mutation occurs at a random node in the contact network, we calculate the distribution of the number of “descendants,” d, downstream from the initial “patient zero” mutant. We find that the tail of the distribution decays as d^{−2} for complete graphs, random graphs, small-world networks, networks with block-like structure, and other infinite-dimensional networks. This prediction agrees with the observed statistics of memes propagating and mutating on Facebook and is expected to hold for other effectively infinite-dimensional networks, such as the global human contact network. In a wider context, our approach suggests a possible starting point for a mesoscopic theory of contagion. Such a theory would focus on the paths traced by a spreading contagion, thereby furnishing an intermediate level of description between that of individual nodes and the total infected population. We anticipate that contagion pathways will hold valuable lessons, given their role as the conduits through which single mutations, innovations, or failures can sweep through a network as a whole.