2,120 results on '"Waxman, Stephen G."'
Search Results
2. Ion channels in osteoarthritis: emerging roles and potential targets
3. A multi-ancestry genetic study of pain intensity in 598,339 veterans
4. Nav1.7 as a chondrocyte regulator and therapeutic target for osteoarthritis
5. Voltage-gated sodium channels (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database
6. Disordered but effective: short linear motifs as gene therapy targets for hyperexcitability disorders
7. A FAIR, open-source virtual reality platform for dendritic spine analysis
8. Pain-causing stinging nettle toxins target TMEM233 to modulate NaV1.7 function
9. Compartment-specific regulation of NaV1.7 in sensory neurons after acute exposure to TNF-α
10. Gene therapy for chronic pain: emerging opportunities in target-rich peripheral nociceptors
11. Computational Pipeline to probe NaV1.7 gain-of-functions variants in neuropathic painful syndromes
12. High-throughput combined voltage-clamp/current-clamp analysis of freshly isolated neurons
13. The fates of internalized NaV1.7 channels in sensory neurons: Retrograde cotransport with other ion channels, axon-specific recycling, and degradation
14. Voltage-gated sodium channels (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database
15. iPSCs and DRGs: stepping stones to new pain therapies
16. PAK1 inhibition with Romidepsin attenuates H‐reflex hyperexcitability after spinal cord injury.
17. TRPV1 corneal neuralgia mutation: Enhanced pH response, bradykinin sensitization, and capsaicin desensitization.
18. Nav1.8 in small dorsal root ganglion neurons contributes to vincristine-induced mechanical allodynia.
19. A Novel, Open-Source Virtual Reality Platform for Dendritic Spine Analysis
20. Hominini-specific regulation of CBLN2 increases prefrontal spinogenesis
21. Correction to: Non-extensitivity and criticality of atomic hydropathicity around a voltage-gated sodium channel’s pore: a modeling study
22. Intravenous infusion of auto serum-expanded autologous mesenchymal stem cells in spinal cord injury patients: 13 case series
23. Gain-of-function mutation of a voltage-gated sodium channel NaV1.7 associated with peripheral pain and impaired limb development
24. Non-extensitivity and criticality of atomic hydropathicity around a voltage-gated sodium channel’s pore: a modeling study
25. Dendritic Spines and Pain Memory.
26. Exome Sequencing Implicates Impaired GABA Signaling and Neuronal Ion Transport in Trigeminal Neuralgia
27. Resilience to Stress and Resilience to Pain: Lessons from Molecular Neurobiology and Genetics
28. Functionally-selective inhibition of threshold sodium currents and excitability in dorsal root ganglion neurons by cannabinol
29. Sodium currents in naïve mouse dorsal root ganglion neurons: No major differences between sexes
30. Conditional Astrocyte Rac1 Knockout Attenuates Hyperreflexia After Spinal Cord Injury
31. Status of peripheral sodium channel blockers for non-addictive pain treatment
32. Contributions of NaV1.8 and NaV1.9 to excitability in human induced pluripotent stem-cell derived somatosensory neurons
33. Hydropathicity-based prediction of pain-causing NaV1.7 variants
34. Conditional RAC1 knockout in motor neurons restores H-reflex rate-dependent depression after spinal cord injury
35. Pediatric Erythromelalgia and SCN9A Mutations: Systematic Review and Single-Center Case Series
36. Increased Astrocytic GLT-1 Expression in Tripartite Synapses is Associated with SCI-Induced Hyperreflexia
37. Ih current stabilizes excitability in rodent DRG neurons and reverses hyperexcitability in a nociceptive neuron model of inherited neuropathic pain
38. NaV1.7: A central role in pain
39. Genetic, electrophysiological, and pathological studies on patients with SCN9A‐related pain disorders
40. Targeting a Peripheral Sodium Channel to Treat Pain
41. Congenital Insensitivity to Pain: A Case Report With Corneal Esthesiometry and In Vivo Confocal Microscopy
42. Pharmacological characterization of a rat Nav1.7 loss-of-function model with insensitivity to pain
43. Dexpramipexole blocks Nav1.8 sodium channels and provides analgesia in multiple nociceptive and neuropathic pain models
44. Restoration of brain circulation and cellular functions hours post-mortem
45. Sodium Channels and Pain
46. Molecular Changes in Neurons in Multiple Sclerosis: Altered Axonal Expression of Na v 1.2 and Na v 1.6 Sodium Channels and Na + / Ca 2+ Exchanger
47. Detection of vulnerable neurons damaged by environmental insults in utero
48. Nav1.7 gain-of-function mutation I228M triggers age-dependent nociceptive insensitivity and C-LTMR dysregulation
49. Voltage-Gated Ion Channels as Molecular Targets for Pain
50. Computational pipeline to probe NaV1.7 gain-of-function variants in neuropathic painful syndromes
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