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1. Supplementary Tables from HER2 Reactivation through Acquisition of the HER2 L755S Mutation as a Mechanism of Acquired Resistance to HER2-targeted Therapy in HER2+ Breast Cancer

2. Supplementary information from HER2 Reactivation through Acquisition of the HER2 L755S Mutation as a Mechanism of Acquired Resistance to HER2-targeted Therapy in HER2+ Breast Cancer

3. Tables S1-S6 from Engineering and Functional Characterization of Fusion Genes Identifies Novel Oncogenic Drivers of Cancer

4. Data from Engineering and Functional Characterization of Fusion Genes Identifies Novel Oncogenic Drivers of Cancer

5. Figures S1-S4 from Engineering and Functional Characterization of Fusion Genes Identifies Novel Oncogenic Drivers of Cancer

6. Figures S1-S4 from Engineering and Functional Characterization of Fusion Genes Identifies Novel Oncogenic Drivers of Cancer

7. Supplementary Figures from HER2 Reactivation through Acquisition of the HER2 L755S Mutation as a Mechanism of Acquired Resistance to HER2-targeted Therapy in HER2+ Breast Cancer

8. Data from Engineering and Functional Characterization of Fusion Genes Identifies Novel Oncogenic Drivers of Cancer

9. Data from Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

10. Data from Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

11. Data from Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

12. Supplementary Tables S1-S5 from Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

13. supplementary material and method from Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

14. supplementary Figures S1-S10 from Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

15. Data from Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

16. supplementary Figures S1-S10 from Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

17. Supplementary Tables S1-S5 from Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

18. Supplementary Figures S1-S2 from Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

19. supplementary material and method from Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

20. Supplementary Figures S1-S2 from Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

21. POT1 Regulates Proliferation and Confers Sexual Dimorphism in Glioma

22. HER2 Reactivation through Acquisition of the HER2 L755S Mutation as a Mechanism of Acquired Resistance to HER2-targeted Therapy in HER2+ Breast Cancer

23. Engineering and Functional Characterization of Fusion Genes Identifies Novel Oncogenic Drivers of Cancer

25. Identification of Variant-Specific Functions of PIK3CA by Rapid Phenotyping of Rare Mutations

28. Genetic Events That Limit the Efficacy of MEK and RTK Inhibitor Therapies in a Mouse Model of KRAS-Driven Pancreatic Cancer

29. Abstract C199: Coordinate phosphorylation of multiple residues on single AKT1 and AKT2 molecules.

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