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Despite receiving little attention in the scientific literature, earthquakes are known to affect sleep patterns indirectly and can contribute to the development or worsening of sleep disorders. Earthquake-caused disruptions, such as structural damage, displacement, and unstable living conditions, create uncomfortable and anxious sleeping environments. Psychological factors such as fear, stress, and anxiety can contribute to sleep disruption. Physical injuries from earthquakes can disrupt sleep due to pain and discomfort. Additionally, infrastructure disruptions such as power outages and utility damage create uncomfortable or unsafe sleeping conditions. This review presents a comprehensive and systematic overview of the relationship between earthquakes and sleep health.

Despite receiving little attention in the scientific literature, earthquakes are known to affect sleep patterns indirectly and can contribute to the development or worsening of sleep disorders. Earthquake-caused disruptions, such as structural damage, displacement, and unstable living conditions, create uncomfortable and anxious sleeping environments. Psychological factors such as fear, stress, and anxiety can contribute to sleep disruption. Physical injuries from earthquakes can disrupt sleep due to pain and discomfort. Additionally, infrastructure disruptions such as power outages and utility damage create uncomfortable or unsafe sleeping conditions. This review presents a comprehensive and systematic overview of the relationship between earthquakes and sleep health. [ABSTRACT FROM AUTHOR]

Objective: In this study, our objective was to evaluate mean platelet volume (MPV), an indirect marker of platelet activation, in patients with obstructive sleep apnea (OSA), and assess the effect of OSA treatment with continuous positive airway pressure (CPAP) on MPV. Materials and Methods: In this study, records of consecutive patients who underwent polysomnographic evaluation for OSA symptoms in the Sleep Disorders Laboratory during a one-year period were reviewed retrospectively. Patients who had both complete blood count and MPV measurements were included in the study. Results: A total of 158 patients, including 51 females (32.3%) and 107 males (67.7%), were included in the study. The mean age of the patients was 51±13 (min-18, max-82) years. OSA was detected in 74.1% (117/158) of the patients. It was determined that as the severity of OSA increased, hemoglobin and hematocrit values increased significantly. There was no significant difference in platelet count according to the presence and severity of OSA. The MPV was significantly lower in severe OSA cases compared to those without OSA and mild OSA cases. A negative correlation was observed between MPV and the apnea-hypopnea index, desaturation index, and the amount of oxygen saturation below 90% during sleep. There was no significant difference in median erythrocyte and thrombocyte counts, hematocrit percentage and hemoglobin values before and after treatment in OSA patients who used CPAP therapy. However, a significant decrease in MPV was observed after OSA treatment compared to pre-treatment. (p=0.021). Conclusions: The results of the study do not support an increase in MPV and hence platelet activation in severe OSA patients compared with those without OSA. However, the results suggest that one month of CPAP treatment reduces MPV and thus platelet activation in severe OSA patients. Further controlled, prospective studies including treatment outcomes are needed on this subject. [ABSTRACT FROM AUTHOR]