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7. MHC class II–dependent basophil–CD4+ T cell interactions promote TH2 cytokine–dependent immunity

8. Randomized, Placebo Controlled Trial of Experimental Hookworm Infection for Improving Gluten Tolerance in Celiac Disease

11. Peptides derived from hookworm anti-inflammatory proteins suppress inducible colitis in mice and inflammatory cytokine production by human cells

12. Novel antiinflammatory biologics shaped by parasite–host coevolution

13. Histone deacetylase 3 coordinates commensal-bacteria-dependent intestinal homeostasis

14. Thymic stromal lymphopoietin--elicited basophil responses promote eosinophilic esophagitis

19. TSLP-elicited basophil responses mediate the pathogenesis of eosinophilic esophagitis: 282

21. Inflammasome-Independent Role for NLRP3 in Controlling Innate Antihelminth Immunity and Tissue Repair in the Lung

23. Hookworm Secreted Extracellular Vesicles Interact With Host Cells and Prevent Inducible Colitis in Mice

24. The NLRP3 Inflammasome Suppresses Protective Immunity to Gastrointestinal Helminth Infection

25. Major histocompatibility complex class II-dependent basophil-CD4+ T cell interactions promote TH2 cytokine-dependent immunity

27. Hookworm recombinant protein promotes regulatory T cell responses that suppress experimental asthma

30. The Intestinal Microbiota Contributes to the Ability of Helminths to Modulate Allergic Inflammation

31. Carcinogenic Parasite Secretes Growth Factor That Accelerates Wound Healing and Potentially Promotes Neoplasia

32. Epithelial-intrinsic IKKα expression regulates group 3 innate lymphoid cell responses and antibacterial immunity

33. Probing of a Human Proteome Microarray With a Recombinant Pathogen Protein Reveals a Novel Mechanism by Which Hookworms Suppress B-Cell Receptor Signaling

34. 282

38. IL-35-mediated induction of a potent regulatory T cell population

41. 282: TSLP-elicited basophil responses mediate the pathogenesis of eosinophilic esophagitis

42. MHC class II–dependent basophil–CD4+ T cell interactions promote TH2 cytokine–dependent immunity.

43. Loss of Complement Activation and Leukocyte Adherence as Nippostrongylus brasiliensisDevelops within the Murine Host

44. 282: TSLP-elicited basophil responses mediate the pathogenesis of eosinophilic esophagitis.

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