Author: "Vezzoli, G." / Publication Year Range: Last 3 years - Searchworks@Jio Institute Digital Library Search Results

Your search keyword '"Vezzoli, G."' showing total 35 results

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35 results on '"Vezzoli, G."'

1. Effects of empagliflozin on progression of chronic kidney disease: a prespecified secondary analysis from the empa-kidney trial

Author: Staplin, N, Haynes, R, Judge, PK, Wanner, C, Green, JB, Emberson, J, Preiss, D, Mayne, KJ, Ng, SYA, Sammons, E, Zhu, D, Hill, M, Stevens, W, Wallendszus, K, Brenner, S, Cheung, AK, Liu, ZH, Li, J, Hooi, LS, Liu, WJ, Kadowaki, T, Nangaku, M, Levin, A, Cherney, D, Maggioni, AP, Pontremoli, R, Deo, R, Goto, S, Rossello, X, Tuttle, KR, Steubl, D, Petrini, M, Seidi, S, Landray, MJ, Baigent, C, Herrington, WG, Abat, S, Abd Rahman, R, Abdul Cader, R, Abdul Hafidz, MI, Abdul Wahab, MZ, Abdullah, NK, Abdul-Samad, T, Abe, M, Abraham, N, Acheampong, S, Achiri, P, Acosta, JA, Adeleke, A, Adell, V, Adewuyi-Dalton, R, Adnan, N, Africano, A, Agharazii, M, Aguilar, F, Aguilera, A, Ahmad, M, Ahmad, MK, Ahmad, NA, Ahmad, NH, Ahmad, NI, Ahmad Miswan, N, Ahmad Rosdi, H, Ahmed, I, Ahmed, S, Aiello, J, Aitken, A, AitSadi, R, Aker, S, Akimoto, S, Akinfolarin, A, Akram, S, Alberici, F, Albert, C, Aldrich, L, Alegata, M, Alexander, L, Alfaress, S, Alhadj Ali, M, Ali, A, Alicic, R, Aliu, A, Almaraz, R, Almasarwah, R, Almeida, J, Aloisi, A, Al-Rabadi, L, Alscher, D, Alvarez, P, Al-Zeer, B, Amat, M, Ambrose, C, Ammar, H, An, Y, Andriaccio, L, Ansu, K, Apostolidi, A, Arai, N, Araki, H, Araki, S, Arbi, A, Arechiga, O, Armstrong, S, Arnold, T, Aronoff, S, Arriaga, W, Arroyo, J, Arteaga, D, Asahara, S, Asai, A, Asai, N, Asano, S, Asawa, M, Asmee, MF, Aucella, F, Augustin, M, Avery, A, Awad, A, Awang, IY, Awazawa, M, Axler, A, Ayub, W, Azhari, Z, Baccaro, R, Badin, C, Bagwell, B, Bahlmann-Kroll, E, Bahtar, AZ, Bains, D, Bajaj, H, Baker, R, Baldini, E, Banas, B, Banerjee, D, Banno, S, Bansal, S, Barberi, S, Barnes, S, Barnini, C, Barot, C, Barrett, K, Barrios, R, Bartolomei Mecatti, B, Barton, I, Barton, J, Basily, W, Bavanandan, S, Baxter, A, Becker, L, Beddhu, S, Beige, J, Beigh, S, Bell, S, Benck, U, Beneat, A, Bennett, A, Bennett, D, Benyon, S, Berdeprado, J, Bergler, T, Bergner, A, Berry, M, Bevilacqua, M, Bhairoo, J, Bhandari, S, Bhandary, N, Bhatt, A, Bhattarai, M, Bhavsar, M, Bian, W, Bianchini, F, Bianco, S, Bilous, R, Bilton, J, Bilucaglia, D, Bird, C, Birudaraju, D, Biscoveanu, M, Blake, C, Bleakley, N, Bocchicchia, K, Bodine, S, Bodington, R, Boedecker, S, Bolduc, M, Bolton, S, Bond, C, Boreky, F, Boren, K, Bouchi, R, Bough, L, Bovan, D, Bowler, C, Bowman, L, Brar, N, Braun, C, Breach, A, Breitenfeldt, M, Brettschneider, B, Brewer, A, Brewer, G, Brindle, V, Brioni, E, Brown, C, Brown, H, Brown, L, Brown, R, Brown, S, Browne, D, Bruce, K, Brueckmann, M, Brunskill, N, Bryant, M, Brzoska, M, Bu, Y, Buckman, C, Budoff, M, Bullen, M, Burke, A, Burnette, S, Burston, C, Busch, M, Bushnell, J, Butler, S, Büttner, C, Byrne, C, Caamano, A, Cadorna, J, Cafiero, C, Cagle, M, Cai, J, Calabrese, K, Calvi, C, Camilleri, B, Camp, S, Campbell, D, Campbell, R, Cao, H, Capelli, I, Caple, M, Caplin, B, Cardone, A, Carle, J, Carnall, V, Caroppo, M, Carr, S, Carraro, G, Carson, M, Casares, P, Castillo, C, Castro, C, Caudill, B, Cejka, V, Ceseri, M, Cham, L, Chamberlain, A, Chambers, J, Chan, CBT, Chan, JYM, Chan, YC, Chang, E, Chant, T, Chavagnon, T, Chellamuthu, P, Chen, F, Chen, J, Chen, P, Chen, TM, Chen, Y, Cheng, C, Cheng, H, Cheng, MC, Ching, CH, Chitalia, N, Choksi, R, Chukwu, C, Chung, K, Cianciolo, G, Cipressa, L, Clark, S, Clarke, H, Clarke, R, Clarke, S, Cleveland, B, Cole, E, Coles, H, Condurache, L, Connor, A, Convery, K, Cooper, A, Cooper, N, Cooper, Z, Cooperman, L, Cosgrove, L, Coutts, P, Cowley, A, Craik, R, Cui, G, Cummins, T, Dahl, N, Dai, H, Dajani, L, D'Amelio, A, Damian, E, Damianik, K, Danel, L, Daniels, C, Daniels, T, Darbeau, S, Darius, H, Dasgupta, T, Davies, J, Davies, L, Davis, A, Davis, J, Davis, L, Dayanandan, R, Dayi, S, Dayrell, R, De Nicola, L, Debnath, S, Deeb, W, Degenhardt, S, DeGoursey, K, Delaney, M, DeRaad, R, Derebail, V, Dev, D, Devaux, M, Dhall, P, Dhillon, G, Dienes, J, Dobre, M, Doctolero, E, Dodds, V, Domingo, D, Donaldson, D, Donaldson, P, Donhauser, C, Donley, V, Dorestin, S, Dorey, S, Doulton, T, Draganova, D, Draxlbauer, K, Driver, F, Du, H, Dube, F, Duck, T, Dugal, T, Dugas, J, Dukka, H, Dumann, H, Durham, W, Dursch, M, Dykas, R, Easow, R, Eckrich, E, Eden, G, Edmerson, E, Edwards, H, Ee, LW, Eguchi, J, Ehrl, Y, Eichstadt, K, Eid, W, Eilerman, B, Ejima, Y, Eldon, H, Ellam, T, Elliott, L, Ellison, R, Epp, R, Er, A, Espino-Obrero, M, Estcourt, S, Estienne, L, Evans, G, Evans, J, Evans, S, Fabbri, G, Fajardo-Moser, M, Falcone, C, Fani, F, Faria-Shayler, P, Farnia, F, Farrugia, D, Fechter, M, Fellowes, D, Feng, F, Fernandez, J, Ferraro, P, Field, A, Fikry, S, Finch, J, Finn, H, Fioretto, P, Fish, R, Fleischer, A, Fleming-Brown, D, Fletcher, L, Flora, R, Foellinger, C, Foligno, N, Forest, S, Forghani, Z, Forsyth, K, Fottrell-Gould, D, Fox, P, Frankel, A, Fraser, D, Frazier, R, Frederick, K, Freking, N, French, H, Froment, A, Fuchs, B, Fuessl, L, Fujii, H, Fujimoto, A, Fujita, A, Fujita, K, Fujita, Y, Fukagawa, M, Fukao, Y, Fukasawa, A, Fuller, T, Funayama, T, Fung, E, Furukawa, M, Furukawa, Y, Furusho, M, Gabel, S, Gaidu, J, Gaiser, S, Gallo, K, Galloway, C, Gambaro, G, Gan, CC, Gangemi, C, Gao, M, Garcia, K, Garcia, M, Garofalo, C, Garrity, M, Garza, A, Gasko, S, Gavrila, M, Gebeyehu, B, Geddes, A, Gentile, G, George, A, George, J, Gesualdo, L, Ghalli, F, Ghanem, A, Ghate, T, Ghavampour, S, Ghazi, A, Gherman, A, Giebeln-Hudnell, U, Gill, B, Gillham, S, Girakossyan, I, Girndt, M, Giuffrida, A, Glenwright, M, Glider, T, Gloria, R, Glowski, D, Goh, BL, Goh, CB, Gohda, T, Goldenberg, R, Goldfaden, R, Goldsmith, C, Golson, B, Gonce, V, Gong, Q, Goodenough, B, Goodwin, N, Goonasekera, M, Gordon, A, Gordon, J, Gore, A, Goto, H, Gowen, D, Grace, A, Graham, J, Grandaliano, G, Gray, M, Greene, T, Greenwood, G, Grewal, B, Grifa, R, Griffin, D, Griffin, S, Grimmer, P, Grobovaite, E, Grotjahn, S, Guerini, A, Guest, C, Gunda, S, Guo, B, Guo, Q, Haack, S, Haase, M, Haaser, K, Habuki, K, Hadley, A, Hagan, S, Hagge, S, Haller, H, Ham, S, Hamal, S, Hamamoto, Y, Hamano, N, Hamm, M, Hanburry, A, Haneda, M, Hanf, C, Hanif, W, Hansen, J, Hanson, L, Hantel, S, Haraguchi, T, Harding, E, Harding, T, Hardy, C, Hartner, C, Harun, Z, Harvill, L, Hasan, A, Hase, H, Hasegawa, F, Hasegawa, T, Hashimoto, A, Hashimoto, C, Hashimoto, M, Hashimoto, S, Haskett, S, Hauske, SJ, Hawfield, A, Hayami, T, Hayashi, M, Hayashi, S, Hazara, A, Healy, C, Hecktman, J, Heine, G, Henderson, H, Henschel, R, Hepditch, A, Herfurth, K, Hernandez, G, Hernandez Pena, A, Hernandez-Cassis, C, Herzog, C, Hewins, S, Hewitt, D, Hichkad, L, Higashi, S, Higuchi, C, Hill, C, Hill, L, Himeno, T, Hing, A, Hirakawa, Y, Hirata, K, Hirota, Y, Hisatake, T, Hitchcock, S, Hodakowski, A, Hodge, W, Hogan, R, Hohenstatt, U, Hohenstein, B, Hooi, L, Hope, S, Hopley, M, Horikawa, S, Hosein, D, Hosooka, T, Hou, L, Hou, W, Howie, L, Howson, A, Hozak, M, Htet, Z, Hu, X, Hu, Y, Huang, J, Huda, N, Hudig, L, Hudson, A, Hugo, C, Hull, R, Hume, L, Hundei, W, Hunt, N, Hunter, A, Hurley, S, Hurst, A, Hutchinson, C, Hyo, T, Ibrahim, FH, Ibrahim, S, Ihana, N, Ikeda, T, Imai, A, Imamine, R, Inamori, A, Inazawa, H, Ingell, J, Inomata, K, Inukai, Y, Ioka, M, Irtiza-Ali, A, Isakova, T, Isari, W, Iselt, M, Ishiguro, A, Ishihara, K, Ishikawa, T, Ishimoto, T, Ishizuka, K, Ismail, R, Itano, S, Ito, H, Ito, K, Ito, M, Ito, Y, Iwagaitsu, S, Iwaita, Y, Iwakura, T, Iwamoto, M, Iwasa, M, Iwasaki, H, Iwasaki, S, Izumi, K, Izumi, T, Jaafar, SM, Jackson, C, Jackson, Y, Jafari, G, Jahangiriesmaili, M, Jain, N, Jansson, K, Jasim, H, Jeffers, L, Jenkins, A, Jesky, M, Jesus-Silva, J, Jeyarajah, D, Jiang, Y, Jiao, X, Jimenez, G, Jin, B, Jin, Q, Jochims, J, Johns, B, Johnson, C, Johnson, T, Jolly, S, Jones, L, Jones, S, Jones, T, Jones, V, Joseph, M, Joshi, S, Judge, P, Junejo, N, Junus, S, Kachele, M, Kadoya, H, Kaga, H, Kai, H, Kajio, H, Kaluza-Schilling, W, Kamaruzaman, L, Kamarzarian, A, Kamimura, Y, Kamiya, H, Kamundi, C, Kan, T, Kanaguchi, Y, Kanazawa, A, Kanda, E, Kanegae, S, Kaneko, K, Kang, HY, Kano, T, Karim, M, Karounos, D, Karsan, W, Kasagi, R, Kashihara, N, Katagiri, H, Katanosaka, A, Katayama, A, Katayama, M, Katiman, E, Kato, K, Kato, M, Kato, N, Kato, S, Kato, T, Kato, Y, Katsuda, Y, Katsuno, T, Kaufeld, J, Kavak, Y, Kawai, I, Kawai, M, Kawase, A, Kawashima, S, Kazory, A, Kearney, J, Keith, B, Kellett, J, Kelley, S, Kershaw, M, Ketteler, M, Khai, Q, Khairullah, Q, Khandwala, H, Khoo, KKL, Khwaja, A, Kidokoro, K, Kielstein, J, Kihara, M, Kimber, C, Kimura, S, Kinashi, H, Kingston, H, Kinomura, M, Kinsella-Perks, E, Kitagawa, M, Kitajima, M, Kitamura, S, Kiyosue, A, Kiyota, M, Klauser, F, Klausmann, G, Kmietschak, W, Knapp, K, Knight, C, Knoppe, A, Knott, C, Kobayashi, M, Kobayashi, R, Kobayashi, T, Koch, M, Kodama, S, Kodani, N, Kogure, E, Koizumi, M, Kojima, H, Kojo, T, Kolhe, N, Komaba, H, Komiya, T, Komori, H, Kon, SP, Kondo, M, Kong, W, Konishi, M, Kono, K, Koshino, M, Kosugi, T, Kothapalli, B, Kozlowski, T, Kraemer, B, Kraemer-Guth, A, Krappe, J, Kraus, D, Kriatselis, C, Krieger, C, Krish, P, Kruger, B, Ku