15 results on '"Simon Laban"'
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2. Hif-1α-abhängige Expression des Adenosinrezeptors 2B (ADORA2B) fördert das autonome Wachstum, EMT sowie CSC-Anreicherung in Zellkulturen, die von Kopf-Hals- Plattenepithelkarzinomen abgeleitet sind
3. Abundance of immune checkpoint molecules and their ligands in Oropharyngeal Squamous cell carcinoma (OPSCC) depends on HPV status and can be modulated by demethylation
4. Charakterisierung von zirkulierenden Exosomen als Biomarker für die Therapieüberwachung in Patienten mit Kopf-Hals-Tumor
5. Influence of the clinical care of head and neck cancer patients by the distance to the clinical center
6. Die Aktivität des Adenosinrezeptors A2B fördert autonomes Wachstum, Migration und Vaskularisation von Kopf-Hals-Karzinom-Zellen
7. Hif-1α-dependent expression of Adenosine Receptor 2B (ADORA2B) promotes autonomous growth, EMT as well as CSC enrichment in cultures of cells derived from head and neck squamous cell carcinomas
8. First-line treatment of locally advanced HNSCC with double checkpoint inhibition and radiotherapy based on CD8+ T cell infiltration
9. Cervical adipocytes and tumor cells interact in patients with head and neck cancer
10. Characterization of adenosine receptor A2A (ADORA2A) mediated effects on the immunogenic tumor microenvironment (TME) in a head and neck squamous cell carcinoma (HNSCC) mouse model
11. Changes of immune checkpoint expression under conventional radiochemotherapy of head and neck carcinomas
12. HLA-Allele-frequencies differ between patiens with head neck cancer (HNSCC) and healthy controls and between HPV-positiv and HPV-negative HNSCC patients
13. Adenosine receptor 2B activity promotes autonomous growth, migration as well as vascularization of head and neck squamous cell carcinoma cells
14. Erstlinientherapie fortgeschrittener Kopf-Hals-Karzinome mit Doppel-Checkpoint-Blockade und Radiotherapie in Abhängigkeit der CD8+-T-Zellinfiltration
15. Die Expression von Immun-Checkpoint Molekülen und Liganden in Oropharyngealen Plattenepithelkarzinomen (OPSCC) hängt vom HPV Status ab und wird durch Demethylierung moduliert
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