31 results on '"Gilson, Paul R."'
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2. Bottoms up! Malaria parasite invasion the right way around
3. Conditional expression of NanoLuc luciferase through a multimodular system offers rapid detection of antimalarial drug activity
4. Characterisation of PfCZIF1 and PfCZIF2 in Plasmodium falciparum asexual stages
5. Defining species-specific and conserved interactions of apical membrane protein 1 during erythrocyte invasion in malaria to inform multi-species vaccines
6. Property activity refinement of 2-anilino 4-amino substituted quinazolines as antimalarials with fast acting asexual parasite activity
7. Activity Refinement of Aryl Amino Acetamides that Target the P. Falciparum Star-Related Lipid Transfer 1 Protein
8. Sequence elements within the PEXEL motif and its downstream region modulate PTEX‐dependent protein export in Plasmodium falciparum
9. Aryl amino acetamides prevent the development ofPlasmodium falciparumrings via inhibition of the lipid transfer protein PfSTART1
10. The dual action of human antibodies specific to Plasmodium falciparum PfRH5 and PfCyRPA: Blocking invasion and inactivating extracellular merozoites
11. A Pyridyl-Furan Series Developed from the Open Global Health Library Block Red Blood Cell Invasion and Protein Trafficking in Plasmodium falciparum through Potential Inhibition of the Parasite’s PI4KIIIB Enzyme
12. PTEX helps efficiently traffic haemoglobinases to the food vacuole in Plasmodium falciparum
13. The delayed bloodstream clearance of Plasmodium falciparum parasites after M5717 treatment is attributable to the inability to modify their red blood cell hosts
14. Sequence elements within the PEXEL motif and its downstream region modulate PTEX‐dependent protein export in Plasmodium falciparum.
15. A pyridyl-furan series developed from Open Global Health Library blocks red blood cell invasion and protein trafficking inPlasmodium falciparumthrough potential inhibition of the parasite’s PI4KIIIb enzyme
16. The delayed bloodstream clearance of Plasmodium falciparum parasites after M5717 treatment is attributable to the inability to modify their red blood cell hosts.
17. Sulfonylpiperazine compounds prevent Plasmodium falciparum invasion of red blood cells through interference with actin-1/profilin dynamics
18. Sequence elements within the PEXEL motif and its downstream region modulate PTEX dependent protein export in Plasmodium falciparum .
19. The Dual Action of Human Antibodies Specific toPlasmodium falciparumPfRH5 and PfCyRPA: Blocking Invasion and Inactivating Extracellular Merozoites
20. PfATP4 inhibitors in the Medicines for Malaria Venture Malaria Box and Pathogen Box block the schizont-to-ring transition by inhibiting egress rather than invasion
21. PTEX helps efficiently traffic haemoglobinases to the food vacuole inPlasmodium falciparum
22. The sulfonylpiperazine MMV020291 prevents red blood cell invasion by the malaria parasite Plasmodium falciparum through interference with actin-1/profilin dynamics
23. The Medicines for Malaria Venture Malaria Box contains inhibitors of protein secretion in Plasmodium falciparum blood stage parasites
24. The delayed bloodstream clearance of Plasmodium falciparum parasites after M5717 treatment is attributable to the inability to modify their red blood cell hosts Molly.
25. The Plasmodium falciparum parasitophorous vacuole protein P113 interacts with the parasite protein export machinery and maintains normal vacuole architecture
26. A revised mechanism for how Plasmodium falciparum recruits and exports proteins into its erythrocytic host cell
27. Characterisation of PfCZIF1 and PfCZIF2 in Plasmodium falciparum asexual stages
28. Author Correction: Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1.
29. Aryl amino acetamides prevent Plasmodium falciparum ring development via targeting the lipid-transfer protein PfSTART1.
30. Dissecting EXP2 sequence requirements for protein export in malaria parasites.
31. Pf ATP4 inhibitors in the Medicines for Malaria Venture Malaria Box and Pathogen Box block the schizont-to-ring transition by inhibiting egress rather than invasion.
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