Md Razi, KR, Kuan, Y, Kubota, S, Kuhn, S, Kumar, P, Kume, S, Kummer, I, Kumuji, R, Küpper, A, Kuramae, T, Kurian, L, Kuribayashi, C, Kurien, R, Kuroda, E, Kurose, T, Kutschat, A, Kuwabara, N, Kuwata, H, La Manna, G, Lacey, M, Lafferty, K, LaFleur, P, Lai, V, Laity, E, Lambert, A, Langlois, M, Latif, F, Latore, E, Laundy, E, Laurienti, D, Lawson, A, Lay, M, Leal, I, Lee, AK, Lee, J, Lee, KQ, Lee, R, Lee, SA, Lee, YY, Lee-Barkey, Y, Leonard, N, Leoncini, G, Leong, CM, Lerario, S, Leslie, A, Lewington, A, Li, N, Li, X, Li, Y, Liberti, L, Liberti, ME, Liew, A, Liew, YF, Lilavivat, U, Lim, SK, Lim, YS, Limon, E, Lin, H, Lioudaki, E, Liu, H, Liu, J, Liu, L, Liu, Q, Liu, X, Liu, Z, Loader, D, Lochhead, H, Loh, CL, Lorimer, A, Loudermilk, L, Loutan, J, Low, CK, Low, CL, Low, YM, Lozon, Z, Lu, Y, Lucci, D, Ludwig, U, Luker, N, Lund, D, Lustig, R, Lyle, S, Macdonald, C, MacDougall, I, Machicado, R, MacLean, D, Macleod, P, Madera, A, Madore, F, Maeda, K, Maegawa, H, Maeno, S, Mafham, M, Magee, J, Mah, DY, Mahabadi, V, Maiguma, M, Makita, Y, Makos, G, Manco, L, Mangiacapra, R, Manley, J, Mann, P, Mano, S, Marcotte, G, Maris, J, Mark, P, Markau, S, Markovic, M, Marshall, C, Martin, M, Martinez, C, Martinez, S, Martins, G, Maruyama, K, Maruyama, S, Marx, K, Maselli, A, Masengu, A, Maskill, A, Masumoto, S, Masutani, K, Matsumoto, M, Matsunaga, T, Matsuoka, N, Matsushita, M, Matthews, M, Matthias, S, Matvienko, E, Maurer, M, Maxwell, P, Mazlan, N, Mazlan, SA, Mbuyisa, A, McCafferty, K, McCarroll, F, McCarthy, T, McClary-Wright, C, McCray, K, McDermott, P, McDonald, C, McDougall, R, McHaffie, E, McIntosh, K, McKinley, T, McLaughlin, S, McLean, N, McNeil, L, Measor, A, Meek, J, Mehta, A, Mehta, R, Melandri, M, Mené, P, Meng, T, Menne, J, Merritt, K, Merscher, S, Meshykhi, C, Messa, P, Messinger, L, Miftari, N, Miller, R, Miller, Y, Miller-Hodges, E, Minatoguchi, M, Miners, M, Minutolo, R, Mita, T, Miura, Y, Miyaji, M, Miyamoto, S, Miyatsuka, T, Miyazaki, M, Miyazawa, I, Mizumachi, R, Mizuno, M, Moffat, S, Mohamad Nor, FS, Mohamad Zaini, SN, Mohamed Affandi, FA, Mohandas, C, Mohd, R, Mohd Fauzi, NA, Mohd Sharif, NH, Mohd Yusoff, Y, Moist, L, Moncada, A, Montasser, M, Moon, A, Moran, C, Morgan, N, Moriarty, J, Morig, G, Morinaga, H, Morino, K, Morisaki, T, Morishita, Y, Morlok, S, Morris, A, Morris, F, Mostafa, S, Mostefai, Y, Motegi, M, Motherwell, N, Motta, D, Mottl, A, Moys, R, Mozaffari, S, Muir, J, Mulhern, J, Mulligan, S, Munakata, Y, Murakami, C, Murakoshi, M, Murawska, A, Murphy, K, Murphy, L, Murray, S, Murtagh, H, Musa, MA, Mushahar, L, Mustafa, R, Mustafar, R, Muto, M, Nadar, E, Nagano, R, Nagasawa, T, Nagashima, E, Nagasu, H, Nagelberg, S, Nair, H, Nakagawa, Y, Nakahara, M, Nakamura, J, Nakamura, R, Nakamura, T, Nakaoka, M, Nakashima, E, Nakata, J, Nakata, M, Nakatani, S, Nakatsuka, A, Nakayama, Y, Nakhoul, G, Naverrete, G, Navivala, A, Nazeer, I, Negrea, L, Nethaji, C, Newman, E, Ng, TJ, Ngu, LLS, Nimbkar, T, Nishi, H, Nishi, M, Nishi, S, Nishida, Y, Nishiyama, A, Niu, J, Niu, P, Nobili, G, Nohara, N, Nojima, I, Nolan, J, Nosseir, H, Nozawa, M, Nunn, M, Nunokawa, S, Oda, M, Oe, M, Oe, Y, Ogane, K, Ogawa, W, Ogihara, T, Oguchi, G, Ohsugi, M, Oishi, K, Okada, Y, Okajyo, J, Okamoto, S, Okamura, K, Olufuwa, O, Oluyombo, R, Omata, A, Omori, Y, Ong, LM, Ong, YC, Onyema, J, Oomatia, A, Oommen, A, Oremus, R, Orimo, Y, Ortalda, V, Osaki, Y, Osawa, Y, Osmond Foster, J, O'Sullivan, A, Otani, T, Othman, N, Otomo, S, O'Toole, J, Owen, L, Ozawa, T, Padiyar, A, Page, N, Pajak, S, Paliege, A, Pandey, A, Pandey, R, Pariani, H, Park, J, Parrigon, M, Passauer, J, Patecki, M, Patel, M, Patel, R, Patel, T, Patel, Z, Paul, R, Paulsen, L, Pavone, L, Peixoto, A, Peji, J, Peng, BC, Peng, K, Pennino, L, Pereira, E, Perez, E, Pergola, P, Pesce, F, Pessolano, G, Petchey, W, Petr, EJ, Pfab, T, Phelan, P, Phillips, R, Phillips, T, Phipps, M, Piccinni, G, Pickett, T, Pickworth, S, Piemontese, M, Pinto, D, Piper, J, Plummer-Morgan, J, Poehler, D, Polese, L, Poma, V, Postal, A, Pötz, C, Power, A, Pradhan, N, Pradhan, R, Preiss, E, Preston, K, Prib, N, Price, L, Provenzano, C, Pugay, C, Pulido, R, Putz, F, Qiao, Y, Quartagno, R, Quashie-Akponeware, M, Rabara, R, Rabasa-Lhoret, R, Radhakrishnan, D, Radley, M, Raff, R, Raguwaran, S, Rahbari-Oskoui, F, Rahman, M, Rahmat, K, Ramadoss, S, Ramanaidu, S, Ramasamy, S, Ramli, R, Ramli, S, Ramsey, T, Rankin, A, Rashidi, A, Raymond, L, Razali, WAFA, Read, K, Reiner, H, Reisler, A, Reith, C, Renner, J, Rettenmaier, B, Richmond, L, Rijos, D, Rivera, R, Rivers, V, Robinson, H, Rocco, M, Rodriguez-Bachiller, I, Rodriquez, R, Roesch, C, Roesch, J, Rogers, J, Rohnstock, M, Rolfsmeier, S, Roman, M, Romo, A, Rosati, A, Rosenberg, S, Ross, T, Roura, M, Roussel, M, Rovner, S, Roy, S, Rucker, S, Rump, L, Ruocco, M, Ruse, S, Russo, F, Russo, M, Ryder, M, Sabarai, A, Saccà, C, Sachson, R, Sadler, E, Safiee, NS, Sahani, M, Saillant, A, Saini, J, Saito, C, Saito, S, Sakaguchi, K, Sakai, M, Salim, H, Salviani, C, Sampson, A, Samson, F, Sandercock, P, Sanguila, S, Santorelli, G, Santoro, D, Sarabu, N, Saram, T, Sardell, R, Sasajima, H, Sasaki, T, Satko, S, Sato, A, Sato, D, Sato, H, Sato, J, Sato, T, Sato, Y, Satoh, M, Sawada, K, Schanz, M, Scheidemantel, F, Schemmelmann, M, Schettler, E, Schettler, V, Schlieper, GR, Schmidt, C, Schmidt, G, Schmidt, U, Schmidt-Gurtler, H, Schmude, M, Schneider, A, Schneider, I, Schneider-Danwitz, C, Schomig, M, Schramm, T, Schreiber, A, Schricker, S, Schroppel, B, Schulte-Kemna, L, Schulz, E, Schumacher, B, Schuster, A, Schwab, A, Scolari, F, Scott, A, Seeger, W, Segal, M, Seifert, L, Seifert, M, Sekiya, M, Sellars, R, Seman, MR, Shah, S, Shainberg, L, Shanmuganathan, M, Shao, F, Sharma, K, Sharpe, C, Sheikh-Ali, M, Sheldon, J, Shenton, C, Shepherd, A, Shepperd, M, Sheridan, R, Sheriff, Z, Shibata, Y, Shigehara, T, Shikata, K, Shimamura, K, Shimano, H, Shimizu, Y, Shimoda, H, Shin, K, Shivashankar, G, Shojima, N, Silva, R, Sim, CSB, Simmons, K, Sinha, S, Sitter, T, Sivanandam, S, Skipper, M, Sloan, K, Sloan, L, Smith, R, Smyth, J, Sobande, T, Sobata, M, Somalanka, S, Song, X, Sonntag, F, Sood, B, Sor, SY, Soufer, J, Sparks, H, Spatoliatore, G, Spinola, T, Squyres, S, Srivastava, A, Stanfield, J, Staylor, K, Steele, A, Steen, O, Steffl, D, Stegbauer, J, Stellbrink, C, Stellbrink, E, Stevenson, A, Stewart-Ray, V, Stickley, J, Stoffler, D, Stratmann, B, Streitenberger, S, Strutz, F, Stubbs, J, Stumpf, J, Suazo, N, Suchinda, P, Suckling, R, Sudin, A, Sugamori, K, Sugawara, H, Sugawara, K, Sugimoto, D, Sugiyama, H, Sugiyama, T, Sullivan, M, Sumi, M, Suresh, N, Sutton, D, Suzuki, H, Suzuki, R, Suzuki, Y, Swanson, E, Swift, P, Syed, S, Szerlip, H, Taal, M, Taddeo, M, Tailor, C, Tajima, K, Takagi, M, Takahashi, K, Takahashi, M, Takahashi, T, Takahira, E, Takai, T, Takaoka, M, Takeoka, J, Takesada, A, Takezawa, M, Talbot, M, Taliercio, J, Talsania, T, Tamori, Y, Tamura, R, Tamura, Y, Tan, CHH, Tan, EZZ, Tanabe, A, Tanabe, K, Tanaka, A, Tanaka, N, Tang, S, Tang, Z, Tanigaki, K, Tarlac, M, Tatsuzawa, A, Tay, JF, Tay, LL, Taylor, J, Taylor, K, Te, A, Tenbusch, L, Teng, KS, Terakawa, A, Terry, J, Tham, ZD, Tholl, S, Thomas, G, Thong, KM, Tietjen, D, Timadjer, A, Tindall, H, Tipper, S, Tobin, K, Toda, N, Tokuyama, A, Tolibas, M, Tomita, A, Tomita, T, Tomlinson, J, Tonks, L, Topf, J, Topping, S, Torp, A, Torres, A, Totaro, F, Toth, P, Toyonaga, Y, Tripodi, F, Trivedi, K, Tropman, E, Tschope, D, Tse, J, Tsuji, K, Tsunekawa, S, Tsunoda, R, Tucky, B, Tufail, S, Tuffaha, A, Turan, E, Turner, H, Turner, J, Turner, M, Tye, YL, Tyler, A, Tyler, J, Uchi, H, Uchida, H, Uchida, T, Udagawa, T, Ueda, S, Ueda, Y, Ueki, K, Ugni, S, Ugwu, E, Umeno, R, Unekawa, C, Uozumi, K, Urquia, K, Valleteau, A, Valletta, C, van Erp, R, Vanhoy, C, Varad, V, Varma, R, Varughese, A, Vasquez, P, Vasseur, A, Veelken, R, Velagapudi, C, Verdel, K, Vettoretti, S, Vezzoli, G, Vielhauer, V, Viera, R, Vilar, E, Villaruel, S, Vinall, L, Vinathan, 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Published: 2024
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2. Impact of primary kidney disease on the effects of empagliflozin in patients with chronic kidney disease: secondary analyses of the EMPA-KIDNEY trial

