209 results on '"Lemieux, Madeleine"'
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2. Obesity-linked suppression of membrane-bound O-acyltransferase 7 (MBOAT7) drives non-alcoholic fatty liver disease.
3. Dynamic Chromatin Targeting of BRD4 Stimulates Cardiac Fibroblast Activation
4. Matrix-Degrading Enzyme Expression and Aortic Fibrosis During Continuous-Flow Left Ventricular Mechanical Support
5. BET bromodomain inhibition suppresses innate inflammatory and profibrotic transcriptional networks in heart failure.
6. Glucocorticoids enhance muscle endurance and ameliorate Duchenne muscular dystrophy through a defined metabolic program
7. A chromosome-level genome of Astyanax mexicanus surface fish for comparing population-specific genetic differences contributing to trait evolution
8. BPTF regulates growth of adult and pediatric high-grade glioma through the MYC pathway
9. Substrate stiffness modulates cardiac fibroblast activation, senescence, and proinflammatory secretory phenotypeSubstrate stiffness modulates cardiac fibroblast activation, senescence, and proinflammatory secretory phenotype.
10. EZH2 Cooperates with BRD4-NUT to Drive NUT Carcinoma Growth by Silencing Key Tumor Suppressor Genes
11. Torsional and strain dysfunction precede overt heart failure in a mouse model of dilated cardiomyopathy pathogenesis
12. EZH2 synergizes with BRD4-NUT to drive NUT carcinoma growth through silencing of key tumor suppressor genes
13. Ectopic protein interactions within BRD4–chromatin complexes drive oncogenic megadomain formation in NUT midline carcinoma
14. Supplemental Figures from Combined Targeting of the BRD4–NUT–p300 Axis in NUT Midline Carcinoma by Dual Selective Bromodomain Inhibitor, NEO2734
15. Data from Chemical Screen Identifies Diverse and Novel Histone Deacetylase Inhibitors as Repressors of NUT Function: Implications for NUT Carcinoma Pathogenesis and Treatment
16. Data from NSD3–NUT Fusion Oncoprotein in NUT Midline Carcinoma: Implications for a Novel Oncogenic Mechanism
17. Supplementary Figure 4 from NSD3–NUT Fusion Oncoprotein in NUT Midline Carcinoma: Implications for a Novel Oncogenic Mechanism
18. Supplementary Figure 3 from NSD3–NUT Fusion Oncoprotein in NUT Midline Carcinoma: Implications for a Novel Oncogenic Mechanism
19. Supplementary Methods from Combined Targeting of the BRD4–NUT–p300 Axis in NUT Midline Carcinoma by Dual Selective Bromodomain Inhibitor, NEO2734
20. Supplemental Figures 1-9 from Combined Targeting of the BRD4–NUT–p300 Axis in NUT Midline Carcinoma by Dual Selective Bromodomain Inhibitor, NEO2734
21. Supplementary Figure 2 from NSD3–NUT Fusion Oncoprotein in NUT Midline Carcinoma: Implications for a Novel Oncogenic Mechanism
22. Table S1 from Chemical Screen Identifies Diverse and Novel Histone Deacetylase Inhibitors as Repressors of NUT Function: Implications for NUT Carcinoma Pathogenesis and Treatment
23. Supplementary Tables 1-3 from Combined Targeting of the BRD4–NUT–p300 Axis in NUT Midline Carcinoma by Dual Selective Bromodomain Inhibitor, NEO2734
24. Supplementary Figure 1 from NSD3–NUT Fusion Oncoprotein in NUT Midline Carcinoma: Implications for a Novel Oncogenic Mechanism
25. Supplementary Data from Combined Targeting of the BRD4–NUT–p300 Axis in NUT Midline Carcinoma by Dual Selective Bromodomain Inhibitor, NEO2734
26. Supplementary Methods from Chemical Screen Identifies Diverse and Novel Histone Deacetylase Inhibitors as Repressors of NUT Function: Implications for NUT Carcinoma Pathogenesis and Treatment
27. Supplementary Figure Legends from Chemical Screen Identifies Diverse and Novel Histone Deacetylase Inhibitors as Repressors of NUT Function: Implications for NUT Carcinoma Pathogenesis and Treatment
28. Supplementary Figures 1-4 from Chemical Screen Identifies Diverse and Novel Histone Deacetylase Inhibitors as Repressors of NUT Function: Implications for NUT Carcinoma Pathogenesis and Treatment
29. Macrophages directly contribute collagen to scar formation during zebrafish heart regeneration and mouse heart repair
30. Supplemental Table S1 from High-throughput Chemical Screening Identifies Focal Adhesion Kinase and Aurora Kinase B Inhibition as a Synergistic Treatment Combination in Ewing Sarcoma
31. Supplementary Data from High-throughput Chemical Screening Identifies Focal Adhesion Kinase and Aurora Kinase B Inhibition as a Synergistic Treatment Combination in Ewing Sarcoma
32. Data from Differentiation of NUT Midline Carcinoma by Epigenomic Reprogramming
33. Supplementary Figures 1-2 from Differentiation of NUT Midline Carcinoma by Epigenomic Reprogramming
34. Inhibition of Eicosanoid Degradation Mitigates Fibrosis of the Heart
35. Stable inhibitory activity of regulatory T cells requires the transcription factor Helios
36. Phenotypic screening uncovers eicosanoid degradation in fibroblasts as a therapeutic target for cardiac fibrosis
37. Correction: BPTF regulates growth of adult and pediatric high-grade glioma through the MYC pathway
38. Mediator kinase inhibition further activates super-enhancer-associated genes in AML
39. Monocytes transition to monocyte‐macrophages within the inflamed vasculature via CCR2 on monocytes and endothelial TNFR2
40. Monocytes transition to macrophages within the inflamed vasculature via monocyte CCR2 and endothelial TNFR2
41. Discordant Genome Assemblies Drastically Alter the Interpretation of Single-Cell RNA Sequencing Data Which Can Be Mitigated by a Novel Integration Method
42. T CELL IMMUNITY: Stable inhibitory activity of regulatory T cells requires the transcription factor Helios
43. Hyperglycemia Induces Trained Immunity in Macrophages and Their Precursors and Promotes Atherosclerosis
44. Chemical Screen Identifies Diverse and Novel Histone Deacetylase Inhibitors as Repressors of NUT Function: Implications for NUT Carcinoma Pathogenesis and Treatment
45. Early Detection of Lung Cancer with Meso Tetra (4-Carboxyphenyl) Porphyrin-Labeled Sputum
46. Distinct dynamics of the extracellular matrix during heart regeneration in different Astyanax mexicanus populations
47. Condensin II protein dysfunction impacts mitochondrial respiration and mitochondrial oxidative stress responses
48. GATA6 regulates aging of human mesenchymal stem/stromal cells
49. IER5, a DNA damage response gene, is required for Notch-mediated induction of squamous cell differentiation
50. Author response: IER5, a DNA damage response gene, is required for Notch-mediated induction of squamous cell differentiation
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