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1. Data from Tumor Mutation Burden and Structural Chromosomal Aberrations Are Not Associated with T-cell Density or Patient Survival in Acral, Mucosal, and Cutaneous Melanomas

2. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

3. Supplementary Figure Legends from Tumor Mutation Burden and Structural Chromosomal Aberrations Are Not Associated with T-cell Density or Patient Survival in Acral, Mucosal, and Cutaneous Melanomas

4. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

5. Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

6. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

7. Data from Tumor Mutation Burden and Structural Chromosomal Aberrations Are Not Associated with T-cell Density or Patient Survival in Acral, Mucosal, and Cutaneous Melanomas

8. Supplementary Figures and Tables from Tumor Mutation Burden and Structural Chromosomal Aberrations Are Not Associated with T-cell Density or Patient Survival in Acral, Mucosal, and Cutaneous Melanomas

9. Supplementary Figures and Tables from Tumor Mutation Burden and Structural Chromosomal Aberrations Are Not Associated with T-cell Density or Patient Survival in Acral, Mucosal, and Cutaneous Melanomas

10. Supplementary Figure Legends from Tumor Mutation Burden and Structural Chromosomal Aberrations Are Not Associated with T-cell Density or Patient Survival in Acral, Mucosal, and Cutaneous Melanomas

11. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

12. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

13. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

14. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

15. Supplementary Data from Comparative Genomics Provides Etiologic and Biological Insight into Melanoma Subtypes

17. Supplementary Table 1 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

18. Supplementary Table 2 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

19. Supplementary Table 2 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

20. Data from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

22. Data from Loss-of-Function Fibroblast Growth Factor Receptor-2 Mutations in Melanoma

23. Supplementary Table 3 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

24. Supplementary Data from Loss-of-Function Fibroblast Growth Factor Receptor-2 Mutations in Melanoma

26. Supplementary Table 3 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

28. Supplementary Table 4 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

35. Supplementary Data from Loss-of-Function Fibroblast Growth Factor Receptor-2 Mutations in Melanoma

36. Supplementary Table 1 from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

37. Data from Loss-of-Function Fibroblast Growth Factor Receptor-2 Mutations in Melanoma

39. Data from A High-Throughput Panel for Identifying Clinically Relevant Mutation Profiles in Melanoma

40. Data from Accuracy of Self-Reported Nevus and Pigmentation Phenotype Compared with Clinical Assessment in a Population-Based Study of Young Australian Adults

41. Supplementary Figure Legend from BRAF/NRAS Wild-Type Melanomas Have a High Mutation Load Correlating with Histologic and Molecular Signatures of UV Damage

42. Figure S5 from PD-L1 Negative Status is Associated with Lower Mutation Burden, Differential Expression of Immune-Related Genes, and Worse Survival in Stage III Melanoma

43. Supplementary materials (clean version) from A Pilot Randomized Controlled Trial of the Feasibility, Acceptability, and Impact of Giving Information on Personalized Genomic Risk of Melanoma to the Public

44. Supplementary Figure S1 from Accuracy of Self-Reported Nevus and Pigmentation Phenotype Compared with Clinical Assessment in a Population-Based Study of Young Australian Adults

45. Supplementary Table 2 from BRAF/NRAS Wild-Type Melanomas Have a High Mutation Load Correlating with Histologic and Molecular Signatures of UV Damage

46. Supplementary Data from Distinct Molecular Profiles and Immunotherapy Treatment Outcomes of V600E and V600K BRAF-Mutant Melanoma

47. Supplementary Table S1 from Accuracy of Self-Reported Nevus and Pigmentation Phenotype Compared with Clinical Assessment in a Population-Based Study of Young Australian Adults

48. Data from Accuracy of Self-Reported Nevus and Pigmentation Phenotype Compared with Clinical Assessment in a Population-Based Study of Young Australian Adults

49. Personalised risk booklet - an example from A Pilot Randomized Controlled Trial of the Feasibility, Acceptability, and Impact of Giving Information on Personalized Genomic Risk of Melanoma to the Public

50. Supplementary materials (clean version) from A Pilot Randomized Controlled Trial of the Feasibility, Acceptability, and Impact of Giving Information on Personalized Genomic Risk of Melanoma to the Public

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