Author: Judge, PK, Staplin, N, Mayne, KJ, Wanner, C, Green, JB, Hauske, SJ, Emberson, JR, Preiss, D, Ng, SYA, Roddick, AJ, Sammons, E, Zhu, D, Hill, M, Stevens, W, Wallendszus, K, Brenner, S, Cheung, AK, Liu, ZH, Li, J, Hooi, LS, Liu, WJ, Kadowaki, T, Nangaku, M, Levin, A, Cherney, D, Maggioni, AP, Pontremoli, R, Deo, R, Goto, S, Rossello, X, Tuttle, KR, Steubl, D, Massey, D, Landray, MJ, Baigent, C, Haynes, R, Herrington, WG, Abat, S, Abd Rahman, R, Abdul Cader, R, Abdul Hafidz, MI, Abdul Wahab, MZ, Abdullah, NK, Abdul-Samad, T, Abe, M, Abraham, N, Acheampong, S, Achiri, P, Acosta, JA, Adeleke, A, Adell, V, Adewuyi-Dalton, R, Adnan, N, Africano, A, Agharazii, M, Aguilar, F, Aguilera, A, Ahmad, M, Ahmad, MK, Ahmad, NA, Ahmad, NH, Ahmad, NI, Ahmad Miswan, N, Ahmad Rosdi, H, Ahmed, I, Ahmed, S, Aiello, J, Aitken, A, AitSadi, R, Aker, S, Akimoto, S, Akinfolarin, A, Akram, S, Alberici, F, Albert, C, Aldrich, L, Alegata, M, Alexander, L, Alfaress, S, Alhadj Ali, M, Ali, A, Alicic, R, Aliu, 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Miyatsuka, T, Miyazaki, M, Miyazawa, I, Mizumachi, R, Mizuno, M, Moffat, S, Mohamad Nor, FS, Mohamad Zaini, SN, Mohamed Affandi, FA, Mohandas, C, Mohd, R, Mohd Fauzi, NA, Mohd Sharif, NH, Mohd Yusoff, Y, Moist, L, Moncada, A, Montasser, M, Moon, A, Moran, C, Morgan, N, Moriarty, J, Morig, G, Morinaga, H, Morino, K, Morisaki, T, Morishita, Y, Morlok, S, Morris, A, Morris, F, Mostafa, S, Mostefai, Y, Motegi, M, Motherwell, N, Motta, D, Mottl, A, Moys, R, Mozaffari, S, Muir, J, Mulhern, J, Mulligan, S, Munakata, Y, Murakami, C, Murakoshi, M, Murawska, A, Murphy, K, Murphy, L, Murray, S, Murtagh, H, Musa, MA, Mushahar, L, Mustafa, R, Mustafar, R, Muto, M, Nadar, E, Nagano, R, Nagasawa, T, Nagashima, E, Nagasu, H, Nagelberg, S, Nair, H, Nakagawa, Y, Nakahara, M, Nakamura, J, Nakamura, R, Nakamura, T, Nakaoka, M, Nakashima, E, Nakata, J, Nakata, M, Nakatani, S, Nakatsuka, A, Nakayama, Y, Nakhoul, G, Naverrete, G, Navivala, A, Nazeer, I, Negrea, L, Nethaji, C, Newman, E, Ng, TJ, Ngu, LLS, Nimbkar, T, Nishi, H, Nishi, M, Nishi, S, Nishida, Y, Nishiyama, A, Niu, J, Niu, P, Nobili, G, Nohara, N, Nojima, I, Nolan, J, Nosseir, H, Nozawa, M, Nunn, M, Nunokawa, S, Oda, M, Oe, M, Oe, Y, Ogane, K, Ogawa, W, Ogihara, T, Oguchi, G, Ohsugi, M, Oishi, K, Okada, Y, Okajyo, J, Okamoto, S, Okamura, K, Olufuwa, O, Oluyombo, R, Omata, A, Omori, Y, Ong, LM, Ong, YC, Onyema, J, Oomatia, A, Oommen, A, Oremus, R, Orimo, Y, Ortalda, V, Osaki, Y, Osawa, Y, Osmond Foster, J, O'Sullivan, A, Otani, T, Othman, N, Otomo, S, O'Toole, J, Owen, L, Ozawa, T, Padiyar, A, Page, N, Pajak, S, Paliege, A, Pandey, A, Pandey, R, Pariani, H, Park, J, Parrigon, M, Passauer, J, Patecki, M, Patel, M, Patel, R, Patel, T, Patel, Z, Paul, R, Paulsen, L, Pavone, L, Peixoto, A, Peji, J, Peng, BC, Peng, K, Pennino, L, Pereira, E, Perez, E, Pergola, P, Pesce, F, Pessolano, G, Petchey, W, Petr, EJ, Pfab, T, Phelan, P, Phillips, R, Phillips, T, Phipps, M, Piccinni, G, Pickett, T, Pickworth, S, Piemontese, M, Pinto, D, Piper, J, Plummer-Morgan, J, Poehler, D, Polese, L, Poma, V, Postal, A, Pötz, C, Power, A, Pradhan, N, Pradhan, R, Preiss, E, Preston, K, Prib, N, Price, L, Provenzano, C, Pugay, C, Pulido, R, Putz, F, Qiao, Y, Quartagno, R, Quashie-Akponeware, M, Rabara, R, Rabasa-Lhoret, R, Radhakrishnan, D, Radley, M, Raff, R, Raguwaran, S, Rahbari-Oskoui, F, Rahman, M, Rahmat, K, Ramadoss, S, Ramanaidu, S, Ramasamy, S, Ramli, R, Ramli, S, Ramsey, T, Rankin, A, Rashidi, A, Raymond, L, Razali, WAFA, Read, K, Reiner, H, Reisler, A, Reith, C, Renner, J, Rettenmaier, B, Richmond, L, Rijos, D, Rivera, R, Rivers, V, Robinson, H, Rocco, M, Rodriguez-Bachiller, I, Rodriquez, R, Roesch, C, Roesch, J, Rogers, J, Rohnstock, M, Rolfsmeier, S, Roman, M, Romo, A, Rosati, A, Rosenberg, S, Ross, T, Roura, M, Roussel, M, Rovner, S, Roy, S, Rucker, S, Rump, L, Ruocco, M, Ruse, S, Russo, F, Russo, M, Ryder, M, Sabarai, A, Saccà, C, Sachson, R, Sadler, E, Safiee, NS, Sahani, M, Saillant, A, Saini, J, Saito, C, Saito, S, Sakaguchi, K, Sakai, M, Salim, H, Salviani, C, Sampson, A, Samson, F, Sandercock, P, Sanguila, S, Santorelli, G, Santoro, D, Sarabu, N, Saram, T, Sardell, R, Sasajima, H, Sasaki, T, Satko, S, Sato, A, Sato, D, Sato, H, Sato, J, Sato, T, Sato, Y, Satoh, M, Sawada, K, Schanz, M, Scheidemantel, F, Schemmelmann, M, Schettler, E, Schettler, V, Schlieper, GR, Schmidt, C, Schmidt, G, Schmidt, U, Schmidt-Gurtler, H, Schmude, M, Schneider, A, Schneider, I, Schneider-Danwitz, C, Schomig, M, Schramm, T, Schreiber, A, Schricker, S, Schroppel, B, Schulte-Kemna, L, Schulz, E, Schumacher, B, Schuster, A, Schwab, A, Scolari, F, Scott, A, Seeger, W, Segal, M, Seifert, L, Seifert, M, Sekiya, M, Sellars, R, Seman, MR, Shah, S, Shainberg, L, Shanmuganathan, M, Shao, F, Sharma, K, Sharpe, C, Sheikh-Ali, M, Sheldon, J, Shenton, C, Shepherd, A, Shepperd, M, Sheridan, R, Sheriff, Z, Shibata, Y, Shigehara, T, Shikata, K, Shimamura, K, Shimano, H, Shimizu, Y, Shimoda, H, Shin, K, Shivashankar, G, Shojima, N, Silva, R, Sim, CSB, Simmons, K, Sinha, S, Sitter, T, Sivanandam, S, Skipper, M, Sloan, K, Sloan, L, Smith, R, Smyth, J, Sobande, T, Sobata, M, Somalanka, S, Song, X, Sonntag, F, Sood, B, Sor, SY, Soufer, J, Sparks, H, Spatoliatore, G, Spinola, T, Squyres, S, Srivastava, A, Stanfield, J, Staylor, K, Steele, A, Steen, O, Steffl, D, Stegbauer, J, Stellbrink, C, Stellbrink, E, Stevenson, A, Stewart-Ray, V, Stickley, J, Stoffler, D, Stratmann, B, Streitenberger, S, Strutz, F, Stubbs, J, Stumpf, J, Suazo, N, Suchinda, P, Suckling, R, Sudin, A, Sugamori, K, Sugawara, H, Sugawara, K, Sugimoto, D, Sugiyama, H, Sugiyama, T, Sullivan, M, Sumi, M, Suresh, N, Sutton, D, Suzuki, H, Suzuki, R, Suzuki, Y, Swanson, E, Swift, P, Syed, S, Szerlip, H, Taal, M, Taddeo, M, Tailor, C, Tajima, K, Takagi, M, Takahashi, K, Takahashi, M, Takahashi, T, Takahira, E, Takai, T, Takaoka, M, Takeoka, J, Takesada, A, Takezawa, M, Talbot, M, Taliercio, J, Talsania, T, Tamori, Y, Tamura, R, Tamura, Y, Tan, CHH, Tan, EZZ, Tanabe, A, Tanabe, K, Tanaka, A, Tanaka, N, Tang, S, Tang, Z, Tanigaki, K, Tarlac, M, Tatsuzawa, A, Tay, JF, Tay, LL, Taylor, J, Taylor, K, Te, A, Tenbusch, L, Teng, KS, Terakawa, A, Terry, J, Tham, ZD, Tholl, S, Thomas, G, Thong, KM, Tietjen, D, Timadjer, A, Tindall, H, Tipper, S, Tobin, K, Toda, N, Tokuyama, A, Tolibas, M, Tomita, A, Tomita, T, Tomlinson, J, Tonks, L, Topf, J, Topping, S, Torp, A, Torres, A, Totaro, F, Toth, P, Toyonaga, Y, Tripodi, F, Trivedi, K, Tropman, E, Tschope, D, Tse, J, Tsuji, K, Tsunekawa, S, Tsunoda, R, Tucky, B, Tufail, S, Tuffaha, A, Turan, E, Turner, H, Turner, J, Turner, M, Tye, YL, Tyler, A, Tyler, J, Uchi, H, Uchida, H, Uchida, T, Udagawa, T, Ueda, S, Ueda, Y, Ueki, K, Ugni, S, Ugwu, E, Umeno, R, Unekawa, C, Uozumi, K, Urquia, K, Valleteau, A, Valletta, C, van Erp, R, Vanhoy, C, Varad, V, Varma, R, Varughese, A, Vasquez, P, Vasseur, A, Veelken, R, Velagapudi, C, Verdel, K, Vettoretti, S, Vezzoli, G, Vielhauer, V, Viera, R, Vilar, E, Villaruel, S, Vinall, L, Vinathan, J, Visnjic, M, Voigt, E, von-Eynatten, M, Vourvou, M, Wada, J, Wada, T, Wada, Y, Wakayama, K, Wakita, Y, Walters, T, Wan Mohamad, WH, Wang, L, Wang, W, Wang, X, Wang, Y, Wanninayake, S, Watada, H, Watanabe, K, Watanabe, M, Waterfall, H, Watkins, D, Watson, S, Weaving, L, Weber, B, Webley, Y, Webster, A, Webster, M, Weetman, M, Wei, W, Weihprecht, H, Weiland, L, Weinmann-Menke, J, Weinreich, T, Wendt, R, Weng, Y, Whalen, M, Whalley, G, Wheatley, R, Wheeler, A, Wheeler, J, Whelton, P, White, K, Whitmore, B, Whittaker, S, Wiebel, J, Wiley, J, Wilkinson, L, Willett, M, Williams, A, Williams, E, Williams, K, Williams, T, Wilson, A, Wilson, P, Wincott, L, Wines, E, Winkelmann, B, Winkler, M, Winter-Goodwin, B, Witczak, J, Wittes, J, Wittmann, M, Wolf, G, Wolf, L, Wolfling, R, Wong, C, Wong, E, Wong, HS, Wong, LW, Wong, YH, Wonnacott, A, Wood, A, Wood, L, Woodhouse, H, Wooding, N, Woodman, A, Wren, K, Wu, J, Wu, P, Xia, S, Xiao, H, Xiao, X, Xie, Y, Xu, C, Xu, Y, Xue, H, Yahaya, H, Yalamanchili, H, Yamada, A, Yamada, N, Yamagata, K, Yamaguchi, M, Yamaji, Y, Yamamoto, A, Yamamoto, S, Yamamoto, T, Yamanaka, A, Yamano, T, Yamanouchi, Y, Yamasaki, N, Yamasaki, Y, Yamashita, C, Yamauchi, T, Yan, Q, Yanagisawa, E, Yang, F, Yang, L, Yano, S, Yao, S, Yao, Y, Yarlagadda, S, Yasuda, Y, Yiu, V, Yokoyama, T, Yoshida, S, Yoshidome, E, Yoshikawa, H, Young, A, Young, T, Yousif, V, Yu, H, Yu, Y, Yuasa, K, Yusof, N, Zalunardo, N, Zander, B, Zani, R, Zappulo, F, Zayed, M, Zemann, B, Zettergren, P, Zhang, H, Zhang, L, Zhang, N, Zhang, X, Zhao, J, Zhao, L, Zhao, S, Zhao, Z, Zhong, H, Zhou, N, Zhou, S, Zhu, L, Zhu, S, Zietz, M, Zippo, M, Zirino, F, and Zulkipli, FH
Published: 2024
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3. Climatic influence on sediment distribution and transport in the Thar Desert (Sindh and Cholistan, Pakistan)

