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1. Peroxiredoxin 3 deficiency induces cardiac hypertrophy and dysfunction by impaired mitochondrial quality control

5. Anti-Inflammatory Actions of Soluble Ninjurin-1 Ameliorate Atherosclerosis

7. Supplementary Methods and Materials, Tables 1-4, Figure Legends 1-3 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis

8. Supplementary Figure 3 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis

9. Supplementary Figure 1 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis

10. Supplementary Figure 5 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

11. Supplementary Figure 2 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis

12. Supplementary Materials and Methods, Figure Legends 1-7, Table 1 Legend from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

13. Data from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis

14. Data from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

15. Supplementary Figure 3 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

16. Supplementary Figure 4 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

17. Supplementary Figure 6 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

18. Supplementary Figure 2 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

19. Supplementary Figure 7 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

20. Supplementary Figure 1 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

21. Supplementary Table 1 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

25. Topoisomerase IIIβ Deficiency Induces Neuro-Behavioral Changes and Brain Connectivity Alterations in Mice

26. Naa12 compensates for Naa10 in mice in the amino-terminal acetylation pathway

27. Author response: Naa12 compensates for Naa10 in mice in the amino-terminal acetylation pathway

28. Naa12 compensates for Naa10 in mice in the amino-terminal acetylation pathway

31. Prdx1 (peroxiredoxin 1) deficiency reduces cholesterol efflux via impaired macrophage lipophagic flux

33. ARD1-mediated Hsp70 acetylation balances stress-induced protein refolding and degradation

34. Prdx1 (peroxiredoxin 1) deficiency reduces cholesterol efflux via impaired macrophage lipophagic flux.

35. Correction: Corrigendum: The adipokine Retnla modulates cholesterol homeostasis in hyperlipidemic mice

36. NAA10 controls osteoblast differentiation and bone formation as a feedback regulator of Runx2

37. The adipokine Retnla modulates cholesterol homeostasis in hyperlipidemic mice

40. Peroxiredoxin 2 Deficiency Exacerbates Atherosclerosis in Apolipoprotein E–Deficient Mice

41. 5-(4-Hydroxy-2,3,5-trimethylbenzylidene) thiazolidine-2,4-dione attenuates atherosclerosis possibly by reducing monocyte recruitment to the lesion

42. Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation

43. CD137 (4–1BB) Deficiency Reduces Atherosclerosis in Hyperlipidemic Mice

44. RhoGDI2 Expression Is Associated with Tumor Growth and Malignant Progression of Gastric Cancer

45. Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis

46. Inhibitory effects of tilianin on the expression of inducible nitric oxide synthase in low density lipoprotein receptor deficiency mice

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