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2. Synthetic retinoid-mediated preconditioning of cancer-associated fibroblasts and macrophages improves cancer response to immune checkpoint blockade

6. Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics

8. The Origin and Contribution of Cancer-Associated Fibroblasts in Colorectal Carcinogenesis

9. The Balance of Stromal BMP Signaling Mediated by GREM1 and ISLR Drives Colorectal Carcinogenesis

12. Machine learning-assisted chemical design of highly efficient deicers.

23. Correction: Pharmacologic conversion of cancer-associated fibroblasts from a protumor phenotype to an antitumor phenotype improves the sensitivity of pancreatic cancer to chemotherapeutics

24. Meflin is a marker of pancreatic stellate cells involved in fibrosis and epithelial regeneration in the pancreas

33. Meflin is a marker of pancreatic stellate cells involved in fibrosis and epithelial regeneration in the pancreas.

37. Supplementary Data from Meflin-Positive Cancer-Associated Fibroblasts Inhibit Pancreatic Carcinogenesis

38. Supplemental Table S1 from Akt–Girdin Signaling in Cancer-Associated Fibroblasts Contributes to Tumor Progression

40. Supplemental Figure S1 from Akt–Girdin Signaling in Cancer-Associated Fibroblasts Contributes to Tumor Progression

48. Data from A Novel Mouse Model for De novo Melanoma

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