19 results on '"Meyer, Anne R."'
Search Results
2. Recruitment of Polarity Complexes and Tight Junction Proteins to the Site of Apical Bulk Endocytosis
3. Cystine/Glutamate Antiporter (xCT) Is Required for Chief Cell Plasticity After Gastric Injury
4. Loss of MYO5B Leads to Reductions in Na+ Absorption With Maintenance of CFTR-Dependent Cl– Secretion in Enterocytes
5. Epithelial Smad4 Deletion Up-Regulates Inflammation and Promotes Inflammation-Associated Cancer
6. Chief cell plasticity is the origin of metaplasia following acute injury in the stomach mucosa
7. Sa119 RECRUITMENT OF POLARITY COMPLEXES AND TIGHT JUNCTION PROTEINS TO THE SITE OF APICAL BULK ENDOCYTOSIS
8. Chief cell plasticity is the origin of metaplasia following acute injury in the stomach mucosa.
9. Group 2 Innate Lymphoid Cells Coordinate Damage Response in the Stomach
10. Editing Myosin VB Gene to Create Porcine Model of Microvillus Inclusion Disease, With Microvillus-Lined Inclusions and Alterations in Sodium Transporters
11. Loss of myosin Vb promotes apical bulk endocytosis in neonatal enterocytes
12. Evaluation of Lineage Changes in the Gastric Mucosa Following Infection With Helicobacter pylori and Specified Intestinal Flora in INS-GAS Mice
13. Injury, repair, inflammation and metaplasia in the stomach
14. A signalling cascade of IL-33 to IL-13 regulates metaplasia in the mouse stomach
15. Maturity and age influence chief cell ability to transdifferentiate into metaplasia
16. Evaluation of Lineage Changes in the Gastric Mucosa Following Infection With Helicobacter pylori and Specified Intestinal Flora in INS-GAS Mice.
17. A signalling cascade of IL-33 to IL-13 regulates metaplasia in the mouse stomach.
18. Evaluation of Lineage Changes in the Gastric Mucosa Following Infection With Helicobacter pyloriand Specified Intestinal Flora in INS-GAS Mice
19. Maturity and age influence chief cell ability to transdifferentiate into metaplasia.
Catalog
Books, media, physical & digital resources
Discovery Service for Jio Institute Digital Library
For full access to our library's resources, please sign in.