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Your search keyword '"Nieva-Zuluaga, Ane"' showing total 21 results

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21 results on '"Nieva-Zuluaga, Ane"'

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1. FRI-472-YI Mitochondrial metabolism is disrupted by ciprofloxacin preventing cholangiocarcinoma cell proliferation

2. THU-210 Dysfunctional activation of the DNA damage response is associated with MASLD progression through an E2F2-dependent mechanism

3. FRI-343-YI APAP induced liver damage is prevented by activation of PPARgamma and PPAR-alpha

4. OS-101-YI Remodelling of hepatocyte cholesterol metabolism mediates colorectal liver metastasis

5. TOP-229-YI The E2F2 target glycerophosphodiester phosphodiesterase domain containing 3 is involved in MASLD progression to HCC and related dyslipidemias

6. Methionine adenosyltransferase 1a antisense oligonucleotides activate the liver-brown adipose tissue axis preventing obesity and associated hepatosteatosis

7. The E2F2-miR34a-5p axis is involved in the biliary metabolism dysregulation in NASH

8. Targeting the E2F/MCM axis in cholangiocarcinoma halts disease progression in experimental models by rewiring lipid metabolism

9. E2F2 deficiency protects from acetaminophen-induced hepatotoxicity while E2F1 is required to prevent the devastating effects

10. E2F2-promoted DNA damage in NASH worsens the metabolic scenario

11. The uptake of extracellular lipids promotes cholangiocarcinoma progression

12. miR34a-5p is a target of E2F2 transcription factor in MAFLD-related HCC

13. Methionine adenosyltransferase 1a antisense oligonucleotides induce the fibroblast growth factor 21-driven recovery from obesity and associated hepatoesteatosis

14. The DNA damage response is involved in the metabolic dysregulation of MAFLD patients via inefficient fatty acid oxidation

15. Methionine adenosyltransferase 1a antisense oligonucleotides activate the liver-brown adipose tissue axis preventing obesity and associated hepatosteatosis

16. Cholangiocarcinoma progression depends on the uptake and metabolization of extracellular lipids

17. Cholangiocarcinoma progression depends on the uptake and metabolization of extracellular lipids

18. WED-408 - E2F2-promoted DNA damage in NASH worsens the metabolic scenario

19. WED-404 - The E2F2-miR34a-5p axis is involved in the biliary metabolism dysregulation in NASH

20. SAT-215 - Targeting the E2F/MCM axis in cholangiocarcinoma halts disease progression in experimental models by rewiring lipid metabolism

21. FRI-395 - E2F2 deficiency protects from acetaminophen-induced hepatotoxicity while E2F1 is required to prevent the devastating effects

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