1. Loss of Soluble (Pro)renin Receptor Attenuates Angiotensin-II Induced Hypertension and Renal Injury.
- Author
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Ramkumar N, Stuart D, Peterson CS, Hu C, Wheatley W, Min Cho J, Symons JD, and Kohan DE
- Subjects
- Animals, Disease Models, Animal, Hypertension chemically induced, Hypertension genetics, Hypertension metabolism, Kidney pathology, Kidney physiopathology, Kidney Diseases chemically induced, Kidney Diseases genetics, Kidney Diseases metabolism, Male, Mesenteric Arteries physiopathology, Mice, Inbred C57BL, Mice, Mutant Strains, Mutation, Proton-Translocating ATPases genetics, Receptors, Cell Surface genetics, Renin-Angiotensin System, Signal Transduction, Vasoconstriction, Vasodilation, Water-Electrolyte Balance, Prorenin Receptor, Mice, Angiotensin II, Blood Pressure, Hypertension prevention & control, Kidney metabolism, Kidney Diseases prevention & control, Mesenteric Arteries metabolism, Proton-Translocating ATPases deficiency, Receptors, Cell Surface deficiency
- Abstract
[Figure: see text].
- Published
- 2021
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