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2. Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma

3. iCLOTS: open-source, artificial intelligence-enabled software for analyses of blood cells in microfluidic and microscopy-based assays

4. Genetic reversal of the globin switch concurrently modulates both fetal and sickle hemoglobin and reduces red cell sickling

11. Next generation microfluidics: fulfilling the promise of lab-on-a-chip technologies

12. CDK4/6 inhibition enhances SHP2 inhibitor efficacy and is dependent upon RB function in malignant peripheral nerve sheath tumors

16. Ex vivo to in vivo model of malignant peripheral nerve sheath tumors for precision oncology

19. High-throughput quantification of red blood cell deformability and oxygen saturation to probe mechanisms of sickle cell disease.

20. CDK4/6 inhibition enhances SHP2 inhibitor efficacy and is dependent upon restoration of RB function in malignant peripheral nerve sheath tumors

21. Fluorescence Lifetime Measurement of Prefibrillar Sickle Hemoglobin Oligomers as a Platform for Drug Discovery in Sickle Cell Disease

22. Ex vivo to in vivo model of malignant peripheral nerve sheath tumors for precision oncology

30. Effects of BCL11A Shmir-Induced Post-Transcriptional Silencing on Hemoglobin Polymer Inhibition in Single Red Blood Cells at Physiologic Oxygen Tension

34. An irradiated marrow niche reveals a small non-collagenous protein mediator of homing, dermatopontin

38. Stromal architecture directs early dissemination in pancreatic ductal adenocarcinoma

39. Robust Pre-Clinical Results and Large-Scale Manufacturing Process for Edit-301: An Autologous Cell Therapy for the Potential Treatment of SCD

46. Validation of BCL11A As Therapeutic Target in Sickle Cell Disease: Results from the Adult Cohort of a Pilot/Feasibility Gene Therapy Trial Inducing Sustained Expression of Fetal Hemoglobin Using Post-Transcriptional Gene Silencing

47. MetAP2 Inhibition Modifies Hemoglobin S (HbS) to Delay Polymerization and Improve Blood Flow in Sickle Cell Disease

48. SEMA4C is a novel target to limit osteosarcoma growth, progression, and metastasis

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