Author: Usman, M, Clift, P, Pastore, G, Vezzoli, G, Ando', S, Barbarano, M, Vermeesch, P, Garzanti, E, Clift, PD, Usman, M, Clift, P, Pastore, G, Vezzoli, G, Ando', S, Barbarano, M, Vermeesch, P, Garzanti, E, and Clift, PD
Abstract: The Thar Desert is a major sediment depocenter located in southwestern Asia and bordering the Indus drainage system to its east. It is unclear where the sediment that built the desert is coming from, and when the desert experienced phases of construction. In particular, we seek to establish the role of the South Asian monsoon in the initial formation and subsequent expansion of the desert. Here we integrate bulk-petrography and heavy-mineral data with U-Pb ages of detrital zircon grains to understand how the Thar Desert relates to the major potential sediment sources in the Himalayan orogen and to the large rivers that adjoin it to the west and north. Bulk petrography and heavy-mineral data from eolian sand in Cholistan (NE Pakistan) show closer similarity with that of Himalayan tributaries than eolian sand in Sindh (S Pakistan), which contains heavy-mineral suites close to those of mainstream Indus sand largely supplied by erosion of the Karakorum and Kohistan ranges. Kohistan is a particularly rich source of heavy minerals and is thus over-represented in provenance budgets based on that proxy alone. U[sbnd]Pb ages of detrital-zircon fail to show a sharp difference between dune sands in Sindh and Cholistan but confirms a somewhat greater supply from the Himalaya in Cholistan and from the Karakorum, Kohistan, and Nanga Parbat in Sindh. Zircon ages are similar in Sindh desert sand and in the Indus Delta, and are most similar to deltaic sand dated as 7 ka or older in the deglacial period. In parallel, the age signature of Cholistan sand resembles more that of older river channels found along the northwestern edge of the desert (e.g., paleo-Ghaggar-Hakra) than that of modern Himalayan tributaries (e.g., Sutlej). Both Cholistan and Sindh sands suggest that sediment supply to the desert was greater during the early Holocene when the summer monsoon was stronger. The southwesterly summer monsoon was the most effective agent of eolian transport and recycling of Indus delta
Published: 2024

4. Fast-eroding Taiwan and transfer of orogenic sediment to forearc basins and trenches in the Philippine and South China seas

Author: Garzanti, E, Nayak, K, Padoan, M, Vezzoli, G, Resentini, A, Castelltort, S, Lin, A, Babonneau, N, Ratzov, G, Hsu, S, Huang, K, Garzanti E., Nayak K., Padoan M., Vezzoli G., Resentini A., Castelltort S., Lin A. T. -S., Babonneau N., Ratzov G., Hsu S. -K., Huang K. -F., Garzanti, E, Nayak, K, Padoan, M, Vezzoli, G, Resentini, A, Castelltort, S, Lin, A, Babonneau, N, Ratzov, G, Hsu, S, Huang, K, Garzanti E., Nayak K., Padoan M., Vezzoli G., Resentini A., Castelltort S., Lin A. T. -S., Babonneau N., Ratzov G., Hsu S. -K., and Huang K. -F.
Abstract: This article reviews what is known about sediment composition and transport within and offshore Taiwan Island, a place on Earth where ultrarapid erosion is induced by strong tectonic activity, powerful earthquakes, high relief, and intense precipitation during the passage of typhoons. Literature information is here integrated with new petrographic, heavy-mineral, clay-mineral, elemental-geochemistry, and Sr and Nd isotope-geochemistry data on river sands and fluvial, shelf, and deep-sea muds to obtain a general overview of sediment generation and offshore dispersal pathways. The Taiwan thrust belt is bracketed by two opposite-verging subduction zones, and orogenic detritus is transferred across rugged relief to surrounding forearc basins and trenches in the deep sea. Despite humid tropical climate, physical erosion is so fast that both sand petrography and clay mineralogy faithfully reflect the lithology of source rocks. In the Coastal Range, Miocene Luzon Arc andesites shed feldspatho-lithic volcaniclastic sediment rich in pyroxenes and smectite, whereas quartzo-lithic sand is recycled from the overlying Pliocene-Pleistocene sandstones. Basement rocks of the Tananao Complex supply quartzo-lithic metamorphiclastic sand with epidote and amphibole and mud rich in chlorite and illite. Lower Cenozoic mudrocks of the Slate Belt produce lithic to quartzo-lithic metasedimentaclastic sand with durable ZTR minerals and mostly illite, whereas upper Cenozoic strata of the Western Foothills shed recycled quartzo-lithic to litho-quartzose sedimentaclastic sand with ZTR minerals, garnet, and mixed clay-mineral assemblages. Scarce kaolinite, inconsistent behavior of mobile elements in sediment derived from opposite sides of Taiwan Island, and lack of correlation between geochemical and climatic parameters consistently indicate a weathering-limited regime. High weathering indices in sediment of western Taiwan rivers thus cannot be ascribed to present conditions but were largely inh
Published: 2023

5. Mapping Surface Features of an Alpine Glacier through Multispectral and Thermal Drone Surveys

Author: Rossini, M, Garzonio, R, Panigada, C, Tagliabue, G, Bramati, G, Vezzoli, G, Cogliati, S, Colombo, R, Di Mauro, B, Rossini M., Garzonio R., Panigada C., Tagliabue G., Bramati G., Vezzoli G., Cogliati S., Colombo R., Di Mauro B., Rossini, M, Garzonio, R, Panigada, C, Tagliabue, G, Bramati, G, Vezzoli, G, Cogliati, S, Colombo, R, Di Mauro, B, Rossini M., Garzonio R., Panigada C., Tagliabue G., Bramati G., Vezzoli G., Cogliati S., Colombo R., and Di Mauro B.
Abstract: Glacier surfaces are highly heterogeneous mixtures of ice, snow, light-absorbing impurities and debris material. The spatial and temporal variability of these components affects ice surface characteristics and strongly influences glacier energy and mass balance. Remote sensing offers a unique opportunity to characterize glacier optical and thermal properties, enabling a better understanding of different processes occurring at the glacial surface. In this study, we evaluate the potential of optical and thermal data collected from field and drone platforms to map the abundances of predominant glacier surfaces (i.e., snow, clean ice, melting ice, dark ice, cryoconite, dusty snow and debris cover) on the Zebrù glacier in the Italian Alps. The drone surveys were conducted on the ablation zone of the glacier on 29 and 30 July 2020, corresponding to the middle of the ablation season. We identified very high heterogeneity of surface types dominated by melting ice (30% of the investigated area), dark ice (24%), clean ice (19%) and debris cover (17%). The surface temperature of debris cover was inversely related to debris-cover thickness. This relation is influenced by the petrology of debris cover, suggesting the importance of lithology when considering the role of debris over glaciers. Multispectral and thermal drone surveys can thus provide accurate high-resolution maps of different snow and ice types and their temperature, which are critical elements to better understand the glacier’s energy budget and melt rates.
Published: 2023

6. Tracing erosion patterns in South Tibet: Balancing sediment supply to the Yarlung Tsangpo from the Himalaya versus Lhasa Block

Author: Liang, W, Garzanti, E, Hu, X, Resentini, A, Vezzoli, G, Yao, W, Liang W., Garzanti E., Hu X., Resentini A., Vezzoli G., Yao W., Liang, W, Garzanti, E, Hu, X, Resentini, A, Vezzoli, G, Yao, W, Liang W., Garzanti E., Hu X., Resentini A., Vezzoli G., and Yao W.
Abstract: The Yarlung Tsangpo, draining the Himalayan-Tibetan orogen along the Indus-Yarlung suture zone, receives detritus from the deformed remnants of both Indian margin to the south and Asian margin to the north. High resolution petrographic, heavy-mineral and geochemical datasets, combined with published geochronological data, allow us to monitor compositional changes, estimate erosion rates and investigate lithologic, climatic and anthropic controls on sediment fluxes along this large sediment-routing system entirely developed within high mountain areas. Sediment generated from the Lhasa Block along the southern margin of Asia is characterized by abundant feldspar and volcanic rock fragments, amphibole-dominated transparent heavy mineral suite and high concentrations of K, Rb, Be, Th and Pb. Himalayan-derived sand, instead, is characterized by sedimentary to low-rank metasedimentary rock fragments, poor transparent heavy mineral suite dominated by durable recycled (zircon, tourmaline) or metamorphic minerals (chloritoid, garnet) and high Ca concentration. Sand from the ophiolitic suture is distinguished by serpentinite grains and mafic volcanic or metavolcanic detritus, transparent heavy mineral suite including olivine, Cr-spinel, enstatite and clinopyroxene, and high Mg, Cr, Ni and low rare earth elements. Himalayan detritus is prominent in Yarlung Tsangpo upper reaches, whereas detritus from the Lhasa Block becomes progressively predominant in the middle and lower reaches. Provenance budgets based on integrated petrographic, heavy-mineral and geochemical datasets indicate that ca. 83% of the detritus is supplied by the Lhasa Block and the rest mostly from the Himalaya (ca. 12%) and subordinately from the ophiolitic suture (5%). A low average erosion rate of ca. 0.06 mm/a was estimated for the Yarlung Tsangpo catchment upstream of the Namche Barwa syntaxis, which resulted from dry climate, relatively mild average relief and sediment storage in wide valley tracts o
Published: 2022

7. Multi-proxy signatures in deep-water fine-grained sediments: Inherited versus syn-sedimentary factors controlling sediment compositions within a foredeep basin (Paleogene Adriatic foredeep – Northern Italy)

