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1. Systems-based identification of the Hippo pathway for promoting fibrotic mesenchymal differentiation in systemic sclerosis

2. Single-cell sequencing reveals Hippo signaling as a driver of fibrosis in hidradenitis suppurativa.

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3. Single-cell profiling of prurigo nodularis demonstrates immune-stromal crosstalk driving profibrotic responses and reversal with nemolizumab.

4. Single cell and spatial sequencing define processes by which keratinocytes and fibroblasts amplify inflammatory responses in psoriasis.

5. Differential cell composition and split epidermal differentiation in human palm, sole, and hip skin

14. Association of germline variants in telomere maintenance genes (POT1, TERF2IP, ACD, and TERT) with spitzoid morphology in familial melanoma: A multi-center case series

16. Intersection of immune and oncometabolic pathways drives cancer hyperprogression during immunotherapy

17. A case of NONO::TFE3 cutaneous epithelioid and spindle cell tumor with local recurrence after complete excision.

21. GATA-3 is a proto-oncogene in T-cell lymphoproliferative neoplasms

23. Porocarcinomas with PAK1/2/3 fusions: a series of 12 cases.

27. Image-based multiplex immune profiling of cancer tissues: translational implications. A report of the International Immuno-oncology Biomarker Working Group on Breast Cancer

28. Single-cell sequencing reveals Hippo signaling as a driver of fibrosis in hidradenitis suppurativa

29. Image-based multiplex immune profiling of cancer tissues:translational implications. A report of the International Immuno-oncology Biomarker Working Group on Breast Cancer

30. Image‐based multiplex immune profiling of cancer tissues: translational implications. A report of the International Immuno‐oncology Biomarker Working Group on Breast Cancer

31. Merkel cell carcinoma: updates in tumor biology, emerging therapies, and preclinical models.

32. Single cell sequencing reveals Hippo signaling as a driver of fibrosis in hidradenitis suppurativa

34. Corrigendum to “Characterizing the Therapeutic Potential of a Potent BET Degrader in Merkel Cell Carcinoma” [Neoplasia, Volume 21, Issue 3 (2019) 322–330]

35. Direct cellular reprogramming enables development of viral T antigen-driven Merkel cell carcinoma in mice

36. Translational implications of Th17-skewed inflammation due to genetic deficiency of a cadherin stress sensor

37. Transcriptional profiling of rare acantholytic disorders suggests common mechanisms of pathogenesis

39. Single-cell profiling of prurigo nodularis demonstrates immune-stromal crosstalk driving profibrotic responses and reversal with nemolizumab

40. CD30 expression in cutaneous B‐cell lymphomas

43. Data from Comprehensive Molecular Profiling of Olfactory Neuroblastoma Identifies Potentially Targetable FGFR3 Amplifications

44. Supplementary Data from Comprehensive Molecular Profiling of Olfactory Neuroblastoma Identifies Potentially Targetable FGFR3 Amplifications

45. Supplementary Table 2 from Age and Gender Associations of Virus Positivity in Merkel Cell Carcinoma Characterized Using a Novel RNA In Situ Hybridization Assay

46. SFigure S1 from Age and Gender Associations of Virus Positivity in Merkel Cell Carcinoma Characterized Using a Novel RNA In Situ Hybridization Assay

47. Supplementary Data from Virus-positive Merkel Cell Carcinoma Is an Independent Prognostic Group with Distinct Predictive Biomarkers

49. Figure S3 from Age and Gender Associations of Virus Positivity in Merkel Cell Carcinoma Characterized Using a Novel RNA In Situ Hybridization Assay

50. Supplementary Table 1 from Age and Gender Associations of Virus Positivity in Merkel Cell Carcinoma Characterized Using a Novel RNA In Situ Hybridization Assay