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2. SHP2 blockade enhances anti-tumor immunity via tumor cell intrinsic and extrinsic mechanisms

3. TGFβ-blockade uncovers stromal plasticity in tumors by revealing the existence of a subset of interferon-licensed fibroblasts

4. TIM3 Mediates T Cell Exhaustion during Mycobacterium tuberculosis Infection.

5. Targeting the IL1β Pathway for Cancer Immunotherapy Remodels the Tumor Microenvironment and Enhances Antitumor Immune Responses

6. Supplementary Tables and Figures from Targeting the IL1β Pathway for Cancer Immunotherapy Remodels the Tumor Microenvironment and Enhances Antitumor Immune Responses

7. Data from Targeting the IL1β Pathway for Cancer Immunotherapy Remodels the Tumor Microenvironment and Enhances Antitumor Immune Responses

8. Targeting stromal cell sialylation reverses T cell-mediated immunosuppression in the tumor microenvironment

9. 772 A phase 1/2 dose escalation/expansion study evaluating the safety, pharmacokinetics, pharmacodynamics, and antitumor activity of E-602, a bi-sialidase fusion protein, in advanced cancer (GLIMMER-01)

10. TGFβ-blockade uncovers stromal plasticity in tumors by revealing the existence of a subset of interferon-licensed fibroblasts

11. Abstract CT034: GLIMMER-01: initial results from a phase 1 dose escalation trial of a first-in-class bi-sialidase (E-602) in solid tumors

12. Characterization of sabatolimab, a novel immunotherapy with immuno-myeloid activity directed against TIM-3 receptor

13. 439 Dual modes of action for anti-TIM-3 antibody MBG453 in myelodysplastic syndromes (MDS) and acute myeloid leukemia (AML): preclinical evidence for immune-mediated and anti-leukemic activity

14. Multiple Inflammatory Cytokines Converge To Regulate CD8+ T Cell Expansion and Function during Tuberculosis

15. Abstract 5640: Targeting tumor-promoting inflammation (TPI) via the IL-1βpathway for cancer immunotherapy

16. Abstract A76: Anti IL-1b as a cancer immunotherapy

17. Abstract C103: Targeting IL-1β pathway for cancer immunotherapy

18. IL-21 signaling is essential for optimal host resistance against Mycobacterium tuberculosis infection

19. TIM3 Mediates T Cell Exhaustion during Mycobacterium tuberculosis Infection

20. Defining the contribution of T cell exhaustion to failed immunity during TB

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