Author: Di Capua, A, Miano, S, Campagnolo, D, Bonomo, S, Caruso, A, Carugati, G, Cavallo, A, Livio, F, Vezzoli, G, Di Capua, Andrea, Miano, Samuele, Campagnolo, Davide, Bonomo, Sergio, Caruso, Antonio, Carugati, Gabriele, Cavallo, Alessandro, Livio, Franz A., Vezzoli, Giovanni, Di Capua, A, Miano, S, Campagnolo, D, Bonomo, S, Caruso, A, Carugati, G, Cavallo, A, Livio, F, Vezzoli, G, Di Capua, Andrea, Miano, Samuele, Campagnolo, Davide, Bonomo, Sergio, Caruso, Antonio, Carugati, Gabriele, Cavallo, Alessandro, Livio, Franz A., and Vezzoli, Giovanni
Abstract: This work explores the significance of sedimentary proxies recorded within the Paleogene fine-grained detritus of the Adriatic foredeep (Northern Italy). Such sequences represent the only sedimentary record describing the source-to-sink system delivering detritus from the growing Alpine belt to the basin during the Eocene – Oligocene boundary. The combination of x-ray powder diffractometric, petrographic, SEM-EDS and biostratigraphic analyses on more than twenty samples from the Ternate-Travedona Formation, the Chiasso Formation, and the Villa Olmo Conglomerate allowed to identify the source areas of detritus and potential fluvial paleo-drainages, possibly ascribable to two different sectors of the Southalpine domain. Comparison between these results and the petrographic composition of the Cretaceous wedge in the same area opens to the possibility that Cenozoic drainages were inherited from more limited paleo-drainages developed during the end of the Mesozoic. Results of provenance analysis also gives new insights on how detrital compositions may vary in function of grain-size in tropical-subtropical environments.
Published: 2023

8. Geosites in the Gole della Breggia Geopark, Ticino, Southern Switzerland

Author: Oppizzi, P, Pasquaré Mariotto, F, Stockar, R, Stella, A, Corti, N, Pedicini, M, Ando', S, Vezzoli, G, Bonali, F, Oppizzi, Paolo, Pasquaré Mariotto, Federico, Stockar, Rudolf, Stella, Andrea, Corti, Noemi, Pedicini, Martina, Ando', Sergio, Vezzoli, Giovanni, Bonali, Fabio Luca, Oppizzi, P, Pasquaré Mariotto, F, Stockar, R, Stella, A, Corti, N, Pedicini, M, Ando', S, Vezzoli, G, Bonali, F, Oppizzi, Paolo, Pasquaré Mariotto, Federico, Stockar, Rudolf, Stella, Andrea, Corti, Noemi, Pedicini, Martina, Ando', Sergio, Vezzoli, Giovanni, and Bonali, Fabio Luca
Abstract: This work is aimed at illustrating one of the most engaging geoparks in Europe, the Gole della Breggia Geopark. By following its geotrail, the visitors can observe a great deal of features that encompass a wide variety of sedimentary, stratigraphic, tectonic and paleogeographic processes. Its contents are popularized by a comprehensive geotrail that encompasses 23 geostops, enriched with explanatory panels, which enable viewers to gain an overall picture of the complex processes that led to the present-day configuration of the outcrops encountered in the geopark. We have selected five significative outcrops, which we have regarded as fully-fledged geosites, and which encompass the whole 170-million-year interval, from Early Jurassic to Late Miocene times, during which the rocks of the geopark were deposited and deformed. We have performed an assessment of the five geosites, using the most common criteria that can be applied for geosite evaluation. The results of the assessment show that the most valuable geosites are the Lombardian Maiolica, and the Limestone with Pelagic Bivalves. All five geosites, thanks to a QR code added to each figure along the text, can be viewed as virtual geosites (VGs), integrated by 360° videos.
Published: 2023

9. Long-term safety and efficacy of left atrial appendage occlusion in dialysis patients with atrial fibrillation: a multi-center, prospective, open label, observational study

Author: Genovesi, S, Porcu, L, Rebora, P, Slaviero, G, Casu, G, Bertoli, S, Airoldi, F, Buskermolen, M, Gallieni, M, Pieruzzi, F, Rovaris, G, Montoli, A, Piccaluga, E, Molon, G, Alberici, F, Adamo, M, Gaspardone, A, D'Angelo, G, Merella, P, Vezzoli, G, Trezzi, B, Mazzone, P, Genovesi, S, Porcu, L, Rebora, P, Slaviero, G, Casu, G, Bertoli, S, Airoldi, F, Buskermolen, M, Gallieni, M, Pieruzzi, F, Rovaris, G, Montoli, A, Piccaluga, E, Molon, G, Alberici, F, Adamo, M, Gaspardone, A, D'Angelo, G, Merella, P, Vezzoli, G, Trezzi, B, and Mazzone, P
Abstract: As children transition to adulthood, do they remain a major determinant of parental labor supply? To answer this question, we examine how college costs affect the labor supply of mothers and fathers by exploiting the roll-out of nine generous state merit aid programs in the United States from 1993 to 2004, which made college more affordable. Mothers of college-age children decreased their annual hours of work after the introduction of these state-wide programs, while fathers did not adjust their labor supply. Mothers of college-going children were entirely responsible for the decline in hours of work, where mothers of children who did not go to college experienced no change in hours of work. The decline in labor supply was mainly due to adjustments among high-income, married, more educated, and white mothers, whose labor supply was more elastic to college costs.
Published: 2023

10. The Timing of Collision Between Asia and the West Burma Terrane, and the Development of the Indo-Burman Ranges

Author: Najman, Y, Sobel, E, Millar, I, Luan, X, Zapata, S, Garzanti, E, Parra, M, Vezzoli, G, Zhang, P, Aung, D, Paw, S, Lwin, T, Sobel, ER, Luan, XW, Aung, DW, Paw, SMTL, Lwin, TN, Najman, Y, Sobel, E, Millar, I, Luan, X, Zapata, S, Garzanti, E, Parra, M, Vezzoli, G, Zhang, P, Aung, D, Paw, S, Lwin, T, Sobel, ER, Luan, XW, Aung, DW, Paw, SMTL, and Lwin, TN
Abstract: The West Burma Terrane (WBT) is a small terrane bounded to the east by the Asian Sibumasu Block and to the west by the Indo-Burman Ranges (IBR), the latter being an exhumed accretionary prism that formed during subduction of Indian oceanic lithosphere beneath Asia. Understanding the geological history of the WBT is important for reconstruction of the closure history of the Tethys Ocean and India-Asia collision. Currently there are major discrepancies in the proposed timings of collision between the WBT with both India and Asia; whether the WBT collided with India or Asia first is debated, and proposed timings of collisions stretch from the Mesozoic to the Cenozoic. We undertook a multi-technique provenance study involving petrography, detrital zircon U-Pb and Hf analyses, rutile U-Pb analyses and Sr-Nd bulk rock analyses on sediments of the Central Myanmar Basins of the WBT. We determined that the first arrival of Asian material into the basin occurred after the earliest late Eocene and by the early Oligocene, thus placing a minimum constraint on the timing of WBT-Asia collision. Our low temperature thermochronological study of the IBR records two periods of exhumation, in the early-middle Eocene, and at the Oligo-Miocene boundary. The Eocene event may be associated with the collision of the WBT with India. The later event at the Oligo-Miocene boundary may be associated with changes in wedge dynamics resulting from increased sediment supply to the system; however a number of other possible causes provide equally plausible explanations for both events.
Published: 2022

11. Andean retroarc-basin dune fields and Pampean Sand Sea (Argentina): Provenance and drainage changes driven by tectonics and climate

Author: Garzanti, E, Capaldi, T, Tripaldi, A, Zarate, M, Limonta, M, Vezzoli, G, Garzanti, E, Capaldi, T, Tripaldi, A, Zarate, M, Limonta, M, and Vezzoli, G
Abstract: We here review in terms of tectono-magmatic setting and Quaternary landscape dynamics what is known about the provenance of Argentine dune fields and their fluvial feeder systems draining the Andean Cordillera. The detrital signatures of these eolian sediments were previously investigated based on either framework petrography and heavy minerals or detrital-zircon geochronology, and their peculiar volcaniclastic nature was long recognized. Compositional variability, however, was only broadly evaluated, and quantitative provenance analysis based on a systematic multimethod approach across the entire region was not carried out so far. For this reason, here we integrate original and previously obtained petrographic, heavy-mineral, and detrital-zircon geochronology data to present the first comprehensive provenance study of dune fields stretching for 1000 km across central Argentina from the Andean piedmont to the Atlantic Ocean. In dune fields along the Andean retroarc basin, sediment composition defines a steady northward decrease in volcanic detritus. This reflects active magmatism in the Southern Volcanic Zone and Payenia province (38°-34°S), in contrast with the ~600-km-long Pliocene-Quaternary magmatic gap in the Pampean flat-slab segment (33°-27°S), where sediment is derived from deeper-seated tectono-stratigraphic levels of the continental arc and uplifted blocks of retroarc-basin basement. In distal Pampean lowlands extending across the bulge and backbulge depozones, instead, sand dunes display notably homogeneous compositional signatures, indicating that detritus was mostly generated north of 34°S and transported by a paleo-Desaguadero trunk river that formed during southward-progressing diachronous uplift of the Sierras Pampeanas since the late Miocene. In contrast with huge African and Arabian deserts that contain multiply recycled quartzose to pure quartzose sand, even very fragile volcanic clasts, plagioclase feldspar, and unstable ferromagnesian minerals a
Published: 2022

12. Interplay of Holocene surface faulting and climate in the Central Po Plain, Italy

Author: Zanchi, A, Ravazzi, C, Cavallin, A, Deaddis, M, De Amicis, M, Arosio, T, Marchetti, M, Vezzoli, G, De Amicis M, Zanchi, A, Ravazzi, C, Cavallin, A, Deaddis, M, De Amicis, M, Arosio, T, Marchetti, M, Vezzoli, G, and De Amicis M
Abstract: Understanding the recent events marking the late Quaternary history of the Po Plain (N-Italy) is of overriding importance to decipher the record of depositional versus erosional phases, and their interplay with climatic, tectonic, and human forcing. We reconstructed the structural setting and chronostratigraphy of a Holocene succession crosscut by a thrust fault located south of Montodine (Cremona, Italy) within the Po Plain. The fault shows a maximum displacement up to one meter. Radiocarbon dating fixes a minimum age of 11.9 cal ka BP for the postglacial river entrenchment and constrains the fault movement age between 5.9 and 3.4 cal ka BP. Undeformed Late Medieval coarse gravels cover the faulted succession. Due to the outcrop position, lying above the buried frontal thrusts of the Southern Alps and North Apennines, we propose that faulting results from secondary surface effects induced by seismic shaking. We discuss two main mechanisms, both related to lateral spreading, that can result in the formation of reverse faults close to the surface. The Soncino area, recording one of the strongest historical earthquakes of the central Po Plain (1802), is considered as a possible source for seismic shaking. The results of this study are a contribution for the assessment of the potential seismic hazard in one of the most populated regions of Europe.
Published: 2022

13. Interplay of Holocene surface faulting and climate in the Central Po Plain, Italy

Author: Andrea Zanchi, Cesare Ravazzi, Angelo Cavallin, Massimiliano Deaddis, Mattia De Amicis, Tito Arosio, Mauro Marchetti, Giovanni Vezzoli, Zanchi, A, Ravazzi, C, Cavallin, A, Deaddis, M, De Amicis, M, Arosio, T, Marchetti, M, and Vezzoli, G
Subjects: Arts and Humanities (miscellaneous), Holocene, Po Plain, GEO/03 - GEOLOGIA STRUTTURALE, GEO/04 - GEOGRAFIA FISICA E GEOMORFOLOGIA, Paleo-liquefaction, General Earth and Planetary Sciences, Thrust faulting, River diversion, Earth-Surface Processes, River entrenchment, Seismic hazard
Abstract: Understanding the recent events marking the late Quaternary history of the Po Plain (N-Italy) is of overriding importance to decipher the record of depositional versus erosional phases, and their interplay with climatic, tectonic, and human forcing. We reconstructed the structural setting and chronostratigraphy of a Holocene succession crosscut by a thrust fault located south of Montodine (Cremona, Italy) within the Po Plain. The fault shows a maximum displacement up to one meter. Radiocarbon dating fixes a minimum age of 11.9 cal ka BP for the postglacial river entrenchment and constrains the fault movement age between 5.9 and 3.4 cal ka BP. Undeformed Late Medieval coarse gravels cover the faulted succession. Due to the outcrop position, lying above the buried frontal thrusts of the Southern Alps and North Apennines, we propose that faulting results from secondary surface effects induced by seismic shaking. We discuss two main mechanisms, both related to lateral spreading, that can result in the formation of reverse faults close to the surface. The Soncino area, recording one of the strongest historical earthquakes of the central Po Plain (1802), is considered as a possible source for seismic shaking. The results of this study are a contribution for the assessment of the potential seismic hazard in one of the most populated regions of Europe.
Published: 2022
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14. Beyond the kidneys: investigating airway involvement in autosomal dominant polycystic kidney disease through nasal nitric oxide profiling : A transversal single-center observational study.

