126 results on '"Zhang, Xu-dong"'
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2. Abstract 3687: Disruption of nucleotide homeostasis confers cancer cell susceptibility to oxidative phosphorylation inhibition independently of energy depletion
3. Supplementary Figure 3 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
4. Supplementary Figure 1 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
5. Supplementary Figure 1 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
6. Data from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
7. Supplementary Figure 2 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
8. Supplementary Figures S1-S13; Supplementary Tables S1-S3, Supplementary Materials and Methods and Supplementary References from Inhibition of HSP90 by AUY922 Preferentially Kills Mutant KRAS Colon Cancer Cells by Activating Bim through ER Stress
9. Supplementary Figure 6 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
10. Supplementary Figure Legend from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
11. Supplementary Figure Legend from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
12. Supplementary Figure 5 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
13. Supplementary Figure 4 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
14. Supplementary Figure 3 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
15. Supplementary Figure 4 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
16. Supplementary Figure 6 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
17. Supplementary Figure 5 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
18. Supplementary Figure 2 from Antiproliferative Effects of Continued Mitogen-Activated Protein Kinase Pathway Inhibition following Acquired Resistance to BRAF and/or MEK Inhibition in Melanoma
19. Data from A p53-Responsive miRNA Network Promotes Cancer Cell Quiescence
20. Supplementary Table S1 (Excel file) from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
21. Data from A p53-Responsive miRNA Network Promotes Cancer Cell Quiescence
22. Supplementary Materials and Methods, Figures, Figure Legends - Revised from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
23. Supplementary Figure 6 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
24. Supplementary Table S2 from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
25. Data from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
26. Supplementary Information from A p53-Responsive miRNA Network Promotes Cancer Cell Quiescence
27. Data from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
28. Supplementary Figures from A p53-Responsive miRNA Network Promotes Cancer Cell Quiescence
29. Supplementary Materials and Methods, Figures, Figure Legends - Revised from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
30. Table S1-S5 from Skp2-Mediated Stabilization of MTH1 Promotes Survival of Melanoma Cells upon Oxidative Stress
31. Data from Skp2-Mediated Stabilization of MTH1 Promotes Survival of Melanoma Cells upon Oxidative Stress
32. Supplementary Table S1 (Excel file) from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
33. Supplementary Table S2 from The Histone Methyltransferase DOT1L Promotes Neuroblastoma by Regulating Gene Transcription
34. Supplementary Table S1 from Small Molecular Weight Variants of p53 Are Expressed in Human Melanoma Cells and Are Induced by the DNA-Damaging Agent Cisplatin
35. Supplementary Figure 1 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
36. Supplementary Table S1 from Small Molecular Weight Variants of p53 Are Expressed in Human Melanoma Cells and Are Induced by the DNA-Damaging Agent Cisplatin
37. Figure S1-S7 from Skp2-Mediated Stabilization of MTH1 Promotes Survival of Melanoma Cells upon Oxidative Stress
38. Supplementary Table 2 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
39. Supplementary Figures from A p53-Responsive miRNA Network Promotes Cancer Cell Quiescence
40. Supplementary Figure 2 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
41. Supplementary Figure 2 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
42. Supplementary Figure 4 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
43. Supplementary Figure 3 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
44. Supplementary Information from A p53-Responsive miRNA Network Promotes Cancer Cell Quiescence
45. Supplementary Figure 1 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
46. Figure S1-S7 from Skp2-Mediated Stabilization of MTH1 Promotes Survival of Melanoma Cells upon Oxidative Stress
47. Supplementary Figure 4 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
48. Supplementary Table 1 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
49. Supplementary Figure 5 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
50. Supplementary Figure 6 from Modulation of NOXA and MCL-1 as a Strategy for Sensitizing Melanoma Cells to the BH3-Mimetic ABT-737
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