253 results on '"Gadeau, Alain-Pierre"'
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2. Signalling by senescent melanocytes hyperactivates hair growth
3. Thrombosis in the Coronary Microvasculature Impairs Cardiac Relaxation and Induces Diastolic Dysfunction
4. Thrombosis in the Coronary Microvasculature Impairs Cardiac Relaxation and Induces Diastolic Dysfunction
5. Empagliflozin inhibits angiotensin II-induced hypertrophy in H9c2 cardiomyoblasts through inhibition of NHE1 expression
6. Evaluating the effects of sodium glucose co-transporter -2 inhibitors from a renin–angiotensin–aldosterone system perspective in patients infected with COVID-19: contextualizing findings from the dapagliflozin in respiratory failure in patients with COVID-19 study
7. Desert Hedgehog-Driven Endothelium Integrity Is Enhanced by Gas1 (Growth Arrest-Specific 1) but Negatively Regulated by Cdon (Cell Adhesion Molecule-Related/Downregulated by Oncogenes)
8. Abstract P3107: Sexual Dimorphism In Diastolic Dysfunction And Cardiac Small Vessel Disease In Heart Failure With Preserved Ejection Fraction Mouse Models
9. Abstract P2082: Thrombosis In The Coronary Microvasculature May Be Responsible For Impaired Cardiac Relaxation And Diastolic Dysfunction
10. Partial Mural Cell Ablation Disrupts Coronary Vasculature Integrity and Induces Systolic Dysfunction
11. A new reliable, transposable and cost-effective assay for absolute quantification of total plasmatic bevacizumab by LC–MS/MS in human plasma comparing two internal standard calibration approaches
12. Intra-articular Injection of Mesenchymal Stem Cells and Platelet-Rich Plasma to Treat Patellofemoral Osteoarthritis: Preliminary Results of a Long-Term Pilot Study
13. Tamoxifen Accelerates Endothelial Healing by Targeting ERα in Smooth Muscle Cells
14. Thrombosis in the coronary microvasculature may be responsible for impaired cardiac relaxation and diastolic dysfunction
15. Cardiac pericytes may contribute to heart disease
16. ICAM1 expression by the microvasculature impairs capillary perfusion which compromises hind limb ischemia recovery in diabetic mice
17. Diastolic dysfunction is associated with cardiac small vessel disease in ovariectomized females but not in males
18. Increased endothelium activation and leakage do not promote diastolic dysfunction in mice fed with a high fat diet and treated with L-NAME
19. Praliciguat Promotes Ischemic Leg Reperfusion in Leptin Receptor-Deficient Mice
20. Sonic hedgehog mediates a novel pathway of PDGF-BB–dependent vessel maturation
21. Restoring Endothelial Function by Targeting Desert Hedgehog Downstream of Klf2 Improves Critical Limb Ischemia in Adults
22. Cardiac pericytes are necessary for coronary vasculature integrity and cardiomyocyte differentiation
23. Testosterone Prevents Cutaneous Ischemia and Necrosis in Males Through Complementary Estrogenic and Androgenic Actions
24. Na+/H+ exchanger isoform 1 induced osteopontin expression in cardiomyocytes involves NFAT3/Gata4
25. Endothelial protection does not prevent diastolic dysfunction in high fat diet + L-NAME treated mice
26. Soluble guanylate cyclase stimulator praliciguat promotes ischemic leg reperfusion in db/db mice
27. Desert Hedgehog related endothelial dysfunction is sufficient to induce diastolic dysfunction
28. Activation function 2 (AF2) of estrogen receptor-α is required for the atheroprotective action of estradiol but not to accelerate endothelial healing
29. Cardiac and brain pericytes are able to regenerate over time
30. Impaired Hedgehog signalling-induced endothelial dysfunction is sufficient to induce neuropathy: implication in diabetes
31. Crosstalk between Sodium–Glucose Cotransporter Inhibitors and Sodium–Hydrogen Exchanger 1 and 3 in Cardiometabolic Diseases
32. The Estrogen Effects on Endothelial Repair and Mitogen-Activated Protein Kinase Activation Are Abolished in Endothelial Nitric-Oxide (NO) Synthase Knockout Mice, but Not by NO Synthase Inhibition by N-Nitro- l-arginine Methyl Ester
33. Mast Cells Are the Trigger of Small Vessel Disease and Diastolic Dysfunction in Diabetic Obese Mice
34. Estradiol accelerates endothelial healing through the retrograde commitment of uninjured endothelium
35. Blood-brain barrier genetic disruption leads to protective barrier formation at the Glia Limitans
36. Ca2+-Activated K+ Channel-3.1 Blocker TRAM-34 Attenuates Airway Remodeling and Eosinophilia in a Murine Asthma Model
37. Osteopontin expression in normal and fibrotic liver. Altered liver healing in osteopontin-deficient mice
38. Blood–brain barrier genetic disruption leads to protective barrier formation at the Glia Limitans
39. Full-length Dhh and N-terminal Shh act as competitive antagonists to regulate angiogenesis and vascular permeability
40. Mast cells participate in the development of diastolic dysfunction in diabetic obese mice
41. Wavelet Analysis of Microcirculatory Flowmotion Reveals Cardiovascular Regulatory Mechanisms–Data from a Beta-Blocker
42. Desert Hedgehog-driven endothelium integrity is enhanced by Gas1 but negatively regulated by Cdon
43. Genetic disruption of the Blood Brain Barrier leads to protective barrier formation at the Glia Limitans
44. Endothelial cell response to Hedgehog ligands depends on their processing
45. UTP Induces Osteopontin Expression through a Coordinate Action of NFκB, Activator Protein-1, and Upstream Stimulatory Factor in Arterial Smooth Muscle Cells
46. Characterizing Vascular Dysfunction in Genetically Modified Mice through the Hyperoxia Model
47. Osteopontin: A Promising Therapeutic Target in Cardiac Fibrosis
48. Full-length Dhh and N-terminal Shh act as competitive antagonists to regulate angiogenesis and vascular permeability.
49. Exogenous ATP induces a limited cell cycle progression of arterial smooth muscle cells
50. Evidence on microcirculatory dynamics that preserves the equivalent perfusion steady state in both inferior limbs
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