Author: De Rosa LI, Vespa M, Catania M, Kola K, Bucci R, Mancassola G, Carrera P, Manunta P, Vezzoli G, and Sciarrone Alibrandi MT
Published: 2024
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15. Areas of uncertainty on the diagnosis, treatment, and follow-up of hypophosphatemia in adults: an Italian Delphi consensus.

Author: Chiodini I, d'Angela D, Falchetti A, Gennari L, Malavolta N, Masi L, Migliore A, Orso M, Polistena B, Rendina D, Scillitani A, Spandonaro F, Vezzoli G, and Vescini F
Abstract: Purpose: The study aimed to present the results of a Delphi consensus involving Italian experts focusing on the management of hypophosphatemia in adults., Methods: A multidisciplinary advisory board of nine physicians, experts in hypophosphatemia management, was established. Next, a literature search was performed to identify international guidelines, consensus, and clinical pathways, which were later presented to the advisory board. Collaboratively, the advisory board and authoring team selected key statements for the consensus process and focused on areas of uncertainty related to the management of hypophosphatemia. The advisory board also indicated the experts to be invited to participate in the consensus process. The Delphi method was employed to reach a consensus., Results: The literature search yielded one guideline, five consensus documents, and one clinical pathway. While our search strategy aimed to identify documents on the management of all types of hypophosphatemia, most of the guidelines and consensus documents retrieved focused on X-linked hypophosphatemia. The consensus process focused on 11 key issues, achieving strong convergence (over 70% consensus) in the first Delphi round for 8 out of the 11 statements. Three statements proceeded to the second round, with strong agreement reached for two. Notably, consensus was not reached for the statement concerning the measurement of fibroblast growth factor 23 for diagnostic purposes., Conclusion: The study revealed that the community of clinical experts is well-informed and in agreement regarding hypophosphatemia management. It emphasized the importance of developing clear national guidance documents to support clinicians and multidisciplinary teams in patient management. These documents are crucial not only for healthcare professionals but also for those responsible for defining pathways and services, facilitating a more accurate management of hypophosphatemic patients., (© 2024. The Author(s).)
Published: 2024
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16. Highlighting the Impact of Hormonal Factors on Hepatic Cystogenesis: Implications for Pathophysiology and Clinical Practice.

Author: Catania M, Vezzoli G, and Alibrandi MTS
Published: 2024
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17. Is denosumab an efficient and safe drug for osteoporosis in dialysis patients? Considerations and state of the art about its use in this setting.

Author: Simonini M, Bologna A, and Vezzoli G
Subjects: Humans, Renal Insufficiency, Chronic complications, Chronic Kidney Disease-Mineral and Bone Disorder drug therapy, Chronic Kidney Disease-Mineral and Bone Disorder etiology, Female, Denosumab therapeutic use, Bone Density Conservation Agents therapeutic use, Renal Dialysis, Osteoporosis drug therapy, Bone Density drug effects
Abstract: Purpose: In patients with chronic kidney disease (CKD), renal osteodystrophy may be associated with a progressive bone mass loss that increases fracture risk. Denosumab, a monoclonal antibody inhibiting osteoclast activity, is an antiresorptive medication used for the treatment osteoporosis., Methods: Its efficacy and safety were initially established in the FREEDOM study, showing a significant reduction in incident fractures in osteoporotic women treated with denosumab. Subsequent post hoc analyses showed its efficacy in patients stratified by kidney function, but these analyses did not include patients with advanced stages of CKD. The capability of denosumab in improving bone mineral density in uremic patients was evaluated in 12 studies including 461 dialysis patients with low bone mineral density. The improvement of bone mineral density was the final end point in these studies assessed during a follow-up of 6-60 months. Nine of these studies did not have hyperparathyroidism among criteria for patient inclusion and their participants may have low-turnover bone disease. Despite current recommendations, no patients underwent bone biopsy before denosumab therapy., Results: Overall, findings in these studies suggest that denosumab is a viable option for promoting bone mass recovery in patients with advanced stages of CKD having either high or low serum levels of PTH. However, the increase of bone mineral density was lower in patients with low serum markers of bone turnover at baseline. These studies also highlighted the need for calcium and vitamin D supplementation to prevent hypocalcemia that remains a serious concern., Conclusions: Denosumab emerges as a potentially safe and effective option for enhancing bone health in CKD patients., (© 2024. The Author(s), under exclusive licence to Springer Nature B.V.)
Published: 2024
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18. [ADPKD and IPMN: Mere Coincidence or Double Trouble?]

Author: Kola K, De Rosa LI, Catania M, Brambilla Pisoni M, Tunesi F, Farinone S, Petrone M, Manunta P, Vezzoli G, and Sciarrone Alibrandi MT
Subjects: Humans, Adenocarcinoma, Mucinous complications, Adenocarcinoma, Mucinous diagnosis, Adenocarcinoma, Mucinous pathology, Carcinoma, Papillary complications, Carcinoma, Papillary diagnosis, Carcinoma, Papillary pathology, Pancreatic Intraductal Neoplasms complications, Pancreatic Intraductal Neoplasms diagnosis, Pancreatic Intraductal Neoplasms pathology, Polycystic Kidney, Autosomal Dominant complications, Polycystic Kidney, Autosomal Dominant genetics
Abstract: This article constitutes a review of the existing literature on the potential correlation between autosomal dominant polycystic kidney disease (ADPKD) and intraductal papillary mucinous neoplasms (IPMN) of the pancreas. Additionally, it presents a clinical case where familiarity for both pathologies was observed, derived from the direct experience of our clinic, reinforcing the hypothesis of a possible common pathogenetic pathway. The review focuses on the potential genetic correlation between these two pathologies within the realm of ciliopathies, emphasizing the importance of targeted screening and monitoring strategies to detect pancreatic complications early in patients with ADPKD. Furthermore, it highlights the complexity in the clinical management of these rare conditions and underscores the importance of early diagnosis in optimizing clinical outcomes., (Copyright by Società Italiana di Nefrologia SIN, Rome,Italy.)
Published: 2024
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19. Unveiling Neglected Concerns: Possible Severe Hepatic Complications after Nephrectomy in Autosomal Dominant Polycystic Kidney Disease - A Case Report.

Author: De Rosa LI, Catania M, Tunesi F, Vespa M, Bucci R, Kola K, Vezzoli G, and Sciarrone Alibrandi MT
Abstract: Introduction: Autosomal dominant polycystic kidney disease (ADPKD) is the most common genetic kidney disease and the 4th leading cause of renal replacement therapy in the world. ADPKD is a systemic disorder as cysts may develop in several organs. Liver cysts are the most common extrarenal manifestations and are often incidentally detected. Even though cysts do not influence liver function, they can grow to a very great size and can significantly enlarge liver volume, causing structural distortion of the biliary tree and patient discomfort due to the mass effect. Nephrectomy is frequently considered in preparation for renal transplantation in patients with remarkable kidneys' enlargement. There are currently no globally recognized clinical guidelines for nephrectomy. Although cysts do not normally affect liver function in ADPKD, after nephrectomy cases of liver fibrosis and Budd-Chiari have been reported. These are uncommon disorders due to the obstruction of the blood flow in the hepatic venous causing spleen and liver volume enlargement, portal hypertension, and hepatic cirrhosis., Case Presentation: We present a case of hepatic fibrosis with splenomegaly and severe pancytopenia as a tardive complication after bilateral nephrectomy in 47-year-old ADPKD patient., Conclusion: This finding underscores the critical significance of meticulously examining the anatomical relationship between polycystic kidneys and the liver before performing nephrectomy. Additionally, it highlights the importance of assessing liver involvement and associated complications. By integrating liver assessment into the criteria, we can significantly enhance patient care and improve the overall management of ADPKD before kidney transplantation., Competing Interests: All authors declare that they have no conflicts of interest., (© 2024 The Author(s). Published by S. Karger AG, Basel.)
Published: 2024
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20. Assessment and management of heart failure in patients with chronic kidney disease.

Author: Guaricci AI, Sturdà F, Russo R, Basile P, Baggiano A, Mushtaq S, Fusini L, Fazzari F, Bertandino F, Monitillo F, Carella MC, Simonini M, Pontone G, Ciccone MM, Grandaliano G, Vezzoli G, and Pesce F
Subjects: Humans, Biomarkers, Heart Failure therapy, Heart Failure drug therapy, Renal Insufficiency, Chronic complications, Renal Insufficiency, Chronic epidemiology
Abstract: Heart failure (HF) and chronic kidney disease (CKD) are two pathological conditions with a high prevalence in the general population. When they coexist in the same patient, a strict interplay between them is observed, such that patients affected require a clinical multidisciplinary and personalized management. The diagnosis of HF and CKD relies on signs and symptoms of the patient but several additional tools, such as blood-based biomarkers and imaging techniques, are needed to clarify and discriminate the main characteristics of these diseases. Improved survival due to new recommended drugs in HF has increasingly challenged physicians to manage patients with multiple diseases, especially in case of CKD. However, the safe administration of these drugs in patients with HF and CKD is often challenging. Knowing up to which values of creatinine or renal clearance each drug can be administered is fundamental. With this review we sought to give an insight on this sizable and complex topic, in order to get clearer ideas and a more precise reference about the diagnostic assessment and therapeutic management of HF and CKD., (© 2023. The Author(s).)
Published: 2024
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21. Role of Female Sex Hormones in ADPKD Progression and a Personalized Approach to Contraception and Hormonal Therapy.

Author: Petrone M, Catania M, De Rosa LI, Degliuomini RS, Kola K, Lupi C, Brambilla Pisoni M, Salvatore S, Candiani M, Vezzoli G, and Sciarrone Alibrandi MT
Abstract: This review navigates the intricate relationship between gender, hormonal influences, and the progression of autosomal dominant polycystic kidney disease (ADPKD), highlighting the limited literature on this crucial topic. The study explores the impact of female sex hormones on liver and renal manifestations, uncovering gender-specific differences in disease progression. Actually, hormonal therapy in women with ADPKD remains a challenging issue and is a source of concern regarding its potential impact on disease outcomes, particularly at the hepatic level. Notably, women with ADPKD exhibit a slower renal disease progression compared to men, attributed to hormonal dynamics. This review sheds light on the role of estrogen in regulating pathways of the renin-angiotensin-aldosterone system, revealing its complex interplay and implications for cardiovascular and renal health. Therapeutic considerations for fertile women with ADPKD, including contraception options, are discussed, emphasizing the necessity for personalized approaches. In the postmenopausal phase, the review evaluates the role of hormonal replacement therapy, considering its potential benefits and risks in the context of ADPKD. The review concludes by underscoring the imperative need for tailored treatment approaches for ADPKD patients, considering individual risks and benefits. The scarcity of literature underlines the call for further research to enhance our understanding of optimal hormonal therapies in the context of ADPKD, ultimately paving the way for innovative and personalized therapeutic interventions.
Published: 2024
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22. The Implications of the Vitamin D Metabolite Diagnostic Ratio in the Assessment of Patients With Idiopathic Hypercalciuria.

Author: Dos Santos Junior ACS, Foligno NE, and Vezzoli G
Published: 2024
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23. [The Management of Patients with Adult Autosomal Dominant Polycystic Kidney Disease (ADPKD) Requires a Multidisciplinary Approach].

Author: Bucci R, De Rosa LI, Brambilla Pisoni M, Farinone S, Vespa M, Joli G, Catania M, Kola K, Tunesi F, Brioni E, Carrera P, Mancassola G, Citterio L, Manunta P, Vezzoli G, and Sciarrone Alibrandi MT
Subjects: Adult, Humans, Kidney, Polycystic Kidney, Autosomal Dominant complications, Polycystic Kidney, Autosomal Dominant therapy, Kidney Failure, Chronic etiology
Abstract: Autosomal dominant polycystic kidney disease (ADPKD) is the most common genetic kidney disease. Its main feature is the progressive enlargement of both kidneys with progressive loss of kidney function. ADPKD is the fourth leading cause of terminal renal failure in the world. Even today there are still uncertainties and poor information. Patients too often have a renunciatory and passive attitude toward the disease. However, there are currently no internationally accepted clinical practice guidelines, and there are significant regional variations in approaches to the diagnosis, clinical evaluation, prevention, and treatment of ADPKD. Therefore, we believe it is important to point out the conduct of our specialist outpatient clinic for ADPKD, which from the beginning has developed a multidisciplinary approach (nephrologists, geneticists, psychologists, radiologists, nutritionists) to face the disease at 360° and therefore not only from a purely nephrological point of view. Such a strategy not only enables patients to receive a timely and accurate diagnosis of the disease, but also ensures that they will receive a thorough and focused follow-up over time, that can prevent or at least slow down the disease in its evolution providing patients with a serene awareness of their condition as much as possible., (Copyright by Società Italiana di Nefrologia SIN, Rome,Italy.)
Published: 2023

24. Congenital solitary kidney in autosomal dominant polycystic kidney disease: Where do known genes end and the unknown begin?

