153 results on '"Hatsell, Sarah"'
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2. Antibodies to sclerostin or G-CSF receptor partially eliminate bone or marrow adipocyte loss, respectively, following vertical sleeve gastrectomy
3. Comparative Transcriptomics Identifies Novel Genes and Pathways Involved in Post-Traumatic Osteoarthritis Development and Progression.
4. SOST/Sclerostin Improves Posttraumatic Osteoarthritis and Inhibits MMP2/3 Expression After Injury
5. Lysosomal degradation of ACVR1-Activin complexes negatively regulates signaling of Activins and Bone Morphogenetic Proteins
6. How Activin A Became a Therapeutic Target in Fibrodysplasia Ossificans Progressiva
7. Global molecular changes in a tibial compression induced ACL rupture model of post‐traumatic osteoarthritis
8. Wnt co-receptors Lrp5 and Lrp6 differentially mediate Wnt3a signaling in osteoblasts
9. Activin A does not drive post-traumatic heterotopic ossification
10. Sclerostin antibody treatment improves fracture outcomes in a Type I diabetic mouse model
11. Anti-ACVRI antibodies exacerbate heterotopic ossification in fibrodysplasia ossificans progressive (FOP) by activating FOP-mutant ACVR1
12. S281: TMPRSS6 INHIBITION WITH A MONOCLONAL ANTIBODY IMPROVES RED BLOOD CELL HEALTH AND IRON OVERLOAD IN A MOUSE MODEL OF BETA-THALASSEMIA
13. The obligatory role of Activin A in the formation of heterotopic bone in Fibrodysplasia Ossificans Progressiva
14. Dissecting the Roles of β-Catenin and Cyclin D1 during Mammary Development and Neoplasia
15. Genetic and functional analysis of syndromic and nonsyndromic genodermatoses
16. 340 Dupilumab treatment of children with moderate-to-severe atopic dermatitis increases bone alkaline phosphatase, a marker of bone mineralization
17. Sclerostin antibody treatment improves fracture outcomes in a Type I diabetic mouse model
18. TMPRSS6 Inhibition with a Monoclonal Antibody Improves Red Blood Cell Health and Reduces Hepatic Iron Loading in Mouse Models of Iron Overload Diseases
19. 676 - Dupilumab increases levels of bone growth biomarker irrespective of prior use of systemic corticosteroids in children with moderate-to-severe atopic dermatitis.
20. Exome sequencing of 300,000 individuals implicates target genes for osteoporosis
21. Conditionals by inversion provide a universal method for the generation of conditional alleles
22. ACVR1 antibodies exacerbate heterotopic ossification in fibrodysplasia ossificans progressiva (FOP) by activating FOP-mutant ACVR1
23. Hedgehog Signaling in Mammary Gland Development and Breast Cancer
24. Estrogen receptor positivity in mammary tumors of Wnt-1 transgenic mice is influenced by collaborating oncogenic mutations
25. Anti-GDF11Treatment of β-Thalassemia Intermedia Mice Does Not Improve Erythropoiesis
26. β-Catenin and Tcfs in Mammary Development and Cancer
27. Dissecting the roles of [beta]-catenin and cyclin D1 during mammary development and neoplasia
28. Abstract 89
29. Novel Splice Site Mutation in Keratin 1 Underlies Mild Epidermolytic Palmoplantar Keratoderma in Three Kindreds
30. Genetic loss of heparanase does not inhibit osteochondromas in Ext1 and Ext2 double heterozygous hereditary multiple osteochondroma mouse model
31. An Acvr1[R258G] conditional on' mouse model of atypical fibrodysplasia ossificans progressiva (FOP) is Activin A dependent
32. Global molecular changes in a tibial compression induced ACL rupture model of post-traumatic osteoarthritis
33. Comparative Transcriptomics Identifies Novel Genes and Pathways Involved in Post-Traumatic Osteoarthritis Development and Progression
34. Monoallelic BMP2 Variants Predicted to Result in Haploinsufficiency Cause Craniofacial, Skeletal, and Cardiac Features Overlapping Those of 20p12 Deletions
35. The Expansion of Heterotopic Bone in Fibrodysplasia Ossificans Progressiva Is Activin A-Dependent
36. 340 Dupilumab treatment of children with moderateto-severe atopic dermatitis increases bone alkaline phosphatase, a marker of bone mineralization.
37. Two tissue-resident progenitor lineages drive distinct phenotypes of heterotopic ossification
38. Global molecular changes in a tibial compression induced ACL rupture model of post‐traumatic osteoarthritis
39. Tracking inflammation in mouse model of fibrodysplasia ossificans progressiva prior to the detection of heterotopic ossification as a potential biomarker
40. In Vivo Quantitative Microcomputed Tomographic Analysis of Vasculature and Organs in a Normal and Diseased Mouse Model
41. ACVR1R206Hreceptor mutation causes fibrodysplasia ossificans progressiva by imparting responsiveness to activin A
42. Correction: MMTV-Wnt1 and -ΔN89β-Catenin Induce Canonical Signaling in Distinct Progenitors and Differentially Activate Hedgehog Signaling within Mammary Tumors
43. Gli Activity Is Critical at Multiple Stages of Embryonic Mammary and Nipple Development
44. Distinct Modes of Inhibition by Sclerostin on Bone Morphogenetic Protein and Wnt Signaling Pathways
45. MMTV-Wnt1 and -ΔN89β-Catenin Induce Canonical Signaling in Distinct Progenitors and Differentially Activate Hedgehog Signaling within Mammary Tumors
46. ACVR1R206H receptor mutation causes fibrodysplasia ossificans progressiva by imparting responsiveness to activin A.
47. Gli3-mediated repression of Hedgehog targets is required for normal mammary development
48. Cadherins and catenins in breast cancer
49. b-Catenin and Cyclin D1: Connecting Development to Breast Cancer
50. Plakoglobin Is O-Glycosylated Close to the N-terminal Destruction Box
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