34 results on '"Varga, Georg"'
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2. Clinical and molecular analysis of a novel variant in heme oxygenase-1 deficiency: Unraveling its role in inflammation, heme metabolism, and pulmonary phenotype
3. Conversion of dendritic cells into tolerogenic or inflammatory cells depends on the activation threshold and kinetics of the mTOR signaling pathway
4. Small Spleen Peptides (SSPs) Shape Dendritic Cell Differentiation through Modulation of Extracellular ATP Synthesis Profile
5. Small spleen peptides prevent development of psoriatic arthritis via restoration of peripheral tolerance
6. A myostatin-CCL20–CCR6 axis regulates Th17 cell recruitment to inflamed joints in experimental arthritis
7. Impaired IFN-γ-dependent STAT3 Activation Is Associated With Dysregulation of Regulatory and Inflammatory Signaling in Monocytes of Ulcerative Colitis Patients
8. The Impact of Phase-Specific Macrophage Depletion on Intestinal Anastomotic Healing
9. The Impact of Phase-Specific Macrophage Depletion on Intestinal Anastomotic Healing
10. Granulocyte Macrophage Colony-Stimulating Factor–Activated CD39+/CD73+ Murine Monocytes Modulate Intestinal Inflammation via Induction of Regulatory T Cells
11. Anti-inflammatory monocytes—interplay of innate and adaptive immunity
12. Redox distress and genetic defects conspire in systemic autoinflammatory diseases
13. 02.27 Myostatin-induced mechanisms of inflammatory bone destruction by invasive fibroblast-like synoviocytes
14. Patient parameters and response after administration of rituximab in pediatric mature B‐cell non‐Hodgkin lymphoma
15. Characterization of tumor response after administration of rituximab in pediatric B-NHL
16. Patient parameters and response after administration of rituximab in pediatric mature B‐cell non‐Hodgkin lymphoma.
17. Myeloid regulatory cells: cross talk of innate and adaptive immunity to maintain the intestinal immune homeostasis in inflammatory bowel disease
18. Differential regulation of JAK/STAT-signaling in patients with ulcerative colitis and Crohn’s disease
19. Spontaneous onset of TNFα‐triggered colonic inflammation depends on functional T lymphocytes, S100A8 / A9 alarmins, and MHC H‐2 haplotype
20. Impaired IFN-γ-dependent STAT3 Activation Is Associated With Dysregulation of Regulatory and Inflammatory Signaling in Monocytes of Ulcerative Colitis Patients.
21. Tofacitinib Reprograms Human Monocytes of IBD Patients and Healthy Controls Toward a More Regulatory Phenotype
22. Spontaneous onset of TNFα‐triggered colonic inflammation depends on functional T lymphocytes, S100A8/A9 alarmins, and MHC H‐2 haplotype.
23. Tofacitinib Reprograms Human Monocytes of IBD Patients and Healthy Controls Toward a More Regulatory Phenotype.
24. Oncolytic influenza virus infection restores immunocompetence of lung tumor-associated alveolar macrophages
25. Balancing JAK/STAT-Signaling with Tofacitinib in Monocytes of Healthy Controls and IBD Patients
26. 02.27 Myostatin-induced mechanisms of inflammatory bone destruction by invasive fibroblast-like synoviocytes
27. Macrophage‐mediated psoriasis can be suppressed by regulatory T lymphocytes
28. Activation-dependent cell death of human monocytes is a novel mechanism of fine-tuning inflammation and autoimmunity
29. Abstract 5114: Multispectral optoacoustic tomography (MSOT) as a novel approach for dynamic, noninvasive assessment of tumor therapy response and chronic inflammation
30. Reprogramming of Monocytes by GM-CSF Contributes to Regulatory Immune Functions during Intestinal Inflammation
31. Optical In Vivo Imaging of the Alarmin S100A9 in Tumor Lesions Allows for Estimation of the Individual Malignant Potential by Evaluation of Tumor–Host Cell Interaction
32. Mo1712 - Balancing JAK/STAT-Signaling with Tofacitinib in Monocytes of Healthy Controls and IBD Patients
33. MAPKAP kinase 3 suppresses Ifng gene expression and attenuates NK cell cytotoxicity and Th1 CD4 T-cell development upon influenza A virus infection.
34. Clinical and molecular analysis of a novel variant in heme oxygenase-1 deficiency: Unraveling its role in inflammation, heme metabolism, and pulmonary phenotype.
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