Author: Bucci R, Tunesi F, De Rosa LI, Carrera P, Mancassola G, Catania M, Vezzoli G, and Sciarrone Alibrandi MT
Abstract: We present the case of a 41-year-old man patient diagnosed with solitary left kidney with few cysts. He has a family history of unilateral renal agenesis (URA) but no for autosomal dominant polycystic kidney disease (ADPKD). Genetic testing revealed PKD1 gene intron 11 heterozygous nucleotide variant c.2854-23G>T, but no gene mutation implicated in URA. Just eight cases of ADPKD with one kidney have been recorded globally. PC1 and PC2 disruption, causing primary cilia malformation or absence resulting in relevant in the first embryonic development alteration. Cillia's crucial significance in many diseases will require more research., Competing Interests: All authors declare that they have no conflicts of interest., (© 2023 The Authors. Clinical Case Reports published by John Wiley & Sons Ltd.)
Published: 2023
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25. [The Educational Aspect in Promoting a Low-Sodium Diet, Physical Activity and Therapy Adherence among Patients with Autosomal Dominant Polycystic Disease: A Literature Review].

Author: Brioni E, Villa G, Manara DF, Farinone S, Magnaghi C, Scuratti D, Moranda D, Galli E, Sciarrone MT, and Vezzoli G
Subjects: Humans, Diet, Sodium-Restricted, Disease Progression, Exercise, Polycystic Kidney, Autosomal Dominant drug therapy, Kidney Failure, Chronic etiology, Kidney Failure, Chronic therapy
Abstract: Background. Polycystic kidney disease (ADPKD) is the most common monogenic cause of End Stage Renal Disease (ESRD), and, thus, of kidney transplantation and dialysis. Educational interventions aimed to improve adherence to therapy, physical performance, and adequate food intake in patients can slow down disease progression by developing self-care skills, which are useful to promote their autonomy while aligning their life plans and required treatments. The aim of this review is to analyze the adherence of patients with polycystic kidney to pharmacological therapy, low-sodium diet, and physical activity, as evidenced in the clinical literature to guide structured educational interventions. Methods. We conducted a literature review from 01/09/2021 to 30/12/2022 through the combination of free keywords and MeSH terms on the databases: PubMed, CINAHL and Cochrane. Results. Findings in medical literature show that physical activity can improve blood pressure control and a low-sodium diet can slow down the progression towards ESRD. Furthermore, although patients may adhere to the complex drug therapy, unresolved educational demands concern choices and behaviors of daily life that, involving the sphere of feelings and emotions, can evolve into manifestations of anxiety and stress. Conclusion. Among ADPKD patients a personalized educational support, considering disease stage and psychological factors, may enable them to acquire knowledge, skills, and behaviors that can improve clinical outcomes., (Copyright by Società Italiana di Nefrologia SIN, Rome,Italy.)
Published: 2023

26. Long-term safety and efficacy of left atrial appendage occlusion in dialysis patients with atrial fibrillation: a multi-center, prospective, open label, observational study.

Author: Genovesi S, Porcu L, Rebora P, Slaviero G, Casu G, Bertoli S, Airoldi F, Buskermolen M, Gallieni M, Pieruzzi F, Rovaris G, Montoli A, Piccaluga E, Molon G, Alberici F, Adamo M, Gaspardone A, D'Angelo G, Merella P, Vezzoli G, Trezzi B, and Mazzone P
Abstract: Background: The prevalence of atrial fibrillation (AF) in end stage kidney disease (ESKD) patients undergoing dialysis is high, however, the high risk of bleeding often hampers with a correct anticoagulation in ESKD patients with AF, despite high thromboembolic risk. Left atrial appendage (LAA) occlusion is a anticoagulation (OAT) for thromboembolism prevention in AF populations with high hemorrhagic risk., Methods and Results: The purpose of the study was to evaluate the efficacy and safety of LAA occlusion in a cohort of dialysis patients undergoing the procedure (LAA occlusion cohort, n = 106), in comparison with two other ESKD cohorts, one taking warfarin (Warfarin cohort, n = 114) and the other without anticoagulation therapy (No-OAT cohort, n = 148). After a median follow-up of 4 years, a Cox regression model, adjusted for possible confounding factors, showed that the hazard ratios (HRs) of thromboembolic events in the LAA occlusion cohort were 0.19 (95%CI 0.04-0.96; p = 0.045) and 0.16 (95%CI 0.04-0.66; p = 0.011) as compared with Warfarin and No-OAT cohorts, respectively. The HR of bleeding in the LAA occlusion cohort was 0.37 (95%CI 0.16-0.83; p = 0.017) compared to Warfarin cohort, while there were no significant differences between the LAA occlusion and the No-OAT cohort (HR 0.51; 95%CI 0.23-1.12; p = 0.094). Adjusted Cox regression models showed lower mortality in patients undergoing LAA occlusion as compared with both the Warfarin cohort (HR 0.60; 95%CI 0.38-0.94; p = 0.027) and no-OAT cohort (HR 0.52; 95%CI 0.34-0.78; p = 0.002). Thromboembolic events in the LAA occlusion cohort were lower than expected according to the CHA 2 DS 2 VASc score (1.7 [95%CI 0.3-3.0] vs 6.7 events per 100 person/years, p < 0.001)., Conclusion: In ESKD patients with AF, LAA occlusion is safe and effective and is associated with reduced mortality compared with OAT or no therapy., Competing Interests: (Honoraria for speaking at symposia or consultation). S.G.: Boston Scientific; G.C.: Abbott Structural Heart, Boston Scientific, Lifetech; G.R.: Boston Scientific, Abbott Structural Heart, Biosense, Medtronic, Biotronik, Hylomorph ; G.M.: Boston Scientific; M.A.: Abbott Structural Heart, Medtronic, Edwards Lifesciences; P.M.: Abbott Vascular, Boston Scientific. Other authors report no relationships that could be construed as a conflict of interest., (© The Author(s) 2023. Published by Oxford University Press on behalf of the ERA.)
Published: 2023
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27. Diagnostic policies on nephrolithiasis/nephrocalcinosis of possible genetic origin by Italian nephrologists: a survey by the Italian Society of Nephrology with an emphasis on primary hyperoxaluria.

Author: Ferraro PM, Caletti C, Capolongo G, Lombardi M, Scolari F, Vezzoli G, Vitale C, and Gambaro G
Subjects: Humans, Nephrologists, Oxalates, Nephrocalcinosis diagnosis, Nephrocalcinosis epidemiology, Nephrocalcinosis genetics, Hyperoxaluria, Primary complications, Hyperoxaluria, Primary diagnosis, Hyperoxaluria, Primary epidemiology, Nephrology, Kidney Calculi complications
Abstract: Background: Primary hyperoxaluria is a genetic disorder of the metabolism of glyoxylate, the precursor of oxalate. It is characterized by high endogenous production and excessive urinary excretion of oxalate, resulting in the development of calcium oxalate nephrolithiasis, nephrocalcinosis, and, in severe cases, end-stage kidney disease and systemic oxalosis. Three different forms of primary hyperoxaluria are currently known, each characterized by a specific enzymatic defect: type 1 (PH1), type 2 (PH2), and type 3 (PH3). According to currently available epidemiological data, PH1 is by far the most common form (about 80% of cases), and is caused by a deficiency of the hepatic enzyme alanine:glyoxylate aminotransferase., Methods: A survey on rare forms of nephrolithiasis and nephrocalcinosis with a focus on primary hyperoxaluria in the setting of Italian Nephrology and Dialysis Centers, using an online questionnaire, was recently conducted by the Project Group "Rare Forms of Nephrolithiasis and Nephrocalcinosis" of the Italian Society of Nephrology, with the aim of assessing the impact and management of this disorder in clinical practice in Italy., Results: Forty-five public and private Italian Centers participated in the survey, and responses to the questionnaire were provided by 54 medical professionals. The survey results indicate that 21 out of the 45 participating Centers are managing or have managed primary hyperoxaluria patients, most of whom are on dialysis, or are recipients of kidney transplants., Conclusions: The data of this survey indicate the need to implement genetic testing in suspected cases of primary hyperoxaluria, not only in the setting of dialysis or transplantation, but also with the aim of encouraging early diagnosis of PH1, which is the only type of primary hyperoxaluria for which specific drug therapy is currently available., (© 2023. The Author(s).)
Published: 2023
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28. Different Efficacy of Burosumab on Physical Performance and Serum Phosphate in Adult Patients with X-Linked Hyphophosphatemic Rickets during the First Six-Month of Treatment.

Author: Arcidiacono T, Foligno NE, Brioni E, Bologna A, Weber G, Mora S, Pitea M, Vitale C, and Vezzoli G
Abstract: Burosumab is a monoclonal anti-FGF23 antibody used to treat patients with X-linked hypophosphatemic rickets (XLH). Its effect on serum phosphate and physical performance was compared in patients during a 6-month treatment with burosumab. Eight adult patients with XHL were treated with burosumab (1 mg/kg s.c. every 28 days). In the first 6 months of treatment, calcium-phosphate metabolism variables were measured, and muscle performance (tested with chair and walking test) and quality of life (tested with fatigue, BPI-pain and BPI-life questionnaires) were estimated. A significant increase in serum phosphate was observed during the treatment. From the 16th week, serum phosphate became significantly lower than its value in the 4th week. No patients had serum phosphate below the normal range at the 10th week, but seven patients were hypophosphatemic in the 20th and 24th week. All patients improved the execution time of the chair test and walking test, which reached a plateau after the 12th week. BPI-pain and BPI-life scores significantly decreased from baseline to the 24th week. In conclusion, a six-month burosumab treatment may significantly improve the general condition and physical performance of adult patients with XLH; this improvement was more stable and more indicative of treatment efficacy than that of serum phosphate.
Published: 2023
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29. [Anti-angiogenic drugs and hypertension: from multidisciplinary collaboration to greater care].

Author: Tunesi F, Simonini M, Sabetta G, Bergamini A, Cioffi R, Raiabiotti E, Manunta P, Vezzoli G, Mangili G, and Lanzani CL
Subjects: Humans, Female, Angiogenesis Inhibitors adverse effects, Bevacizumab adverse effects, Vascular Endothelial Growth Factor A metabolism, Vascular Endothelial Growth Factor A therapeutic use, Retrospective Studies, Neoplasms, Hypertension drug therapy
Abstract: Anti-angiogenic drugs are widely used in cancer therapy. Their main targets of action are the vascular endothelial growth factor (VEGF) and its receptors (VEGF-R). Anti-angiogenic drugs are used to reduce the growth of the tumor and its metastases by acting on the phenomenon of tumor neo-angiogenesis. However, they are known for their side effects such as hypertension, acute kidney injury (AKI), and congestive heart failure. Methods: retrospective study conducted on 57 consecutive patients known for ovarian cancer. Patients treated with Bevacizumab, as first-line, relapse, or maintenance treatment (2015-2022). Results: according to FIGO staging, 98.2% (56 out of 57) of the patients in the study had third degree disease (G3). 49% of patients developed hypertension after starting Bevacizumab therapy (82% grade 2 according to CTCAE v.5). 89% of hypertensive patients started treatment and its management was multidisciplinary with nephrological consultation in 68% of cases. Only 3 out of 57 women discontinued treatment due to hypertension, and in only one of them it was not possible to restart it. Conclusions: the evaluation of the patient by a multidisciplinary team (gynecologist and nephrologist) is essential to minimize the morbidity and mortality of patients, and to avoid the interruption of antineoplastic treatment., (Copyright by Società Italiana di Nefrologia SIN, Rome, Italy.)
Published: 2022

30. New Landmarks to Slow the Progression of Chronic Kidney Disease.

Author: Simonini M and Vezzoli G
Abstract: Chronic kidney disease (CKD) is a serious condition whose incidence is steadily rising, particularly in the Western world, due to the increasing prevalence of diabetes, hypertension, and obesity, which are nowadays the major causes of CKD in the Western population, as well as the aging of the population [...].
Published: 2022
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31. Efficacy of burosumab in healing a long-standing femoral fracture in an adult patient with X-linked hypophosphatemic rickets.

Author: Arcidiacono T, Foligno N, and Vezzoli G
Subjects: Adult, Antibodies, Monoclonal, Antibodies, Monoclonal, Humanized therapeutic use, Fibroblast Growth Factors, Humans, Familial Hypophosphatemic Rickets complications, Familial Hypophosphatemic Rickets drug therapy, Femoral Fractures diagnostic imaging, Femoral Fractures drug therapy
Published: 2022
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32. Serum concentrations of free indoxyl and p-cresyl sulfate are associated with mineral metabolism variables and cardiovascular risk in hemodialysis patients.

