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Your search keyword '"Scemes, Eliana"' showing total 6 results
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6 results on '"Scemes, Eliana"'

2. Resident Neural Stem Cells

Author

Zaverucha-do-Valle, Camila, primary, Gubert, Fernanda, additional, Vasques, Juliana Ferreira, additional, Mesentier-Louro, Louise, additional, Scemes, Eliana, additional, Pitossi, Fernando, additional, Santiago, Marcelo Fellipe, additional, and Mendez-Otero, Rosalia, additional

Published
2024
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3. List of contributors

Author

Antunes, Mariana Alves, primary, Baptista, Leandra Santos, additional, Borges, Luciano Figueiredo, additional, Calloni, Giordano Wosgrau, additional, Caplan, Arnold I., additional, Carvalho, Adriana Bastos, additional, Collino, Federica, additional, Costa, Milene R., additional, Cruz, Fernanda Ferreira, additional, da Silva Meirelles, Lindolfo, additional, de Carvalho, Antonio Carlos Campos, additional, Dias, Marlon Lemos, additional, dos Santos Goldenberg, Regina Coeli, additional, Duarte, Maria Eugenia Leite, additional, Einicker-Lamas, Marcelo, additional, Faccioli, Lanuza A.P., additional, Granjeiro, José Mauro, additional, Gubert, Fernanda, additional, Kasai-Brunswick, Tais Hanae, additional, Kossugue, Patrícia M., additional, Kronemberger, Gabriela S., additional, Lindoso, Rafael S., additional, Lojudice, Fernando H., additional, Manetti, Mirko, additional, Mantovani, Marluce C., additional, Martins-Santos, Ricardo, additional, Mendez-Otero, Rosalia, additional, Mesentier-Louro, Louise, additional, Morales, Marcelo Marcos, additional, Nardi, Nance Beyer, additional, Novis, Taissa, additional, Paredes, Bruno Diaz, additional, Paula de Azambuja, Ana, additional, Pelosi, Paolo, additional, Pitossi, Fernando, additional, Mircea Popescu, Larentiu, additional, Rocco, Patricia Rieken Macedo, additional, Santiago, Marcelo Fellipe, additional, Scemes, Eliana, additional, Senegaglia, Alexandra Cristina, additional, Silva, Isaura Beatriz B., additional, Sogayar, Mari C., additional, Taichman, Russell S., additional, Takiya, Christina Maeda, additional, Trentin, Andréa Gonçalves, additional, Vasques, Juliana Ferreira, additional, Verdoorn, Karine S., additional, Vieira-Beiral, Hellen J., additional, Vieyra, Adalberto, additional, Wessely, Oliver, additional, and Zaverucha-do-Valle, Camila, additional

Published
2024
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4. Aquaporin‐4 and transient receptor potential vanilloid 4 balance in early postnatal neurodevelopment

Author

Cibelli, Antonio, primary, Mola, Maria Grazia, additional, Saracino, Emanuela, additional, Barile, Barbara, additional, Abbrescia, Pasqua, additional, Mogni, Guido, additional, Spray, David C., additional, Scemes, Eliana, additional, Rossi, Andrea, additional, Spennato, Diletta, additional, Svelto, Maria, additional, Frigeri, Antonio, additional, Benfenati, Valentina, additional, and Nicchia, Grazia Paola, additional

Published
2024
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5. Chapter 7 - Resident Neural Stem Cells

Author

Zaverucha-do-Valle, Camila, Gubert, Fernanda, Vasques, Juliana Ferreira, Mesentier-Louro, Louise, Scemes, Eliana, Pitossi, Fernando, Santiago, Marcelo Fellipe, and Mendez-Otero, Rosalia

Published
2024
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6. Pannexin1 Mediates Early-Life Seizure-Induced Social Behavior Deficits.

Author

Obot P, Cibelli A, Pan J, Velíšek L, Velíšková J, and Scemes E

Subjects
Animals, Mice, Male, Mice, Inbred C57BL, Kainic Acid, Disease Models, Animal, Nerve Tissue Proteins metabolism, Nerve Tissue Proteins genetics, Connexins metabolism, Connexins genetics, Seizures metabolism, Social Behavior, Mice, Transgenic
Abstract

There is a high co-morbidity between childhood epilepsy and autism spectrum disorder (ASD), with age of seizure onset being a critical determinant of behavioral outcomes. The interplay between these comorbidities has been investigated in animal models with results showing that the induction of seizures at early post-natal ages leads to learning and memory deficits and to autistic-like behavior in adulthood. Modifications of the excitation/inhibition (glutamate/GABA, ATP/adenosine) balance that follows early-life seizures (ELS) are thought to be the physiological events that underlie neuropsychiatric and neurodevelopmental disorders. Although alterations in purinergic/adenosinergic signaling have been implicated in seizures and ASD, it is unknown whether the ATP release channels, Pannexin1 (Panx1), contribute to ELS-induced behavior changes. To tackle this question, we used the ELS-kainic acid model in transgenic mice with global and cell type specific deletion of Panx1 to evaluate whether these channels were involved in behavioral deficits that occur later in life. Our studies show that ELS results in Panx1 dependent social behavior deficits and also in poor performance in a spatial memory test that does not involve Panx1. These findings provide support for a link between ELS and adult behavioral deficits. Moreover, we identify neuronal and not astrocyte Panx1 as a potential target to specifically limit astrogliosis and social behavioral deficits resultant from early-life seizures.

Published
2024
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