43 results on '"LUO Ping"'
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2. Figure S3 from Early Cancer Detection in Li–Fraumeni Syndrome with Cell-Free DNA
3. Early Cancer Detection in Li-Fraumeni Syndrome with Cell-Free DNA
4. Supplementary Data S3 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
5. FIGURE 2 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
6. FIGURE 3 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
7. TABLE 1 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
8. TABLE 2 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
9. FIGURE 1 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
10. FIGURE 5 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
11. FIGURE 4 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
12. Supplementary Figures S1-S12 from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
13. Data from Multiple Germline Events Contribute to Cancer Development in Patients with Li-Fraumeni Syndrome
14. Table TS6 from Integrated, Longitudinal Analysis of Cell-free DNA in Uveal Melanoma
15. Figure S1 from Integrated, Longitudinal Analysis of Cell-free DNA in Uveal Melanoma
16. Data from Integrated, Longitudinal Analysis of Cell-free DNA in Uveal Melanoma
17. Data from L-Plastin Promotes Gastric Cancer Growth and Metastasis in a Helicobacter pylori cagA-ERK-SP1–Dependent Manner
18. Supplementary Figures 1-6 from L-Plastin Promotes Gastric Cancer Growth and Metastasis in a Helicobacter pylori cagA-ERK-SP1–Dependent Manner
19. Supplementary Tables 1 and 2 from L-Plastin Promotes Gastric Cancer Growth and Metastasis in a Helicobacter pylori cagA-ERK-SP1–Dependent Manner
20. Multiple germline events contribute to cancer development in patients with Li-Fraumeni syndrome
21. Data from Hypoxia Drives Breast Tumor Malignancy through a TET–TNFα–p38–MAPK Signaling Axis
22. Table S2 from Hypoxia Drives Breast Tumor Malignancy through a TET–TNFα–p38–MAPK Signaling Axis
23. Supplementary Figure 2 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
24. Data from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
25. Supplementary Figure 4 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
26. Supplementary Figures S1-19 and legends from Hypoxia Drives Breast Tumor Malignancy through a TET–TNFα–p38–MAPK Signaling Axis
27. Supplementary Figure 3 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
28. Supplementary Table 1 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
29. Supplementary Table 2 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
30. Integrated, Longitudinal Analysis of Cell-Free DNA in Uveal Melanoma
31. Supplementary Figure 1 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
32. Table S3 from Hypoxia Drives Breast Tumor Malignancy through a TET–TNFα–p38–MAPK Signaling Axis
33. Supplementary Materials and Methods from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
34. Supplementary Figure 3 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
35. Supplementary Table 2 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
36. Supplementary Figure 2 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
37. Supplementary Figure 4 from Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
38. Supplementary Figures S1-19 and legends from Hypoxia Drives Breast Tumor Malignancy through a TET–TNFα–p38–MAPK Signaling Axis
39. L-Plastin Promotes Gastric Cancer Growth and Metastasis in a Helicobacter pylori cagA-ERK-SP1–Dependent Manner
40. Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
41. Hypoxia Drives Breast Tumor Malignancy through a TET–TNFα–p38–MAPK Signaling Axis
42. Carboxyl-Terminal Modulator Protein Positively Regulates Akt Phosphorylation and Acts as an Oncogenic Driver in Breast Cancer
43. A Mimic of Tumor Rejection Antigen-Associated Carbohydrates Mediates an Antitumor Cellular Response
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