Author: Arcidiacono T, Macrina L, Premaschi S, Bologna A, Magni G, Foligno N, Avino M, Belloni C, Palmieri N, Conte F, Bisegna S, Simonini M, Slaviero G, Locatelli M, and Vezzoli G
Subjects: Biomarkers, Calcium, Cresols, Heart Disease Risk Factors, Humans, Indican, Indoles, Minerals, Phosphates, Renal Dialysis adverse effects, Risk Factors, Sulfates, Cardiovascular Diseases diagnosis, Cardiovascular Diseases epidemiology, Cardiovascular Diseases etiology, Sulfuric Acid Esters
Abstract: Background: Indoxyl sulfate (IS) and p-cresyl sulfate (PCS) are uremic toxins associated with cardiovascular outcome in CKD patients. The present work is an analysis of the association of serum free, total IS and PCS with cardiovascular events and calcium-phosphate metabolism variables in hemodialysis patients., Methods: Serum levels of total and free IS and PCS were measured in 139 hemodialysis patients. Their relationship with calcium-phosphate metabolism variables were tested in an observational cohort study. In addition, their association with cardiovascular events was investigated during a 4-year follow-up., Results: Patients in the highest tertile (T3) of serum free IS showed lower serum 1,25(OH) 2 D compared to patients in the middle (T2) and lowest tertile (T1); in addition to this, T3 patients showed lower serum irisin than T1 patients and lower serum PTH than all the other subjects (T1 + T2) combined. Serum PTH was also measured during the two years after the baseline measurement and was higher in patients in the T1 than in those in the T3 of serum free IS. Cox regression analysis showed that cardiovascular risk was lower in T1 patients than in those in the T3 of serum free PCS, both using a univariate (OR 2.55, 95% CI 1.2-5.43; p = 0.015) or multivariate model (OR 2.48, 95% CI 1.12-5.51; p = 0.003)., Conclusions: Serum free IS may be associated with PTH and 1,25(OH) 2 D secretion, whereas free PCS may predict cardiovascular risk in hemodialysis patients., (© 2022. The Author(s) under exclusive licence to Italian Society of Nephrology.)
Published: 2022
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33. Serum Irisin May Predict Cardiovascular Events in Elderly Patients With Chronic Kidney Disease Stage 3-5.

Author: Arcidiacono T, Magni G, Macrina L, Sirtori M, Belloni C, Premaschi S, Lanzani C, Simonini M, Citterio L, Locatelli M, Rubinacci A, and Vezzoli G
Subjects: Aged, Disease Progression, Female, Fibronectins, Glomerular Filtration Rate, Humans, Kidney, Male, Cardiovascular Diseases, Renal Insufficiency, Chronic complications
Abstract: Objective: Irisin is a circulating myokine released from skeletal muscles after physical exercise. Irisin production decreases during the course of chronic kidney disease (CKD) as a potential consequence of sarcopenia and physical inactivity., Methods: This observational study explored the relationship of serum irisin with cardiovascular outcome in 79 patients with stage 3-5 CKD., Results: Serum irisin was significantly higher in healthy subjects (n = 20) than that in CKD patients (7 ± 2 vs. 3.1 ± 0.9 μg/mL; P = .0001) and was higher in patients with CKD stage 3 (3.2 ± 1 μg/mL) than in patients at stage 4 and 5 taken together (n = 36, 2.8 ± 0.7 μg/mL, P = .05). Patients in the lowest serum irisin tertile had lower serum 1,25(OH) 2 D levels (21 ± 11 pg/mL) than patients in the middle (30 ± 13 pg/mL; P = .005) and the highest tertile (27 ± 14 pg/mL; P = .047). Patients in the highest tertile had lower Kauppila score (10.6 ± 6.9) than patients in the middle (11.8 ± 5.5; P = .007) and the lowest tertile (6.9 ± 6.8; P = .043). Twenty patients suffered from cardiovascular events during a 3-year follow-up. A Cox regression model using age, body weight, presence of diabetes mellitus, gender, Kauppila calcification score, serum values of FGF23 (as logarithm), phosphate, sclerostin, albumin and cholesterol, estimated glomerular filtration rate, and serum irisin tertiles as covariates showed that patients in the highest tertile of serum irisin had a lower cardiovascular risk than patients in the middle tertile (B, 2.38; odds ratio, 10.8; 95% confidence interval, 1.65-58.13; P = .013) or in the lowest tertile (B, 1.61; odds ratio, 5; 95% confidence interval, 1.09-22.83; P = .038)., Conclusions: These findings suggest that serum irisin may be a marker of cardiovascular outcome in patients with CKD., (Copyright © 2021 National Kidney Foundation, Inc. Published by Elsevier Inc. All rights reserved.)
Published: 2022
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34. Acute Kidney Injury at Hospital Admission for SARS-CoV-2 Infection as a Marker of Poor Prognosis: Clinical Implications for Triage Risk Stratification.

Author: Bravi CA, Cazzaniga W, Simonini M, Larcher A, Messaggio E, Zagato L, Carenzi C, Bertini R, Briganti A, Manunta P, Vezzoli G, Salonia A, Lanzani C, Capitanio U, Zangrillo A, Landoni G, Rovere-Querini P, Tresoldi M, Montorsi F, and Ciceri F
Subjects: Hospital Mortality, Hospitals, Humans, Intensive Care Units, Prognosis, Retrospective Studies, Risk Assessment, Risk Factors, SARS-CoV-2, Triage, Acute Kidney Injury diagnosis, COVID-19
Abstract: Background/aims: The new severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes a wide spectrum of effects, including acute kidney injury (AKI) in up to 40% of hospitalized patients. Given the established relationship between AKI and poor prognosis, whether AKI might be a prognostic indicator for patients admitted to the hospital for SARS-CoV-2 infection would allow for a straightforward risk stratification of these patients., Methods: We analyzed data of 623 patients admitted to San Raffaele Hospital (Milan, IT) between February 25 and April 19, 2020, for laboratory-confirmed SARS-CoV-2 infection. Incidence of AKI at hospital admission was calculated, with AKI defined according to the KDIGO criteria. Multivariable Cox regression models assessed the association between AKI and overall mortality and admission to the intensive care unit (ICU)., Results: Overall, 108 (17%) patients had AKI at hospital admission for SARS-CoV-2 infection. After a median follow-up for survivors of 14 days (interquartile range: 8, 23), 123 patients died, while 84 patients were admitted to the ICU. After adjusting for confounders, patients who had AKI at hospital admission were at increased risk of overall mortality compared to those who did not have AKI (hazards ratio [HR]: 2.00; p = 0.0004), whereas we did not find evidence of an association between AKI and ICU admission (HR: 0.95; p = 0.9)., Conclusions: These data suggest that AKI might be an indicator of poor prognosis for patients with SARS-CoV-2 infection, and as such, given its readily availability, it might be used to improve risk stratification at hospital admission., (© 2022 The Author(s). Published by S. Karger AG, Basel.)
Published: 2022
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35. COVID-19 and the Environment: Pandemics, Climate, and Ecosystems, and the Environmental Challenge in Dialysis.

Author: Brioni E, Magnaghi C, Apuzzo L, Pole C, Pennaccho N, Manunta P, Vezzoli G, Zotti GD, Villa G, and Manara DF
Subjects: Ecosystem, Humans, Renal Dialysis adverse effects, SARS-CoV-2, COVID-19 epidemiology, Pandemics prevention & control
Abstract: The recent COVID-19 pandemic has had a significant impact on the population worldwide. Patients with chronic kidney disease treated with kidney replacement therapy were no exception because they were considered highly vulnerable due to multiple comorbidities. The consequences of the physical, biological, and ecological system on the environment as a result of human activity represent a huge global health care danger. The purpose of this article is to identify strategies that improve environmental sustainability, improve prevention of COVID-19 infection in dialysis centers, and improve the environmental impact of hemodialysis centers., Competing Interests: The authors reported no actual or potential conflict of interest in relation to this nursing continuing professional development (NCPD) activity., (Copyright© by the American Nephrology Nurses Association.)
Published: 2022

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35 results on '"Vezzoli, G."'

1. Effects of empagliflozin on progression of chronic kidney disease: a prespecified secondary analysis from the empa-kidney trial

2. Impact of primary kidney disease on the effects of empagliflozin in patients with chronic kidney disease: secondary analyses of the EMPA-KIDNEY trial

3. Climatic influence on sediment distribution and transport in the Thar Desert (Sindh and Cholistan, Pakistan)

4. Fast-eroding Taiwan and transfer of orogenic sediment to forearc basins and trenches in the Philippine and South China seas

5. Mapping Surface Features of an Alpine Glacier through Multispectral and Thermal Drone Surveys

6. Tracing erosion patterns in South Tibet: Balancing sediment supply to the Yarlung Tsangpo from the Himalaya versus Lhasa Block

7. Multi-proxy signatures in deep-water fine-grained sediments: Inherited versus syn-sedimentary factors controlling sediment compositions within a foredeep basin (Paleogene Adriatic foredeep – Northern Italy)

8. Geosites in the Gole della Breggia Geopark, Ticino, Southern Switzerland

9. Long-term safety and efficacy of left atrial appendage occlusion in dialysis patients with atrial fibrillation: a multi-center, prospective, open label, observational study

10. The Timing of Collision Between Asia and the West Burma Terrane, and the Development of the Indo-Burman Ranges

11. Andean retroarc-basin dune fields and Pampean Sand Sea (Argentina): Provenance and drainage changes driven by tectonics and climate

12. Interplay of Holocene surface faulting and climate in the Central Po Plain, Italy

13. Interplay of Holocene surface faulting and climate in the Central Po Plain, Italy

14. Beyond the kidneys: investigating airway involvement in autosomal dominant polycystic kidney disease through nasal nitric oxide profiling : A transversal single-center observational study.

15. Areas of uncertainty on the diagnosis, treatment, and follow-up of hypophosphatemia in adults: an Italian Delphi consensus.

16. Highlighting the Impact of Hormonal Factors on Hepatic Cystogenesis: Implications for Pathophysiology and Clinical Practice.

17. Is denosumab an efficient and safe drug for osteoporosis in dialysis patients? Considerations and state of the art about its use in this setting.

18. [ADPKD and IPMN: Mere Coincidence or Double Trouble?]

19. Unveiling Neglected Concerns: Possible Severe Hepatic Complications after Nephrectomy in Autosomal Dominant Polycystic Kidney Disease - A Case Report.

20. Assessment and management of heart failure in patients with chronic kidney disease.

21. Role of Female Sex Hormones in ADPKD Progression and a Personalized Approach to Contraception and Hormonal Therapy.

22. The Implications of the Vitamin D Metabolite Diagnostic Ratio in the Assessment of Patients With Idiopathic Hypercalciuria.

23. [The Management of Patients with Adult Autosomal Dominant Polycystic Kidney Disease (ADPKD) Requires a Multidisciplinary Approach].

24. Congenital solitary kidney in autosomal dominant polycystic kidney disease: Where do known genes end and the unknown begin?

25. [The Educational Aspect in Promoting a Low-Sodium Diet, Physical Activity and Therapy Adherence among Patients with Autosomal Dominant Polycystic Disease: A Literature Review].

26. Long-term safety and efficacy of left atrial appendage occlusion in dialysis patients with atrial fibrillation: a multi-center, prospective, open label, observational study.

27. Diagnostic policies on nephrolithiasis/nephrocalcinosis of possible genetic origin by Italian nephrologists: a survey by the Italian Society of Nephrology with an emphasis on primary hyperoxaluria.

28. Different Efficacy of Burosumab on Physical Performance and Serum Phosphate in Adult Patients with X-Linked Hyphophosphatemic Rickets during the First Six-Month of Treatment.

29. [Anti-angiogenic drugs and hypertension: from multidisciplinary collaboration to greater care].

30. New Landmarks to Slow the Progression of Chronic Kidney Disease.

31. Efficacy of burosumab in healing a long-standing femoral fracture in an adult patient with X-linked hypophosphatemic rickets.

32. Serum concentrations of free indoxyl and p-cresyl sulfate are associated with mineral metabolism variables and cardiovascular risk in hemodialysis patients.

33. Serum Irisin May Predict Cardiovascular Events in Elderly Patients With Chronic Kidney Disease Stage 3-5.

34. Acute Kidney Injury at Hospital Admission for SARS-CoV-2 Infection as a Marker of Poor Prognosis: Clinical Implications for Triage Risk Stratification.

35. COVID-19 and the Environment: Pandemics, Climate, and Ecosystems, and the Environmental Challenge in Dialysis.

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